1. Comparative Analysis of Gene Expression Profiles Between Cortex and Thalamus in Chinese Fatal Familial Insomnia Patients
    Chan Tian et al, 2013, Molecular Neurobiology CrossRef
  2. Overexpression of p62/SQSTM1 promotes the degradations of abnormally accumulated PrP mutants in cytoplasm and relieves the associated cytotoxicities via autophagy–lysosome-dependent way
    Yin Xu et al, 2014, Medical Microbiology and Immunology CrossRef
  3. Integrative genomics analysis identifies five promising genes implicated in insomnia risk based on multiple omics datasets
    Haozhen Sun et al, 2020, Bioscience Reports CrossRef
  4. Identification of new molecular alterations in fatal familial insomnia
    Franc Llorens et al, 2016, Human Molecular Genetics CrossRef
  5. Different post-mortem brain regions from three Chinese FFI patients induce different reactive profiles both in the first and second generation RT-QuIC assays
    Kang Xiao et al, 2020, Prion CrossRef
  6. Proteomics Analyses for the Global Proteins in the Brain Tissues of Different Human Prion Diseases*
    Qi Shi et al, 2015, Molecular & Cellular Proteomics CrossRef
  7. The Features of Genetic Prion Diseases Based on Chinese Surveillance Program
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  8. Comparative proteomics analyses for 139A and ME7 scrapie infected mice brains in the middle and terminal stages
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  9. At the centre of neuronal, synaptic and axonal pathology in murine prion disease: degeneration of neuroanatomically linked thalamic and brainstem nuclei
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  10. Genetic Prion Disease: Insight from the Features and Experience of China National Surveillance for Creutzfeldt-Jakob Disease
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