1
|
Liu Y, Gorospe M, Holbrook NJ and Anderson
CW: Post-translational mechanisms leading to mammalian gene
activation in response to genotoxic stress. DNA Damage and Repair.
Nickoloff JA and Hoekstra MF: 2. Humana Press Inc; Totowa, NJ: pp.
263–298. 1998, View Article : Google Scholar
|
2
|
Whitmarsh AJ and Davis RJ: Transcription
factor AP-1 regulation by mitogen-activated protein kinase signal
transduction pathways. J Mol Med. 74:589–607. 1996. View Article : Google Scholar : PubMed/NCBI
|
3
|
Tournier C, Hess P, Yang DD, Xu J, Turner
TK, Nimnual A, Bar-Sagi D, Jones SN, Flavell RA and Davis RJ:
Requirement of JNK for stress-induced activation of the cytochrome
c-mediated death pathway. Science. 288:870–874. 2000. View Article : Google Scholar : PubMed/NCBI
|
4
|
Parra E and Ferreira J: The effect of
siRNA-Egr-1 and camptothecin on growth and chemosensitivity of
breast cancer cell lines. Oncol Rep. 22:1159–1165. 2010.
|
5
|
Dorigo O, Turla ST, Lebedeva S and Gjerset
RA: Sensitization of rat glioblastoma multiforme to cisplatin in
vivo following restoration of wild-type p53 function. J Neurosurg.
88:535–540. 1998. View Article : Google Scholar : PubMed/NCBI
|
6
|
Ru P, Steele R, Hsueh EC and Ray RB:
Anti-miR-203 upregulates SOCS3 expression in breast cancer cells
and enhances cisplatin chemosensitivity. Genes Cancer. 2:720–727.
2011. View Article : Google Scholar : PubMed/NCBI
|
7
|
Keshava R, Jothi M, Gope M and Gope R:
Functional modulation of the p53 gene and its protein in human
brain tumors. Ann Neurosci. 15:32008.
|
8
|
Bullock AN and Fersht AR: Rescuing the
function of mutant p53. Nat Rev Cancer. 1:68–76. 2001. View Article : Google Scholar
|
9
|
Raitano AB, Halpern JR, Hambuch TM and
Sawyers CL: The Bcr-Abl leukemia oncogene activates Jun kinase and
requires Jun for transformation. Proc Natl Acad Sci USA.
92:11746–11750. 1995. View Article : Google Scholar : PubMed/NCBI
|
10
|
Dickens M, Rogers JS, Cavanagh J, Raitano
A, Xia Z, Halpern JR, Greenberg ME, Sawyers CL and Davis RJ: A
cytoplasmic inhibitor of the JNK signal transduction pathway.
Science. 277:693–696. 1997. View Article : Google Scholar : PubMed/NCBI
|
11
|
Shi CS, Tuscano JM, Witte ON and Kehrl JH:
GCKR links the Bcr-Abl oncogene and Ras to the stress-activated
protein kinase pathway. Blood. 93:1338–1345. 1999.PubMed/NCBI
|
12
|
Atfi A, Prunier C, Mazars A, Défachelles
AS, Cayre Y, Gespach C and Bourgeade MF: The oncogenic TEL/PDGFR β
fusion protein induces cell death through JNK/SAPK pathway.
Oncogene. 18:3878–3885. 1999. View Article : Google Scholar : PubMed/NCBI
|
13
|
Bost F, McKay R, Dean N and Mercola D: The
JUN kinase/stress-activated protein kinase pathway is required for
epidermal growth factor stimulation of growth of human A549 lung
carcinoma cells. J Biol Chem. 272:33422–33429. 1997. View Article : Google Scholar
|
14
|
Nielsen C, Thastrup J, Bøttzauw T,
Jäättelä M and Kallunki T: c-Jun NH2-terminal kinase 2
is required for Ras transformation independently of activator
protein 1. Cancer Res. 67:178–185. 2007. View Article : Google Scholar : PubMed/NCBI
|
15
|
Wu S, Loke HN and Rehemtulla A:
Ultraviolet radiation-induced apoptosis is mediated by Daxx.
Neoplasia. 4:486–492. 2002. View Article : Google Scholar : PubMed/NCBI
|
16
|
Mansouri A, Ridgway LD, Korapati AL, Zhang
Q, Tian L, Wang Y, Siddik ZH, Mills GB and Claret FX: Sustained
activation of JNK/p38 MAPK pathways in response to cisplatin leads
to Fas ligand induction and cell death in ovarian carcinoma cells.
J Biol Chem. 278:19245–19256. 2003. View Article : Google Scholar : PubMed/NCBI
|
17
|
Parra E and Ferreira J: Modulation of the
response of prostate cancer cell lines to cisplatin treatment using
small interfering RNA. Oncol Rep. 30:1936–1942. 2013.PubMed/NCBI
|
18
|
Persons DL, Yazlovitskaya EY, Cui W and
Pelling JC: Cisplatin-induced activation of mitogen-activated
protein kinases in ovarian carcinoma cells: inhibition of
extracellular signal-regulated kinase activity increases
sensitivity to cisplatin. Clin Cancer Res. 5:1007–1014.
1999.PubMed/NCBI
|
19
|
Sánchez-Perez I, Murguía JR and Perona R:
Cisplatin induces a persistent activation of JNK that is related to
cell death. Oncogene. 16:533–540. 1998. View Article : Google Scholar : PubMed/NCBI
|
20
|
Costa-Pereira AP, McKenna SL and Cotter
TG: Activation of SAPK/JNK by camptothecin sensitizes
androgen-independent prostate cancer cells to Fas-induced
apoptosis. Br J Cancer. 82:1827–1834. 2000. View Article : Google Scholar : PubMed/NCBI
|
21
|
Woo RA, McLure KG, Lees-Miller SP,
Rancourt DE and Lee PW: DNA-dependent protein kinase acts upstream
of p53 in response to DNA damage. Nature. 394:700–704. 1998.
View Article : Google Scholar : PubMed/NCBI
|
22
|
Still IH, Hamilton M, Vince P, Wolfman A
and Cowell JK: Cloning of TACC1, an embryonically expressed,
potentially transforming coiled coil containing gene, from the 8p11
breast cancer amplicon. Oncogene. 18:4032–4038. 1999. View Article : Google Scholar : PubMed/NCBI
|
23
|
Parra E, Gutiérrez L and Ferreira J:
Increased expression of p21Waf1/Cip1 and JNK with costimulation of
prostate cancer cell activation by an siRNA Egr-1 inhibitor. Oncol
Rep. 30:911–916. 2013.PubMed/NCBI
|
24
|
Parra E: Inhibition of JNK-1 by small
interfering RNA induces apoptotic signaling in PC-3 prostate cancer
cells. Int J Mol Med. 30:923–930. 2012.PubMed/NCBI
|
25
|
Parra E and Ferreira J: Knockdown of the
c-Jun-N-terminal kinase expression by siRNA inhibits MCF-7 breast
carcinoma cell line growth. Oncol Rep. 24:1339–1345. 2010.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Weston CR and Davis RJ: The JNK signal
transduction pathway. Curr Opin Cell Biol. 19:142–149. 2007.
View Article : Google Scholar : PubMed/NCBI
|
27
|
Vucic D, Dixit VM and Wertz IE:
Ubiquitylation in apoptosis: a post-translational modification at
the edge of life and death. Nat Rev Mol Cell Biol. 12:439–452.
2011. View
Article : Google Scholar : PubMed/NCBI
|
28
|
Bertrand MJ, Milutinovic S, Dickson KM, Ho
WC, Boudreault A, Durkin J, Gillard JW, Jaquith JB, Morris SJ and
Barker PA: cIAP1 and cIAP2 facilitate cancer cell survival by
functioning as E3 ligases that promote RIP1 ubiquitination. Mol
Cell. 30:689–700. 2008. View Article : Google Scholar : PubMed/NCBI
|
29
|
Fulda S: Regulation of cell migration,
invasion and metastasis by IAP proteins and their antagonists.
Oncogene. 33:671–676. 2014. View Article : Google Scholar
|
30
|
Gascoyne RD, Krajewska M, Krajewski S,
Connors JM and Reed JC: Prognostic significance of Bax protein
expression in diffuse aggressive non-Hodgkin’s lymphoma. Blood.
90:3173–3178. 1997.PubMed/NCBI
|
31
|
Youle RJ and Strasser A: The BCL-2 protein
family: opposing activities that mediate cell death. Nat Rev Mol
Cell Biol. 9:47–59. 2008. View
Article : Google Scholar
|
32
|
Zha H, Aimé-Sempé C, Sato T and Reed JC:
Proapoptotic protein Bax heterodimerizes with Bcl-2 and
homodimerizes with Bax via a novel domain (BH3) distinct from BH1
and BH2. J Biol Chem. 271:7440–7444. 1996. View Article : Google Scholar : PubMed/NCBI
|
33
|
Reed JC: Mechanisms of Bcl-2 family
protein function and dysfunction in health and disease. Behring
Inst Mitt. 97:72–100. 1996.PubMed/NCBI
|
34
|
Metcalfe AD, Gilmore A, Klinowska T,
Oliver J, Valentijn AJ, Brown R, Ross A, MacGregor G, Hickman JA
and Streuli CH: Developmental regulation of Bcl-2 family protein
expression in the involuting mammary gland. J Cell Sci.
112:1771–1783. 1999.PubMed/NCBI
|
35
|
Basu A and Haldar S: The relationship
between Bcl2, Bax and p53: consequences for cell cycle progression
and cell death. Mol Hum Reprod. 4:1099–1109. 1998. View Article : Google Scholar
|
36
|
Fritz G and Kaina B: Activation of c-Jun
N-terminal kinase 1 by UV irradiation is inhibited by wortmannin
without affecting c-jun expression. Mol Cell Biol. 19:1768–1774.
1999.PubMed/NCBI
|
37
|
Smeal T, Binetruy B, Mercola DA, Birrer M
and Karin M: Oncogenic and transcriptional cooperation with Ha-Ras
requires phosphorylation and c-Jun on serines 63 and 73. Nature.
354:494–496. 1991. View
Article : Google Scholar : PubMed/NCBI
|
38
|
Brown PH, Chen TK and Birrer MJ: Mechanism
of action of a dominant-negative mutant of c-Jun. Oncogene.
9:791–799. 1994.PubMed/NCBI
|
39
|
Shaulian E and Karin M: AP-1 in cell
proliferation and survival. Oncogene. 20:2390–2400. 2001.
View Article : Google Scholar : PubMed/NCBI
|
40
|
Huang LC, Clarkin KC and Wahl GM:
Sensitivity and selectivity of the DNA damage sensor responsible
for activating p53-dependent G1 arrest. Proc Natl Acad
Sci USA. 93:4827–4832. 1996. View Article : Google Scholar
|