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<front>
<journal-meta>
<journal-id journal-id-type="publisher-id">ETM</journal-id>
<journal-title-group>
<journal-title>Experimental and Therapeutic Medicine</journal-title>
</journal-title-group>
<issn pub-type="ppub">1792-0981</issn>
<issn pub-type="epub">1792-1015</issn>
<publisher>
<publisher-name>D.A. Spandidos</publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id pub-id-type="doi">10.3892/etm.2026.13238</article-id>
<article-id pub-id-type="publisher-id">ETM-32-3-13238</article-id>
<article-categories>
<subj-group>
<subject>Correction</subject>
</subj-group>
</article-categories>
<title-group>
<article-title>[Corrigendum] Endogenous Nampt upregulation is associated with diabetic nephropathy inflammatory-fibrosis through the NF-&#x03BA;B p65 and Sirt1 pathway; NMN alleviates diabetic nephropathy inflammatory-fibrosis by inhibiting endogenous Nampt</article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name><surname>Chen</surname><given-names>Ye</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Liang</surname><given-names>Yuzhen</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Hu</surname><given-names>Tingting</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Wei</surname><given-names>Riming</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Cai</surname><given-names>Congjie</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Wang</surname><given-names>Ping</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Wang</surname><given-names>Lingyu</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Qiao</surname><given-names>Wei</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Feng</surname><given-names>Leping</given-names></name>
</contrib>
</contrib-group>
<pub-date pub-type="collection"><month>09</month><year>2026</year></pub-date>
<pub-date pub-type="epub"><day>08</day><month>07</month><year>2026</year></pub-date>
<volume>32</volume>
<issue>3</issue>
<elocation-id>243</elocation-id>
<permissions>
<copyright-statement>Copyright: &#x00A9; 2026 Chen et al.</copyright-statement>
<copyright-year>2026</copyright-year>
<license license-type="open-access">
<license-p>This is an open access article distributed under the terms of the <ext-link ext-link-type="uri" xlink:href="https://creativecommons.org/licenses/by/4.0/">Creative Commons Attribution License</ext-link>, which permits unrestricted use, distribution, reproduction and adaptation in any medium and for any purpose provided that it is properly attributed. For attribution, the original author(s), title, publication source (PeerJ) and either DOI or URL of the article must be cited.</license-p></license>
</permissions>
</article-meta>
</front>
<body>
<p>Exp Ther Med 14:<related-article id="RA1" related-article-type="corrected-article" vol="14" page="4181" ext-link-type="doi" xlink:href="10.3892/etm.2017.5098">4181&#x2013;4193</related-article>, 2017; DOI: 10.3892/etm.2017.5098</p>
<p>Following the publication of this paper, it was drawn to the Editor&#x2019;s attention by a concerned reader that the Sirt1 western blot data shown in Fig. 4A on p. 4187 were strikingly similar to the Sirt1 western blot data shown in <xref rid="f6-ETM-32-3-13238" ref-type="fig">Fig. 6A</xref> on p. 4188, even though the experimental conditions reported for these figure parts were apparently different.</p>
<p>Upon examining the original data in their paper, the authors have realized that the Sirt1 data correctly featured in Fig. 4A had inadvertently been re-used in <xref rid="f6-ETM-32-3-13238" ref-type="fig">Fig. 6A</xref>. The authors have repeated the western blots for Sirt1 protein in <xref rid="f6-ETM-32-3-13238" ref-type="fig">Fig. 6A</xref>, and the results obtained were broadly similar to those obtained originally. The revised version of <xref rid="f6-ETM-32-3-13238" ref-type="fig">Fig. 6</xref>, featuring the new data for Sirt1 in <xref rid="f6-ETM-32-3-13238" ref-type="fig">Fig. 6A</xref>, is shown below. All the authors agree to the publication of this Corrigendum, and are grateful to the Editor of <italic>Experimental and Therapeutic Medicine</italic> for granting them the opportunity to publish this; moreover, they apologize to the readership for any inconvenience caused.</p>
</body>
<floats-group>
<fig id="f6-ETM-32-3-13238" position="float">
<label>Figure 6</label>
<caption><p>(A) Time course of endogenous Nampt, NF-&#x03BA;B p65, Sirt1, FoxO1, and Bax protein expression in HBZY-1 cells exposed to a high-glucose milieu for 24, 48, 72, or 96 h. Quantification of western blot analysis indicated the protein expression levels of (B) Nampt, (C) NF-&#x03BA;B p65, (D) Sirt1, (E) FoxO1 and (F) Bax, and &#x03B2;-actin were indicated. Data are presented as means &#x00B1; standard deviations of three experiments. <sup>&#x0023;</sup>P&lt;0.05, <sup>&#x0023;&#x0023;</sup>P&lt;0.01 vs. 24 h (control). Nampt, nicotinamide phosphoribosyltransferase; NF, nuclear factor; Sirt1, sirtuin 1; FoxO1, forkhead box protein O1; Bax, B-cell lymphoma 2-like protein 4.</p></caption>
<graphic xlink:href="etm-32-03-13238-g00.tif"/>
</fig>
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</article>
