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<front>
<journal-meta>
<journal-id journal-id-type="publisher-id">ETM</journal-id>
<journal-title-group>
<journal-title>Experimental and Therapeutic Medicine</journal-title>
</journal-title-group>
<issn pub-type="ppub">1792-0981</issn>
<issn pub-type="epub">1792-1015</issn>
<publisher>
<publisher-name>D.A. Spandidos</publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id pub-id-type="doi">10.3892/etm.2026.13330</article-id>
<article-id pub-id-type="publisher-id">ETM-32-6-13330</article-id>
<article-categories>
<subj-group>
<subject>Expression of Concern</subject>
</subj-group>
</article-categories>
<title-group>
<article-title>[Expression of Concern] UCHL1 regulated by Sp1 ameliorates cochlear hair cell senescence and oxidative damage</article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name><surname>Li</surname><given-names>Lihua</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Xu</surname><given-names>Kai</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Bai</surname><given-names>Xue</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Wang</surname><given-names>Zhi</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Tian</surname><given-names>Xiaoyan</given-names></name>
</contrib>
<contrib contrib-type="author">
<name><surname>Chen</surname><given-names>Xubo</given-names></name>
</contrib>
</contrib-group>
<pub-date pub-type="collection"><month>12</month><year>2026</year></pub-date>
<pub-date pub-type="epub"><day>08</day><month>10</month><year>2026</year></pub-date>
<volume>32</volume>
<issue>6</issue>
<elocation-id>335</elocation-id>
<permissions>
<copyright-statement>Copyright: &#x00A9; 2026 Li et al.</copyright-statement>
<copyright-year>2026</copyright-year>
<license license-type="open-access">
<license-p>This is an open access article distributed under the terms of the <ext-link ext-link-type="uri" xlink:href="https://creativecommons.org/licenses/by/4.0/">Creative Commons Attribution License</ext-link>, which permits unrestricted use, distribution, reproduction and adaptation in any medium and for any purpose provided that it is properly attributed. For attribution, the original author(s), title, publication source (PeerJ) and either DOI or URL of the article must be cited.</license-p></license>
</permissions>
</article-meta>
</front>
<body>
<p>Exp Ther Med 25:<related-article id="RA1" related-article-type="object-of-concern" vol="25" ext-link-type="doi" xlink:href="10.3892/etm.2023.11793">94</related-article>, 2023; DOI: 10.3892/etm.2023.11793</p>
<p>Following the publication of this paper, it was drawn to the Editor&#x2019;s attention by a concerned reader that the one of the antibodies the authors used in their study was apparently selected inappropriately: As it relates to the western blot experiments shown in Figs. 3B and 5F, the antibody (cat. no. ab51243) that the authors reported to have used for the apoptosis-associated protein p16-INK4a was in fact specific for the protein known as p16 ARC (encoded by the ARPC5 gene), not as the authors had intended.</p>
<p>The authors have been contacted by the Editorial Office to offer an explanation for the apparently incorrect selection of this antibody in their paper, and we are awaiting their response. Owing to the fact that the Editorial Office has been made aware of a potential issue surrounding the scientific integrity of this paper, we are issuing an Expression of Concern to notify readers of this potential problem while the Editorial Office continues to investigate this matter further.</p>
</body>
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