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  2. Mutation update: TGFBI pathogenic and likely pathogenic variants in corneal dystrophies
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  3. Genetic predisposition to ocular surface disorders and opportunities for gene-based therapies
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  4. SUMOylation activates large tumour suppressor 1 to maintain the tissue homeostasis during Hippo signalling
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  5. Biochemical mechanisms of aggregation in TGFBI-linked corneal dystrophies
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  6. Corneal Dystrophy in Dutch Belted Rabbits as a Possible Model of Thiel-Behnke Subtype of Epithelial-Stromal TGFβ-Induced Corneal Dystrophy
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