1. O-Linked β-N-acetylglucosamine (O-GlcNAc) modification: a new pathway to decode pathogenesis of diabetic retinopathy
   Zafer Gurel et al, 2018, Clinical Science CrossRef
2. Angiotensin-(1–7) Attenuates Protein O-GlcNAcylation in the Retina by EPAC/Rap1-Dependent Inhibition of O-GlcNAc Transferase
   Sadie K. Dierschke et al, 2020, Investigative Opthalmology & Visual Science CrossRef
3. O-GlcNAc Modification and Its Role in Diabetic Retinopathy
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4. Potential Role of Cyr61 Induced Degeneration of Human Müller Cells in Diabetic Retinopathy
   Fen Zhou et al, 2014, PLoS ONE CrossRef
5. Consumption of a high fat diet promotes protein O-GlcNAcylation in mouse retina via NR4A1-dependent GFAT2 expression
   Weiwei Dai et al, 2018, Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease CrossRef
6. Retinal Protein O-GlcNAcylation and the Ocular Renin-angiotensin System: Signaling Cross-roads in Diabetic Retinopathy
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7. O-GlcNAcylation promotes astroglial-mesenchymal transition via the connexin43 pathway under high-glucose conditions
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8. The O-Linked N-Acetylglucosamine Containing Epitope H (O-GlcNAcH) is Upregulated in the Trophoblastic and Downregulated in the Fibroblastic Cells in Missed Miscarriage Human Chorionic Villi With Simple Hydropic Degeneration
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9. Consequences of Dicarbonyl Stress on Skeletal Muscle Proteins in Type 2 Diabetes
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10. O‐GlcNAcylation Mediates Glucose‐Induced Alterations in Endothelial Cell Phenotype in Human Diabetes Mellitus
    Nobuyuki Masaki et al, 2020, Journal of the American Heart Association CrossRef
11. Contribution of the hexosamine biosynthetic pathway in the hyperglycemia-dependent and -independent breakdown of the retinal neurovascular unit
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12. The augmentation of O-GlcNAcylation reduces glyoxal-induced cell injury by attenuating oxidative stress in human retinal microvascular endothelial cells
    GUO DONG LIU et al, 2015, International Journal of Molecular Medicine CrossRef
13. Proteomic analysis of diabetic retinas
    Christopher R. Starr et al, 2023, Frontiers in Endocrinology CrossRef
14. Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cells
    Chang Hyun Byon et al, 2016, Redox Biology CrossRef
15. O-GlcNAc transferase inhibitors: current tools and future challenges
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16. The intersection of HIF-1α, O-GlcNAc, and skeletal muscle loss in chronic obstructive pulmonary disease
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17. Validation of RT-qPCR reference genes and determination of Robo4 expression levels in human retinal endothelial cells under hypoxia and/or hyperglycemia
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18. Posttranslational modifications of proteins in diseased retina
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19. Identification of connexin43 in diabetic retinopathy and its downregulation by O-GlcNAcylation to inhibit the activation of glial cells
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20. Increased O-GlcNAcylation of NF-κB Enhances Retinal Ganglion Cell Death in Streptozotocin-induced Diabetic Retinopathy
    Seong-Jae Kim et al, 2016, Current Eye Research CrossRef
21. O‐GlcNAc Transferase: Structural Characteristics, Catalytic Mechanism and Small‐Molecule Inhibitors
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22. Multi-omics in exploring the pathophysiology of diabetic retinopathy
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23. O-GlcNAcylation Inhibition Upregulates Connexin43 Expression in the Endothelium to Protect the Tight Junction Barrier in Diabetic Retinopathy
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24. O-GlcNAcylation of Orphan Nuclear Receptor Estrogen-Related Receptor γ Promotes Hepatic Gluconeogenesis
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25. Hypothermia mediates age-dependent increase of tau phosphorylation in db/db mice
    Noura B. El Khoury et al, 2016, Neurobiology of Disease CrossRef