1. SUMO1/UBC9‑decreased Nox1 activity inhibits reactive oxygen species generation and apoptosis in diabetic retinopathy
   Jiaoli Hu et al, 2017, Molecular Medicine Reports CrossRef
2. O-GlcNAc / Akt pathway regulates glucose metabolism and reduces apoptosis in liver of piglets with acute cold stress
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3. Identification of connexin43 in diabetic retinopathy and its downregulation by O-GlcNAcylation to inhibit the activation of glial cells
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4. Galectin-1 expression imprints a neurovascular phenotype in proliferative retinopathies and delineates responses to anti-VEGF
   Magali E. Ridano et al, 2017, Oncotarget CrossRef
5. Consequences of Dicarbonyl Stress on Skeletal Muscle Proteins in Type 2 Diabetes
   Khurshid Ahmad et al, 2020, Current Protein & Peptide Science CrossRef
6. Nuclear factor-erythroid-2 related transcription factor-1 (Nrf1) is regulated by O-GlcNAc transferase
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7. O-GlcNAcylation Inhibition Upregulates Connexin43 Expression in the Endothelium to Protect the Tight Junction Barrier in Diabetic Retinopathy
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8. Activation of AMPK is neuroprotective in the oxidative stress by advanced glycosylation end products in human neural stem cells
   Chien-Hung Lin et al, 2017, Experimental Cell Research CrossRef
9. O-GlcNAcylation and Diabetes
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10. Contribution of the hexosamine biosynthetic pathway in the hyperglycemia-dependent and -independent breakdown of the retinal neurovascular unit
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11. Impact of Advanced Glycation End Products on Endothelial Function and Their Potential Link to Atherosclerosis
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12. Stress-induced O-GlcNAcylation: an adaptive process of injured cells
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13. Methylglyoxal induces retinopathy‐type lesions in the absence of hyperglycemia: studies in a rat model
    Andrea Schlotterer et al, 2019, The FASEB Journal CrossRef
14. RUNX1 can mediate the glucose and O-GlcNAc-driven proliferation and migration of human retinal microvascular endothelial cells
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15. Advanced Glycation End-Products Induce Apoptosis of Vascular Smooth Muscle Cells: A Mechanism for Vascular Calcification
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16. Glycation and Glycosylation in Cardiovascular Remodeling: Focus on Advanced Glycation End Products and O-Linked Glycosylations as Glucose-Related Pathogenetic Factors and Disease Markers
    Elena Dozio et al, 2021, Journal of Clinical Medicine CrossRef
17. O-GlcNAc Modification and Its Role in Diabetic Retinopathy
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18. O-Linked β-N-acetylglucosamine (O-GlcNAc) modification: a new pathway to decode pathogenesis of diabetic retinopathy
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19. Gastrodin inhibits high glucose‑induced human retinal endothelial cell apoptosis by regulating the SIRT1/TLR4/NF‑κBp65 signaling pathway
    Tong‑He Zhang et al, 2018, Molecular Medicine Reports CrossRef
20. The Role of Stress-Induced O-GlcNAc Protein Modification in the Regulation of Membrane Transport
    Viktória Fisi et al, 2017, Oxidative Medicine and Cellular Longevity CrossRef
21. Posttranslational modifications of proteins in diseased retina
    Christopher R. Starr et al, 2023, Frontiers in Cellular Neuroscience CrossRef
22. O-GlcNAcylation and Its Roles in Neurodegenerative Diseases
    Pengyang Du et al, 2024, Journal of Alzheimer's Disease CrossRef