1. Genomics of Human Fibrotic Diseases: Disordered Wound Healing Response
   Rivka C. Stone et al, 2020, International Journal of Molecular Sciences CrossRef
2. Long non-coding RNA H19 acts as a microRNA-194 sponge to inhibit the apoptosis and promote the proliferation of hypertrophic scar fibroblasts
   Bo Liu et al, 2021, Canadian Journal of Physiology and Pharmacology CrossRef
3. LncRNA TRHDE-AS1 inhibit the scar fibroblasts proliferation via miR-181a-5p/PTEN axis
   Yanping Wei et al, 2021, Journal of Molecular Histology CrossRef
4. Knockdown of lncRNA‑NEAT1 expression inhibits hypoxia‑induced scar fibroblast proliferation through regulation of the miR‑488‑3p/COL3A1 axis
   Huan Xu et al, 2022, Experimental and Therapeutic Medicine CrossRef
5. lncRNA PAPPA-AS1 Induces the Development of Hypertrophic Scar by Upregulating TLR4 through Interacting with TAF15
   Pengju Fan et al, 2021, Mediators of Inflammation CrossRef
6. LncRNA TUG1 exhibits pro-fibrosis activity in hypertrophic scar through TAK1/YAP/TAZ pathway via miR-27b-3p
   Xian-Min Li et al, 2021, Molecular and Cellular Biochemistry CrossRef
7. Current Approaches Targeting the Wound Healing Phases to Attenuate Fibrosis and Scarring
   Amina El Ayadi et al, 2020, International Journal of Molecular Sciences CrossRef
8. The Need for Basic, Translational, and Clinical Research in the Field of Hypertrophic Scars
   Bonnie C. Carney et al, 2022, Recent Advances in Wound Healing CrossRef