1. miRNA signatures in diabetic retinopathy and nephropathy: delineating underlying mechanisms
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2. Sox6, A Potential Target for MicroRNAs in Cardiometabolic Disease
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4. Role of Royal Jelly Treated Adipose-Derived Stem Cell-Extracellular Vesicles on Fibroblast Proliferation, Migration, and Collagen Production
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5. Regulation of Ptch1 by miR-342-5p and FoxO3 Induced Autophagy Involved in Renal Fibrosis
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6. CircHOMER1 aggravates oxidative stress, inflammation and extracellular matrix deposition in high glucose‐induced human mesangial cells
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7. Human Umbilical Cord Mesenchymal Stem Cells Inhibit Pyroptosis of Renal Tubular Epithelial Cells through miR-342-3p/Caspase1 Signaling Pathway in Diabetic Nephropathy
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10. miR-362-3p Targets Orosomucoid 1 to Promote Cell Proliferation, Restrain Cell Apoptosis and Thereby Mitigate Hypoxia/Reoxygenation-Induced Cardiomyocytes Injury
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12. Non-Coding RNAs as Biomarkers and Therapeutic Targets for Diabetic Kidney Disease
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13. To the Future: The Role of Exosome-Derived microRNAs as Markers, Mediators, and Therapies for Endothelial Dysfunction in Type 2 Diabetes Mellitus
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14. The intellectual base and global trends in inflammation of diabetic kidney disease: a bibliometric analysis
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15. Identification of miRNA Signature Associated With Erectile Dysfunction in Type 2 Diabetes Mellitus by Support Vector Machine-Recursive Feature Elimination
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    Roberta Resaz et al, 2021, International Journal of Molecular Sciences CrossRef
18. MiR-27b-3p inhibits the progression of renal fibrosis via suppressing STAT1
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19. Markers of end-stage renal disease progression: beyond the GFR
    E. I Yakupova et al, 2023, Биохимия CrossRef
20. Transcriptome expression profiles associated with diabetic nephropathy development
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21. Effect of miR-499-5p/SOX6 axis on atrial fibrosis in rats with atrial fibrillation
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23. Knockdown of lncRNA NORAD inhibits the proliferation, inflammation and fibrosis of human mesangial cells under high‑glucose conditions by regulating the miR‑485/NRF1 axis
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24. lncRNA KCNQ1OT1 regulated high glucose-induced proliferation, oxidative stress, extracellular matrix accumulation, and inflammation by miR-147a/SOX6 in diabetic nephropathy (DN)
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