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  2. Psoriasis Comorbidities and Shared Disease Mechanisms – An Investigation using Systems Biology approaches
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  3. Based on Gene Expression Analysis: Low-Density Neutrophil Expression Is a Characteristic of the Fast Responders Treated With Guselkumab for Psoriasis
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  4. Mesenchymal stem/stromal cells primed by inflammatory cytokines alleviate psoriasis-like inflammation via the TSG-6-neutrophil axis
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  5. Mass Spectrometry-Based Proteomics Analysis Unveils PTPRS Inhibits Proliferation and Inflammatory Response of Keratinocytes in Psoriasis
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  6. Inflammation modulates intercellular adhesion and mechanotransduction in human epidermis via ROCK2
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  9. Construction of a three commitment points for S phase entry cell cycle model and immune-related ceRNA network to explore novel therapeutic options for psoriasis
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  10. Considerations of the Genetic Background of Obesity among Patients with Psoriasis
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  11. Metabolic Network Modeling for Rational Drug Design against Candida albicans
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  12. TNF-α promotes CXCL-1/8 production in keratinocytes by downregulating galectin-3 through NF-κB and hsa-miR-27a-3p pathway to contribute psoriasis development
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  13. Comparative study on gene expression in psoriatic lesions versus chronic wound healing processes
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  14. CCNB1 and CCNB2 involvement in the pathogenesis of psoriasis: a bioinformatics study
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  15. Longitudinal change in CDC42 in psoriasis: correlation with disease activity and treatment response
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