1. Enzalutamide-induced signatures revealed by epigenetic plasticity using single-cell multi-omics sequencing in prostate cancer
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  2. Compounds from Cynomorium songaricum with Estrogenic and Androgenic Activities Suppress the Oestrogen/Androgen-Induced BPH Process
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  3. Overexpression of LeMYB1 enhances shikonin formation by up-regulating key shikonin biosynthesis-related genes in Lithospermum erythrorhizon
    H. Zhao et al, 2015, Biologia plantarum CrossRef
  4. Shikonin causes apoptosis by up-regulating p73 and down-regulating ICBP90 in human cancer cells
    Soon Young Jang et al, 2015, Biochemical and Biophysical Research Communications CrossRef
  5. Shikonin derivatives for cancer prevention and therapy
    Joelle C. Boulos et al, 2019, Cancer Letters CrossRef
  6. Acetylamine derivative of diospyrin, a plant-derived binaphthylquinonoid, inhibits human colon cancer growth in Nod-Scid mice
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  7. The effect of Arnebia purpurea extract on the survival of random pattern skin flaps in rats
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  8. Experimental Study of Hepatocellular Carcinoma Treatment by Shikonin Through Regulating PKM2


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  9. Antagonism of androgen receptor signaling by aloe-emodin
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  10. Overexpression of pregnane X and glucocorticoid receptors and the regulation of cytochrome P450 in human epileptic brain endothelial cells
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  11. Shikonin, dually functions as a proteasome inhibitor and a necroptosis inducer in multiple myeloma cells
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  12. Shikonin inhibits TNF-α-induced growth and invasion of rat aortic vascular smooth muscle cells
    Xuemin Zhang et al, 2015, Canadian Journal of Physiology and Pharmacology CrossRef
  13. Glucose-regulated protein 78 mediates the anticancer efficacy of shikonin in hormone-refractory prostate cancer cells
    Li-Jen Kuo et al, 2015, Tumor Biology CrossRef
  14. Carbohydrate metabolism in prostate cancer
    Tomas Koltai et al, 2021, Prostate Cancer Metabolism CrossRef
  15. Shikonin Reduces Growth of Docetaxel-Resistant Prostate Cancer Cells Mainly through Necroptosis
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  16. Shikonin induces apoptosis and inhibits migration of ovarian carcinoma cells by inhibiting the phosphorylation of Src and FAK
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  17. Revisiting prostate cancer metabolism: From metabolites to disease and therapy
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