1. IGF-I does not prevent myotube atrophy caused by proinflammatory cytokines despite activation of Akt/Foxo and GSK-3β pathways and inhibition of atrogin-1 mRNA
    Mischaël Dehoux et al, 2007, American Journal of Physiology-Endocrinology and Metabolism CrossRef
  2. Role of Renin-Angiotensin System in Cancer Cachexia
    Vivek Bora et al, 2023, The Renin Angiotensin System in Cancer, Lung, Liver and Infectious Diseases CrossRef
  3. Assessment of Tumor Necrosis Factor Alpha Blockade As an Intervention to Improve Tolerability of Dose-Intensive Chemotherapy in Cancer Patients
    J. Paul Monk et al, 2006, Journal of Clinical Oncology CrossRef
  4. Cancer Cachexia Signaling Pathways Continue to Emerge Yet Much Still Points to the Proteasome
    Swarnali Acharyya et al, 2007, Clinical Cancer Research CrossRef
  5. MicroRNA-mRNA Co-sequencing Identifies Transcriptional and Post-transcriptional Regulatory Networks Underlying Muscle Wasting in Cancer Cachexia
    Geysson Javier Fernandez et al, 2020, Frontiers in Genetics CrossRef
  6. Are Peroxisome Proliferator-Activated Receptors Involved in Skeletal Muscle Wasting during Experimental Cancer Cachexia? Role of β2-Adrenergic Agonists
    Gemma Fuster et al, 2007, Cancer Research CrossRef
  7. IGF-1 is downregulated in experimental cancer cachexia
    Paola Costelli et al, 2006, American Journal of Physiology-Regulatory, Integrative and Comparative Physiology CrossRef