1. Autophagy and necroptosis in cisplatin-induced acute kidney injury: Recent advances regarding their role and therapeutic potential
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2. Macrophage angiotensin AT2 receptor activation is protective against early phases of LPS-induced acute kidney injury
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3. Effect of forkhead box O1 in renal tubular epithelial cells on endotoxin-induced acute kidney injury
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4. Mitochondrial dysfunction in kidney injury, inflammation, and disease: Potential therapeutic approaches
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5. Extracellular Vesicles From High Glucose-Treated Podocytes Induce Apoptosis of Proximal Tubular Epithelial Cells
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6. Regulated necrosis role in inflammation and repair in acute kidney injury
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7. Protective Effects of Simvastatin on Endotoxin-Induced Acute Kidney Injury through Activation of Tubular Epithelial Cells’ Survival and Hindering Cytochrome C-Mediated Apoptosis
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