1. Exposure to Maternal Immune Activation Causes Congenital Unfolded Protein Response Defects and Increases the Susceptibility to Postnatal Inflammatory Stimulation in Offspring
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  2. Molecular Insight Into the IRE1α-Mediated Type I Interferon Response Induced by Proteasome Impairment in Myeloid Cells of the Brain
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  3. Bromodomain-containing Protein 4 regulates innate inflammation via modulation of alternative splicing
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  4. Dual role of Endoplasmic Reticulum Stress-Mediated Unfolded Protein Response Signaling Pathway in Carcinogenesis
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  5. Roles of XBP1s in Transcriptional Regulation of Target Genes
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  7. HBV preS Mutations Promote Hepatocarcinogenesis by Inducing Endoplasmic Reticulum Stress and Upregulating Inflammatory Signaling
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  8. Unraveling the Molecular Nexus between GPCRs, ERS, and EMT
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  9. XBP1- IGFBP3 Signaling Pathway Promotes NSCLC Invasion and Metastasis
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