TY - JOUR AB - Following the publication of this article, an interested reader drew to our attention an anomaly associated with the presentation of Figs. 5 and 8; essentially, the β-actin control bands in Fig. 8 appeared to be a mirror image of the same control bands shown in Fig. 5A. After having re‑examined the data, Fig. 5 was correctly presented as shown in the paper, although the β-actin blot in Fig. 8 had been inadvertently misplaced as a mirror image of the control data from Fig. 5. Two similar experiments were performed at that time, yielding similar results, and the error with the control data did not affect the second of the experiments. Therefore, we present a corrected version of Fig. 8 containing the alternative data. The findings and conclusions of this paper are still supported by our experimental data, and were not affected by this error. We sincerely apologize for this mistake, and thank the reader of our article who drew this matter to our attention. Furthermore, we regret any inconvenience this mistake has caused. [the original article was published in the International Journal of Oncology 42: 1113-1119, 2013; DOI: 10.3892/ijo.2013.1789] AD - Department of Veterinary Medicine, School of Veterinary Medicine, National Taiwan University, Taipei, Taiwan, R.O.C. Department of Surgery, Mackay Memorial Hospital, Taipei, Taiwan, R.O.C. Research Institute for Children, Children's Hospital, New Orleans, LA, USA AU - Chen,Chiachen AU - Chang,Yuan-Ching AU - Lan,Michael ,S. AU - Breslin,Mary DA - 2016/08/01 DO - 10.3892/ijo.2016.3564 EP - 847 IS - 2 JO - Int J Oncol KW - leptin ovarian cancer cell growth anti-apoptotic effect JAK2-MEK/ERK1/2 pathway PI3K/Akt pathway PY - 2016 SN - 1019-6439 1791-2423 SP - 847 ST - [Corrigendum] Leptin stimulates ovarian cancer cell growth and inhibits apoptosis by increasing cyclin D1 and Mcl-1 expression via the activation of the MEK/ERK1/2 and PI3K/Akt signaling pathways T2 - International Journal of Oncology TI - [Corrigendum] Leptin stimulates ovarian cancer cell growth and inhibits apoptosis by increasing cyclin D1 and Mcl-1 expression via the activation of the MEK/ERK1/2 and PI3K/Akt signaling pathways UR - https://doi.org/10.3892/ijo.2016.3564 VL - 49 ER -