Related Articles
Role of transforming growth factor β‑1 in the pathogenesis of pelvic organ prolapse: A potential therapeutic target
Advanced glycation end products decrease collagen I levels in fibroblasts from the vaginal wall of patients with POP via the RAGE, MAPK and NF-κB pathways
Extracellular matrix metabolism disorder induced by mechanical strain on human parametrial ligament fibroblasts
Collagen metabolic disorder induced by oxidative stress in human uterosacral ligament‑derived fibroblasts: A possible pathophysiological mechanism in pelvic organ prolapse
Excess mechanical stress and hydrogen peroxide remodel extracellular matrix of cultured human uterosacral ligament fibroblasts by disturbing the balance of MMPs/TIMPs via the regulation of TGF‑β1 signaling pathway