1
|
Martinez C, Cohen AT, Bamber L and
Rietbrock S: Epidemiology of first and recurrent venous
thromboembolism: A population-based cohort study in patients
without active cancer. Thromb Haemost. 112:255–263. 2014.
View Article : Google Scholar : PubMed/NCBI
|
2
|
Monreal M, Barba R, Tolosa C, et al: Deep
vein thrombosis and pulmonary embolism: the same disease?
Pathophysiol Haemost Thromb. 35:133–135. 2006. View Article : Google Scholar : PubMed/NCBI
|
3
|
Nielsen JD: The incidence of pulmonary
embolism during deep vein thrombosis. Phlebology. 28:29–33. 2013.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Blättler W and Partsch H: Leg compression
and ambulation is better than bed rest for the treatment of acute
deep venous thrombosis. Int J Angiol. 22:393–400. 2003.
|
5
|
Malone PC and Agutter PS: The aetiology of
deep venous thrombosis. QJM. 99:581–593. 2006. View Article : Google Scholar : PubMed/NCBI
|
6
|
Schulz C, Engelmann B and Massberg S:
Crossroads of coagulation and innate immunity: the case of deep
vein thrombosis. J Thromb Haemost. 11:233–241. 2013. View Article : Google Scholar : PubMed/NCBI
|
7
|
Watson SP: Platelet activation by
extracellular matrix proteins in haemostasis and thrombosis. Curr
Pharm Des. 15:1358–1372. 2009. View Article : Google Scholar : PubMed/NCBI
|
8
|
Brill A, Fuchs TA, Chauhan AK, et al: von
Willebrand factor-mediated platelet adhesion is critical for deep
vein thrombosisin mouse models. Blood. 117:1400–1407. 2011.
View Article : Google Scholar :
|
9
|
von Brühl ML, Stark K, Steinhart A, et al:
Monocytes, neutrophils, and platelets cooperate to initiate and
propagate venous thrombosis in mice in vivo. J Exp Med.
209:819–835. 2012. View Article : Google Scholar : PubMed/NCBI
|
10
|
Jones CI, Moraes LA and Gibbins JM:
Regulation of platelet biology by platelet endothelial cell
adhesion molecule-1. Platelets. 23:331–335. 2012. View Article : Google Scholar
|
11
|
Berman ME and Muller WA: Ligation of
platelet/endothelial cell adhesion molecule 1 (PECAM-1/CD31) on
monocytes and neutrophils increases binding capacity of leukocyte
CR3 (CD11b/CD18). J Immunol. 154:299–307. 1995.PubMed/NCBI
|
12
|
Cicmil M, Thomas JM, Leduc M, Bon C and
Gibbins JM: Platelet endothelial cell adhesion molecule-1 signaling
inhibits the activation of human platelets. Blood. 99:137–144.
2002. View Article : Google Scholar : PubMed/NCBI
|
13
|
Flynn KM, Michaud M, Canosa S and Madri
JA: CD44 regulates vascular endothelial barrier integrity via a
PECAM-1 dependent mechanism. Angiogenesis. 16:689–705. 2013.
View Article : Google Scholar : PubMed/NCBI
|
14
|
Kellermair J, Redwan B, Alias S, et al:
Platelet endothelial cell adhesion molecule 1 deficiency misguides
venous thrombus resolution. Blood. 122:3376–3384. 2013. View Article : Google Scholar : PubMed/NCBI
|
15
|
Robbins FM and Hartzman RJ: CD31/PECAM-1
genotyping and haplotype analyses show population diversity. Tissue
Antigens. 69:28–37. 2007. View Article : Google Scholar : PubMed/NCBI
|
16
|
Novinska MS, Pietz BC, Ellis TM, Newman DK
and Newman PJ: The alleles of PECAM-1. Gene. 376:95–101. 2006.
View Article : Google Scholar : PubMed/NCBI
|
17
|
Listì F, Caruso C, Di Carlo D, et al:
Association between platelet endothelial cellular adhesion
molecule-1 polymorphisms and atherosclerosis: results of a study on
patients from northern Italy. Rejuvenation Res. 13:237–241. 2010.
View Article : Google Scholar : PubMed/NCBI
|
18
|
Sahebkar A, Morris DR, Biros E and
Golledge J: Association of single nucleotide polymorphisms in the
gene encoding platelet endothelial cell adhesion molecule-1 with
the risk of myocardial infarction: a systematic review and
meta-analysis. Thromb Res. 132:227–233. 2013. View Article : Google Scholar : PubMed/NCBI
|
19
|
Wei YS, Lan Y, Liu YG, Meng LQ, Xu QQ and
Xie HY: Platelet-endothelial cell adhesion molecule-1 gene
polymorphism and its soluble level are associated with ischemic
stroke. DNA Cell Biol. 28:151–158. 2009. View Article : Google Scholar : PubMed/NCBI
|
20
|
Agnelli G, Gallus A, Goldhaber SZ, et al
ODIXa-DVT Study Investigators: Treatment of proximal deep-vein
thrombosis with the oral direct factor Xa inhibitor rivaroxaban
(BAY 59-7939): the ODIXa-DVT (Oral Direct Factor Xa Inhibitor BAY
59-7939 in Patients With Acute Symptomatic Deep-Vein Thrombosis)
study. Circulation. 116:180–187. 2007. View Article : Google Scholar : PubMed/NCBI
|
21
|
Wolberg AS, Aleman MM, Leiderman K and
Machlus KR: Procoagulant activity in hemostasis and thrombosis:
Virchow’s triad revisited. Anesth Analg. 114:275–285. 2012.
View Article : Google Scholar :
|
22
|
Hasstedt SJ, Bezemer ID, Callas PW, et al:
Cell adhesion molecule 1: a novel risk factor for venous
thrombosis. Blood. 114:3084–3091. 2009. View Article : Google Scholar : PubMed/NCBI
|
23
|
Jones CI, Garner SF, Moraes LA, et al:
PECAM-1 expression and activity negatively regulate multiple
platelet signaling pathways. FEBS Lett. 583:3618–3624. 2009.
View Article : Google Scholar : PubMed/NCBI
|
24
|
Fang L, Wei H, Chowdhury SH, et al:
Association of Leu125Val polymorphism of platelet endothelial cell
adhesion molecule-1 (PECAM-1) gene & soluble level of PECAM-1
with coronary artery disease in Asian Indians. Indian J Med Res.
121:92–99. 2005.PubMed/NCBI
|
25
|
Goldberger A, Middleton KA, Oliver JA, et
al: Biosynthesis and processing of the cell adhesion molecule
PECAM-1 includes production of a soluble form. J Biol Chem.
269:17183–17191. 1994.PubMed/NCBI
|
26
|
Fornasa G, Groyer E, Clement M, et al: TCR
stimulation drives cleavage and shedding of the ITIM receptor CD31.
J Immunol. 184:5485–5492. 2010. View Article : Google Scholar : PubMed/NCBI
|
27
|
Moraes LA, Vaiyapuri S, Sasikumar P, et
al: Antithrombotic actions of statins involve PECAM-1 signaling.
Blood. 122:3188–3196. 2013. View Article : Google Scholar : PubMed/NCBI
|
28
|
Eugenin EA, Gamss R, Buckner C, et al:
Shedding of PECAM-1 during HIV infection: a potential role for
soluble PECAM-1 in the pathogenesis of NeuroAIDS. J Leukoc Biol.
79:444–452. 2006. View Article : Google Scholar : PubMed/NCBI
|
29
|
Liao F, Ali J, Greene T and Muller WA:
Soluble domain 1 of platelet-endothelial cell adhesion molecule
(PECAM) is sufficient to block transendothelial migration in vitro
and in vivo. J Exp Med. 185:1349–1357. 1997. View Article : Google Scholar : PubMed/NCBI
|
30
|
Pepene CE: Soluble platelet/endothelial
cell adhesion molecule (sPECAM)-1 is increased in polycystic ovary
syndrome and related to endothelial dysfunction. Gynecol
Endocrinol. 28:370–374. 2012. View Article : Google Scholar : PubMed/NCBI
|
31
|
Okoroh EM, Hooper WC, Atrash HK, Yusuf HR
and Boulet SL: Is polycystic ovary syndrome another risk factor for
venous thromboembolism? United States 2003–2008. Am J Obstet
Gynecol. 207:377.e1–e8. 2012. View Article : Google Scholar
|
32
|
Falati S, Patil S, Gross PL, et al:
Platelet PECAM-1 inhibits thrombus formation in vivo. Blood.
107:535–541. 2006. View Article : Google Scholar
|
33
|
Privratsky JR, Paddock CM, Florey O,
Newman DK, Muller WA and Newman PJ: Relative contribution of
PECAM-1 adhesion and signaling to the maintenance of vascular
integrity. J Cell Sci. 124:1477–1485. 2011. View Article : Google Scholar : PubMed/NCBI
|
34
|
Kahn SR: Post-thrombotic syndrome after
deep venous thrombosis: risk factors, prevention, and therapeutic
options. Clin Adv Hematol Oncol. 7:433–435. 2009.PubMed/NCBI
|