1
|
Jemal A, Bray F, Center MM, et al: Global
cancer statistics. CA Cancer J Clin. 61:69–90. 2011. View Article : Google Scholar : PubMed/NCBI
|
2
|
Chan AT: Current treatment of
nasopharyngeal carcinoma. Eur J Cancer. 47(Suppl 3): S302–303.
2011. View Article : Google Scholar : PubMed/NCBI
|
3
|
Lee AW, Sze WM, Au JS, et al: Treatment
results for nasopharyngeal carcinoma in the modern era: The Hong
Kong experience. Int J Radiat Oncol Biol Phys. 61:1107–1116. 2005.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Chang JT, See LC, Liao CT, et al: Locally
recurrent nasopharyngeal carcinoma. Radiother Oncol. 54:135–142.
2000. View Article : Google Scholar : PubMed/NCBI
|
5
|
Chua DT, Sham JS, Kwong DL, et al: Locally
recurrent nasopharyngeal carcinoma: treatment results for patients
with computed tomography assessment. Int J Radiat Oncol Biol Phys.
41:379–386. 1998. View Article : Google Scholar : PubMed/NCBI
|
6
|
Ng WT, Lee MC, Hung WM, et al: Clinical
outcomes and patterns of failure after intensity-modulated
radiotherapy for nasopharyngeal carcinoma. Int J Radiat Oncol Biol
Phys. 79:420–428. 2011. View Article : Google Scholar
|
7
|
Su SF, Han F, Zhao C, Huang Y, et al:
Treatment outcomes for different subgroups of nasopharyngeal
carcinoma patients treated with intensity-modulated radiation
therapy. Chin J Cancer. 30:565–573. 2011. View Article : Google Scholar : PubMed/NCBI
|
8
|
Yang TS, Ng KT, Wang HM, et al: Prognostic
factors of locoregionally recurrent nasopharyngeal carcinoma - a
retrospective review of 182 cases. Am J Clin Oncol. 19:337–343.
1996. View Article : Google Scholar : PubMed/NCBI
|
9
|
Lee AW, Foo W, Law SC, et al: Recurrent
nasopharyngeal carcinoma: the puzzles of long latency. Int J Radiat
Oncol Biol Phys. 44:149–156. 1999. View Article : Google Scholar : PubMed/NCBI
|
10
|
Xu T, Tang J, Gu M, et al: Recurrent
nasopharyngeal carcinoma: a clinical dilemma and challenge. Curr
Oncol. 20:e406–e419. 2013. View Article : Google Scholar : PubMed/NCBI
|
11
|
Chua DT, Sham JS, Kwong PW, et al: Linear
accelerator-based stereotactic radiosurgery for limited, locally
persistent, and recurrent nasopharyngeal carcinoma: efficacy and
complications. Int J Radiat Oncol Biol Phys. 56:177–183. 2003.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Lee AW, Foo W, Law SC, et al: Total
biological effect on late reactive tissues following reirradiation
for recurrent nasopharyngeal carcinoma. Int J Radiat Oncol Biol
Phys. 46:865–872. 2000. View Article : Google Scholar : PubMed/NCBI
|
13
|
Hwang JM, Fu KK and Phillips TL: Results
and prognostic factors in the retreatment of locally recurrent
nasopharyngeal carcinoma. Int J Radiat Oncol Biol Phys.
41:1099–1111. 1998. View Article : Google Scholar : PubMed/NCBI
|
14
|
Teo PM, Kwan WH, Chan AT, et al: How
successful is high-dose (> or = 60 Gy) reirradiation using
mainly external beams in salvaging local failures of nasopharyngeal
carcinoma? Int J Radiat Oncol Biol Phys. 40:897–913. 1998.
View Article : Google Scholar : PubMed/NCBI
|
15
|
Oksüz DC, Meral G, Uzel O, Cağatay P and
Turkan S: Reirradiation for locally recurrent nasopharyngeal
carcinoma: treatment results and prognostic factors. Int J Radiat
Oncol Biol Phys. 60:388–394. 2004. View Article : Google Scholar : PubMed/NCBI
|
16
|
Chou J, Lin YC, Kim J, et al:
Nasopharyngeal carcinoma - review of the molecular mechanisms of
tumorigenesis. Head Neck. 30:946–963. 2008. View Article : Google Scholar : PubMed/NCBI
|
17
|
Kong QL, Hu LJ, Cao JY, et al:
Epstein-Barr virus-encoded LMP2A induces an epithelial-mesenchymal
transition and increases the number of side population stem-like
cancer cells in nasopharyngeal carcinoma. PLoS Pathog.
6:e10009402010. View Article : Google Scholar : PubMed/NCBI
|
18
|
Port RJ, Pinheiro-Maia S, Hu C, et al:
Epstein-Barr virus induction of the Hedgehog signalling pathway
imposes a stem cell phenotype on human epithelial cells. J Pathol.
231:367–377. 2013.PubMed/NCBI
|
19
|
Cho WC: Nasopharyngeal carcinoma:
molecular biomarker discovery and progress. Mol Cancer. 6:12007.
View Article : Google Scholar : PubMed/NCBI
|
20
|
Gao W, Li JZ, Ho WK, et al: Biomarkers for
use in monitoring responses of nasopharyngeal carcinoma cells to
ionizing radiation. Sensors (Basel). 12:8832–8846. 2012. View Article : Google Scholar
|
21
|
Wang HY, Sun BY, Zhu ZH, et al:
Eight-signature classifier for prediction of nasopharyngeal
[corrected] carcinoma survival. J Clin Oncol. 29:4516–4525. 2011.
View Article : Google Scholar : PubMed/NCBI
|
22
|
Krikelis D, Bobos M, Karayannopoulou G, et
al: Expression profiling of 21 biomolecules in locally advanced
nasopharyngeal carcinomas of Caucasian patients. BMC Clin Pathol.
13:12013. View Article : Google Scholar : PubMed/NCBI
|
23
|
Pan Y, Zhang Q, Atsaves V, et al:
Suppression of Jab1/CSN5 induces radio- and chemo-sensitivity in
nasopharyngeal carcinoma through changes to the DNA damage and
repair pathways. Oncogene. 32:2756–2766. 2013. View Article : Google Scholar :
|
24
|
Pan Y, Wang M, Bu X, et al: Curcumin
analogue T83 exhibits potent antitumor activity and induces
radiosensitivity through inactivation of Jab1 in nasopharyngeal
carcinoma. BMC Cancer. 13:3232013. View Article : Google Scholar : PubMed/NCBI
|
25
|
Pan Y, Zhang Q, Tian L, et al: Jab1/CSN5
negatively regulates p27 and plays a role in the pathogenesis of
nasopharyngeal carcinoma. Cancer Res. 72:1890–1900. 2012.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Chen CC, Chen WC, Lu CH, et al:
Significance of interleukin-6 signaling in the resistance of
pharyngeal cancer to irradiation and the epidermal growth factor
receptor inhibitor. Int J Radiat Oncol Biol Phys. 76:1214–1224.
2010. View Article : Google Scholar : PubMed/NCBI
|
27
|
Pectasides E, Egloff AM, Sasaki C, et al:
Nuclear localization of signal transducer and activator of
transcription 3 in head and neck squamous cell carcinoma is
associated with a better prognosis. Clin Cancer Res. 16:2427–2434.
2010. View Article : Google Scholar : PubMed/NCBI
|
28
|
Liu YP, Tan YN, Wang ZL, et al:
Phosphorylation and nuclear translocation of STAT3 regulated by the
Epstein-Barr virus latent membrane protein 1 in nasopharyngeal
carcinoma. Int J Mol Med. 21:153–162. 2008.PubMed/NCBI
|
29
|
Hsiao JR, Chang KC, Chen CW, et al:
Endoplasmic reticulum stress triggers XBP-1-mediated up-regulation
of an EBV oncoprotein in nasopharyngeal carcinoma. Cancer Res.
69:4461–4467. 2009. View Article : Google Scholar : PubMed/NCBI
|
30
|
Xia Y, Wong NS, Fong WF and Tideman H:
Upregulation of GADD153 expression in the apoptotic signaling of
N-(4-hydroxyphenyl) retinamide (4HPR). Int J Cancer. 102:7–14.
2002. View Article : Google Scholar : PubMed/NCBI
|
31
|
Shackleford TJ and Claret FX: JAB1/CSN5: a
new player in cell cycle control and cancer. Cell Div. 5:262010.
View Article : Google Scholar : PubMed/NCBI
|
32
|
Wang X, Crowe PJ, Goldstein D and Yang JL:
STAT3 inhibition, a novel approach to enhancing targeted therapy in
human cancers (Review). Int J Oncol. 41:1181–1191. 2012.PubMed/NCBI
|
33
|
Germain D and Frank DA: Targeting the
cytoplasmic and nuclear functions of signal transducers and
activators of transcription 3 for cancer therapy. Clin Cancer Res.
13:5665–5669. 2007. View Article : Google Scholar : PubMed/NCBI
|
34
|
Grandis JR, Drenning SD, Zeng Q, et al:
Constitutive activation of Stat3 signaling abrogates apoptosis in
squamous cell carcinogenesis in vivo. Proc Natl Acad Sci USA.
97:4227–4232. 2000. View Article : Google Scholar : PubMed/NCBI
|
35
|
Lui VW, Yau DM, Wong EY, et al:
Cucurbitacin I elicits anoikis sensitization, inhibits cellular
invasion and in vivo tumor formation ability of nasopharyngeal
carcinoma cells. Carcinogenesis. 30:2085–2094. 2009. View Article : Google Scholar : PubMed/NCBI
|
36
|
Hsiao JR, Jin YT, Tsai ST, et al:
Constitutive activation of STAT3 and STAT5 is present in the
majority of nasopharyngeal carcinoma and correlates with better
prognosis. Br J Cancer. 89:344–349. 2003. View Article : Google Scholar : PubMed/NCBI
|
37
|
Morrison JA, Gulley ML, Pathmanathan R and
Raab-Traub N: Differential signaling pathways are activated in the
Epstein-Barr virus-associated malignancies nasopharyngeal carcinoma
and Hodgkin lymphoma. Cancer Res. 64:5251–5260. 2004. View Article : Google Scholar : PubMed/NCBI
|
38
|
Kim IA, Bae SS, Fernandes A, et al:
Selective inhibition of Ras, phosphoinositide 3 kinase, and Akt
isoforms increases the radiosensitivity of human carcinoma cell
lines. Cancer Res. 65:7902–7910. 2005.PubMed/NCBI
|
39
|
Nijkamp MM, Hoogsteen IJ, Span PN, et al:
Spatial relationship of phosphorylated epidermal growth factor
receptor and activated AKT in head and neck squamous cell
carcinoma. Radiother Oncol. 101:165–170. 2011. View Article : Google Scholar : PubMed/NCBI
|
40
|
Seong J, Chung EJ, Kim H, et al:
Assessment of biomarkers in paired primary and recurrent colorectal
adenocarcinomas. Int J Radiat Oncol Biol Phys. 45:1167–1173. 1999.
View Article : Google Scholar : PubMed/NCBI
|