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International Journal of Molecular Medicine
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Print ISSN: 1107-3756 Online ISSN: 1791-244X
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April-2022 Volume 49 Issue 4

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Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

Medicine International

Medicine International

An International Open Access Journal Devoted to General Medicine.

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Correction Open Access

[Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway

  • Authors:
    • Limin Sun
    • Xin He
    • Tao Zhang
    • Yaling Han
    • Guizhou Tao
  • View Affiliations / Copyright

    Affiliations: Department of Medicine, Soochow University, Suzhou, Jiangsu 215123, P.R. China, Department of Cardiology, The First Affiliated Hospital of Jinzhou Medical University, Jinzhou, Liaoning 121001, P.R. China, Department of General Practice, The First Affiliated Hospital of Jinzhou Medical University, Jinzhou, Liaoning 121001, P.R. China, Department of Cardiology, General Hospital of Northern Theater Command, Shenyang, Liaoning 110016, P.R. China
    Copyright: © Sun et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 50
    |
    Published online on: February 11, 2022
       https://doi.org/10.3892/ijmm.2022.5106
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Article

Int J Mol Med 48: [Related article:] 208, 2021; DOI: 10.3892/ijmm.2021.5041

During the preparation of the figures in the above article, the authors have realized that errors were introduced during the assembly of Fig. 6. Specifically, the data were shown incorrectly for the H&E experiments shown in the top row for Fig. 6B, and the Oil Red O staining experiments for the Control and AS data panels in Fig. 6B have also been replaced with more representative data; in addition, the second bars in each of the histograms shown in this figure were mislabelled as 'AS': these bars should have been labelled as 'ox-LDL'.

Figure 6

Exosomal si-LOC100129516 suppresses atherosclerotic progression in vivo. (A) Reverse transcription-quantitative PCR was used to detect the levels of LOC100129516 in the aortic tissues of ApoE−/− mice. (B) Hematoxylin and eosin staining as well as Oil-red O staining were used to observe aortic plaque formation in the ApoE−/− mice. (C) ELISA kits were used to measure the levels of TC and LDL-C in the plasma of ApoE−/− mice. n=6. **P<0.01 vs. control group; #P<0.01, ##P<0.01 vs. AS group. siRNA, small interfering RNA; H&E, hematoxylin and eosin; TC, total cholesterol; LDL-C, low-density lipoprotein cholesterol; ctrl, control; AS, atherosclerosis; Exo, exosome.

The corrected version of Fig. 6 is shown below. These errors did not affect the major conclusions reported in the paper. All the authors have agreed to this Corrigendum, and thank the Editor of International Journal of Molecular Medicine for allowing them the opportunity to publish this. The authors regret these errors went unnoticed before the paper was published, and apologize to the readership for any confusion that it may have caused.

Figure 6

Exosomal si-LOC100129516 suppresses atherosclerotic progression in vivo. (A) Reverse transcription-quantitative PCR was used to detect the levels of LOC100129516 in the aortic tissues of ApoE−/− mice. (B) Hematoxylin and eosin staining as well as Oil-red O staining were used to observe aortic plaque formation in the ApoE−/− mice. (C) ELISA kits were used to measure the levels of TC and LDL-C in the plasma of ApoE−/− mice. n=6. **P<0.01 vs. control group; #P<0.01, ##P<0.01 vs. AS group. siRNA, small interfering RNA; H&E, hematoxylin and eosin; TC, total cholesterol; LDL-C, low-density lipoprotein cholesterol; ctrl, control; AS, atherosclerosis; Exo, exosome.

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Copy and paste a formatted citation
Spandidos Publications style
Sun L, He X, Zhang T, Han Y and Tao G: [Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway. Int J Mol Med 49: 50, 2022.
APA
Sun, L., He, X., Zhang, T., Han, Y., & Tao, G. (2022). [Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway. International Journal of Molecular Medicine, 49, 50. https://doi.org/10.3892/ijmm.2022.5106
MLA
Sun, L., He, X., Zhang, T., Han, Y., Tao, G."[Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway". International Journal of Molecular Medicine 49.4 (2022): 50.
Chicago
Sun, L., He, X., Zhang, T., Han, Y., Tao, G."[Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway". International Journal of Molecular Medicine 49, no. 4 (2022): 50. https://doi.org/10.3892/ijmm.2022.5106
Copy and paste a formatted citation
x
Spandidos Publications style
Sun L, He X, Zhang T, Han Y and Tao G: [Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway. Int J Mol Med 49: 50, 2022.
APA
Sun, L., He, X., Zhang, T., Han, Y., & Tao, G. (2022). [Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway. International Journal of Molecular Medicine, 49, 50. https://doi.org/10.3892/ijmm.2022.5106
MLA
Sun, L., He, X., Zhang, T., Han, Y., Tao, G."[Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway". International Journal of Molecular Medicine 49.4 (2022): 50.
Chicago
Sun, L., He, X., Zhang, T., Han, Y., Tao, G."[Corrigendum] Knockdown of mesenchymal stem cell‑derived exosomal LOC100129516 suppresses the symptoms of atherosclerosis via upregulation of the PPARγ/LXRα/ABCA1 signaling pathway". International Journal of Molecular Medicine 49, no. 4 (2022): 50. https://doi.org/10.3892/ijmm.2022.5106
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