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Molecular Medicine Reports
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Print ISSN: 1791-2997 Online ISSN: 1791-3004
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September-2026 Volume 34 Issue 3

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Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

Medicine International

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Retraction Open Access

[Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  

  • Authors:
    • Jialiang Hu
    • Tao Cheng
    • Lijun Zhang
    • Beicheng Sun
    • Lei Deng
    • Hanmei Xu
  • View Affiliations / Copyright

    Affiliations: Key Laboratory of Modern Chinese Medicines, Ministry of Education, China Pharmaceutical University, Nanjing, Jiangsu 211198, P.R. China, Liver Transplant Center, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu 210029, P.R. China
    Copyright: © Hu et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 240
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    Published online on: June 29, 2026
       https://doi.org/10.3892/mmr.2026.13950
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Following the publication of the above paper, it was drawn to the Editor’s attention by a concerned reader that the following anomalies/potential areas of concern appeared to be associated with the western blot data featured in Figs. 2, 4 and 6 (including the description of additional features that came to light based upon an independent analysis of the data in this paper that was undertaken by the Editorial Office):  i) There were overlapping data bands comparing the cyclin D1 and β‑actin bands in Fig. 2C and D with those in Fig. 4B, where the figure legends indicated that the experimental conditions for these figures were different; and ii) in Fig. 6A, two pairings of cyclin D1 blots appeared to be duplicated/doubled up (giving rise to four sets of blots), and moreover, the associated β‑actin blots also appeared to exhibit different overlaps of individual bands/pairs of bands. Given the identification of so many instances of data duplication comparing both within and between the abovementioned figures in this paper, the Editor has decided that this paper should be retracted from the Journal on account of a lack of confidence in the data. The authors were asked for an explanation to account for these concerns, but the Editorial Office did not receive a reply. The Editor apologizes to the readership for any inconvenience caused. [Molecular Medicine Reports 12: 4396‑4402, 2015; DOI: 10.3892/mmr.2015.3912]

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Copy and paste a formatted citation
Spandidos Publications style
Hu J, Cheng T, Zhang L, Sun B, Deng L and Xu H: [Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  . Mol Med Rep 34: 240, 2026.
APA
Hu, J., Cheng, T., Zhang, L., Sun, B., Deng, L., & Xu, H. (2026). [Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  . Molecular Medicine Reports, 34, 240. https://doi.org/10.3892/mmr.2026.13950
MLA
Hu, J., Cheng, T., Zhang, L., Sun, B., Deng, L., Xu, H."[Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  ". Molecular Medicine Reports 34.3 (2026): 240.
Chicago
Hu, J., Cheng, T., Zhang, L., Sun, B., Deng, L., Xu, H."[Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  ". Molecular Medicine Reports 34, no. 3 (2026): 240. https://doi.org/10.3892/mmr.2026.13950
Copy and paste a formatted citation
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Spandidos Publications style
Hu J, Cheng T, Zhang L, Sun B, Deng L and Xu H: [Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  . Mol Med Rep 34: 240, 2026.
APA
Hu, J., Cheng, T., Zhang, L., Sun, B., Deng, L., & Xu, H. (2026). [Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  . Molecular Medicine Reports, 34, 240. https://doi.org/10.3892/mmr.2026.13950
MLA
Hu, J., Cheng, T., Zhang, L., Sun, B., Deng, L., Xu, H."[Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  ". Molecular Medicine Reports 34.3 (2026): 240.
Chicago
Hu, J., Cheng, T., Zhang, L., Sun, B., Deng, L., Xu, H."[Retracted] Anti‑tumor peptide AP25 decreases cyclin D1 expression and inhibits MGC‑803 proliferation via phospho­extracellular signal‑regulated kinase‑, Src‑, c‑Jun N‑terminal kinase‑ and phosphoinositide 3­-kinase‑associated pathways  ". Molecular Medicine Reports 34, no. 3 (2026): 240. https://doi.org/10.3892/mmr.2026.13950
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