|
1
|
Braunwald E: Shattuck lecture -
cardiovascular medicine at the turn of the millennium: triumphs,
concerns, and opportunities. New Engl J Med. 337:1360–1369. 1997.
View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Ross R: Atherosclerosis - an inflammatory
disease - reply. N Engl J Med. 340:19291999.
|
|
3
|
Agostini L, Martinon F, Burns K, McDermott
MF, Hawkins PN and Tschopp J: NALP3 forms an IL-l β-processing
inflammasome with increased activity in Muckle-Wells
autoinflammatory disorder. Immunity. 20:319–325. 2004.PubMed/NCBI
|
|
4
|
Hoffman HM, Mueller JL, Broide DH,
Wanderer AA and Kolodner RD: Mutation of a new gene encoding a
putative pyrin-like protein causes familial cold autoinflammatory
syndrome and Muckle-Wells syndrome. Nat Genet. 29:301–305. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Schroder K and Tschopp J: The
inflammasomes. Cell. 140:821–832. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Neven B, Prieur AM and Maire PQD:
Cryopyrinopathies: update on pathogenesis and treatment. Nat Clin
Pract Rheumatol. 4:481–489. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Villani AC, Lemire M, Fortin G, et al:
Common variants in the NLRP3 region contribute to Crohn’s disease
susceptibility. Nat Genet. 41:71–76. 2009.
|
|
8
|
Pontillo A, Brandao L, Guimaraes R, Segat
L, Araujo J and Crovella S: Two SNPs in NLRP3 gene are involved in
the predisposition to type-1 diabetes and celiac disease in a
pediatric population from northeast Brazil. Autoimmunity.
43:583–589. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Pontillo A, Vendramin A, Catamo E, Fabris
A and Crovella S: The missense variation Q705K in CIAS1/NALP3/NLRP3
gene and an NLRP1 haplotype are associated with celiac disease. Am
J Gastroenterol. 106:539–544. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Kastbom A, Verma D, Eriksson P, Skogh T,
Wingren G and Soderkvist P: Genetic variation in proteins of the
cryopyrin inflammasome influences susceptibility and severity of
rheumatoid arthritis (The Swedish TIRA project). Rheumatology
(Oxford). 47:415–417. 2008. View Article : Google Scholar
|
|
11
|
Feldmann J, Prieur AM, Quartier P, et al:
Chronic infantile neurological cutaneous and articular syndrome is
caused by mutations in CIAS1, a gene highly expressed in
polymorphonuclear cells and chondrocytes. Am J Hum Genet.
71:198–203. 2002. View
Article : Google Scholar : PubMed/NCBI
|
|
12
|
Aganna E, Martinon F, Hawkins PN, et al:
Association of mutations in the NALP3/CIAS1/PYPAF1 gene with a
broad phenotype including recurrent fever, cold sensitivity,
sensorineural deafness, and AA amyloidosis. Arthritis Rheum.
46:2445–2452. 2002. View Article : Google Scholar
|
|
13
|
Aksentijevich I, Nowak M, Mallah M, et al:
De novo CIAS1 mutations, cytokine activation, and evidence for
genetic heterogeneity in patients with neonatal-onset multisystem
inflammatory disease (NOMID): a new member of the expanding family
of pyrin-associated autoinflammatory diseases. Arthritis Rheum.
46:3340–3348. 2002. View Article : Google Scholar
|
|
14
|
Roberts RL, Van Rij AM, Phillips LV, et
al: Interaction of the inflammasome genes CARD8 and NLRP3 in
abdominal aortic aneurysms. Atherosclerosis. 218:123–126. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Schoultz I, Verma D, Halfvarsson J, et al:
Combined polymorphisms in genes encoding the inflammasome
components NALP3 and CARD8 confer susceptibility to Crohn’s disease
in Swedish men. Am J Gastroenterol. 104:1180–1188. 2009.PubMed/NCBI
|
|
16
|
Roberts RL, Topless RK, Phipps-Green AJ,
Gearry RB, Barclay ML and Merriman TR: Evidence of interaction of
CARD8 rs2043211 with NALP3 rs35829419 in Crohn’s disease. Genes
Immun. 11:351–356. 2010.PubMed/NCBI
|
|
17
|
Bracey NA, Beck PL, Muruve DA, et al: The
Nlrp3 Inflammasome promotes myocardial dysfunction in structural
cardiomyopathy through IL-1β. Exp Physiol. 98:462–472.
2012.PubMed/NCBI
|
|
18
|
Verma D, Sarndahl E, Andersson H, et al:
The Q705K polymorphism in NLRP3 is a gain-of-function alteration
leading to excessive interleukin-1β and IL-18 production. Plos One.
7:e349772012.PubMed/NCBI
|
|
19
|
Van Guelpen B, Hultdin J, Johansson I,
Witthöft C, Weinehall L, Eliasson M, Hallmans G, Palmqvist R,
Jansson JH and Winkvist A: Plasma folate and total homocysteine
levels are associated with the risk of myocardial infarction,
independently of each other and of renal function. J Intern Med.
266:182–195. 2009.
|
|
20
|
Wennberg P, Wensley F, Di Angelantonio E,
et al: Haemostatic and inflammatory markers are independently
associated with myocardial infarction in men and women. Thromb Res.
129:68–73. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Purcell S, Neale B, Todd-Brown K, et al:
PLINK: A tool set for whole-genome association and population-based
linkage analyses. Am J Hum Genet. 81:559–575. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Quaedackers ME, van den Brink CE, van der
Saag PT and Tertoolen LGJ: Direct interaction between estrogen
receptor α and NF-κB in the nucleus of living cells. Mol Cell
Endocrinol. 273:42–50. 2007.
|
|
23
|
Wennberg M, Tornevi A, Johansson I,
Hornell A, Norberg M and Bergdahl IA: Diet and lifestyle factors
associated with fish consumption in men and women: a study of
whether gender differences can result in gender-specific
confounding. Nutr J. 11:1012012. View Article : Google Scholar
|
|
24
|
Malbut-Shennan K and Young A: The
physiology of physical performance and training in old age. Coron
Artery Dis. 10:37–42. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Bauernfeind F, Rieger A, Schildberg FA,
Knolle PA, Schmid-Burgk JL and Hornung V: NLRP3 inflammasome
activity is negatively controlled by miR-223. J Immunol.
189:4175–4181. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Libert C, Dejager L and Pinheiro I: The X
chromosome in immune functions: when a chromosome makes the
difference. Nat Rev Immunol. 10:594–604. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Ganter U, Arcone R, Toniatti C, Morrone G
and Ciliberto G: Dual control of C-reactive protein gene-expression
by interleukin-1 and interleukin-6. EMBO J. 8:3773–3779.
1989.PubMed/NCBI
|
