|
1
|
Kinney FJ and Herrmann RE: Increasing
occurrence of thyroiditis in the Rocky Mountain area. Rocky Mt Med
J. 59:35–37, passim. 1962.
|
|
2
|
Jacobson DL, Gange SJ, Rose NR and Graham
NM: Epidemiology and estimated population burden of selected
autoimmune diseases in the United States. Clin Immunol
Immunopathol. 84:223–243. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
McLeod DS and Cooper DS: The incidence and
prevalence of thyroid autoimmunity. Endocrine. 42:252–265. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Delemer B, Aubert JP, Nys P, Landron F and
Bouée S: An observational study of the initial management of
hypothyroidism in France: The ORCHIDÉE study. Eur J Endocrinol.
167:817–823. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Golden SH, Robinson KA, Saldanha I, Anton
B and Ladenson PW: Clinical review: Prevalence and incidence of
endocrine and metabolic disorders in the United States: a
comprehensive review. J Clin Endocrinol Metab. 94:1853–1878. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Chistiakov DA: Immunogenetics of
Hashimoto's thyroiditis. J Autoimmune Dis. 2:12005. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Chistiakov DA and Turakulov RI: CTLA-4 and
its role in autoimmune thyroid disease. J Mol Endocrinol. 31:21–36.
2003. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Caturegli P, De Remigis A, Chuang K,
Dembele M, Iwama A and Iwama S: Hashimoto's thyroiditis:
Celebrating the centennial through the lens of the Johns Hopkins
hospital surgical pathology records. Thyroid. 23:142–150. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
De Luca F, Santucci S, Corica D, Pitrolo
E, Romeo M and Aversa T: Hashimoto's thyroiditis in childhood:
Presentation modes and evolution over time. Ital J Pediatr.
39:82013. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Li Y, Bai Y, Liu Z, Ozaki T, Taniguchi E,
Mori I, Nagayama K, Nakamura H and Kakudo K: Immunohistochemistry
of IgG4 can help subclassify Hashimoto's autoimmune thyroiditis.
Pathol Int. 59:636–641. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Stagnaro-Green A: Approach to the patient
with postpartum thyroiditis. J Clin Endocrinol Metab. 97:334–342.
2012. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Ahmed R, Al-Shaikh S and Akhtar M:
Hashimoto thyroiditis: A century later. Adv Anat Pathol.
19:181–186. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Caturegli P, De Remigis A and Rose NR:
Hashimoto thyroiditis: Clinical and diagnostic criteria. Autoimmun
Rev. 13:391–397. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Marković S, Kostić G, Igrutinović Z and
Vuletić B: Hashimoto's thyroiditis in children and adolescents. Srp
Arh Celok Lek. 136:262–266. 2008.(In Serbian). View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Zaletel K and Gaberšček S: Hashimoto's
thyroiditis: From genes to the disease. Curr Genomics. 12:576–588.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Menconi F, Monti MC, Greenberg DA, Oashi
T, Osman R, Davies TF, Ban Y, Jacobson EM, Concepcion ES, Li CW, et
al: Molecular amino acid signatures in the MHC class II
peptide-binding pocket predispose to autoimmune thyroiditis in
humans and in mice. Proc Natl Acad Sci USA. 105:14034–14039. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Hiromatsu Y, Satoh H and Amino N:
Hashimoto's thyroiditis: History and future outlook. Hormones
(Athens). 12:12–18. 2013.PubMed/NCBI
|
|
18
|
Han S, Zhang S, Zhang W, Li R, Li Y, Wang
Z, Xie Y and Mao Y: CTLA4 polymorphisms and ophthalmopathy in
Graves' disease patients: Association study and meta-analysis. Hum
Immunol. 67:618–626. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Wang X-Y, Zuo D, Sarkar D and Fisher PB:
Blockade of cytotoxic T-lymphocyte antigen-4 as a new therapeutic
approach for advanced melanoma. Expert Opin Pharmacother.
12:2695–2706. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Walker LS and Sansom DM: The emerging role
of CTLA4 as a cell-extrinsic regulator of T cell responses. Nat Rev
Immunol. 11:852–863. 2011. View
Article : Google Scholar : PubMed/NCBI
|
|
21
|
Narooie-Nejad M, Moossavi M, Torkamanzehi
A and Moghtaderi A: Positive association of vitamin D Receptor gene
variations with multiple sclerosis in South East Iranian
population. BioMed Res Int. 2015:4275192015.PubMed/NCBI
|
|
22
|
Anjos S, Nguyen A, Ounissi-Benkalha H,
Tessier MC and Polychronakos C: A common autoimmunity predisposing
signal peptide variant of the cytotoxic T-lymphocyte antigen 4
results in inefficient glycosylation of the susceptibility allele.
J Biol Chem. 277:46478–46486. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Kouki T, Sawai Y, Gardine CA, Fisfalen ME,
Alegre ML and DeGroot LJ: CTLA-4 gene polymorphism at position 49
in exon 1 reduces the inhibitory function of CTLA-4 and contributes
to the pathogenesis of Graves' disease. J Immunol. 165:6606–6611.
2000. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Qiu H, Tang W, Yin P, Cheng F and Wang L:
Cytotoxic T-lymphocyte associated antigen 4 polymorphism and
Hashimoto's thyroiditis susceptibility: A meta-analysis. Endocrine.
45:198–205. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Ji R, Feng Y and Zhan WW: Updated analysis
of studies on the cytotoxic T-lymphocyte-associated antigen-4 gene
A49G polymorphism and Hashimoto's thyroiditis risk. Genet Mol Res.
12:1421–1430. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Ahmadi S, Rostamzadeh J, Khosravi D,
Shariati P and Shakiba N: Association of CTLA-4 gene 49A/G
polymorphism with the incidence of type 1 diabetes mellitus in the
Iranian Kurdish population. Pak J Biol Sci. 16:1929–1935. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Tang MJ and Zhou ZB: Association of the
CTLA-4 +49A/G polymorphism with rheumatoid arthritis in Chinese Han
population. Mol Biol Rep. 40:2627–2631. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Mora B, Bonamico M, Indovina P, Megiorni
F, Ferri M, Carbone MC, Cipolletta E and Mazzilli MC: CTLA-4 +49
A/G dimorphism in Italian patients with celiac disease. Hum
Immunol. 64:297–301. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Petrone A, Giorgi G, Mesturino CA, Capizzi
M, Cascino I, Nistico L, Osborn J, Di Mario U and Buzzetti R:
Association of DRB1*04-DQB1*0301 haplotype and lack of association
of two polymorphic sites at CTLA-4 gene with Hashimoto's
thyroiditis in an Italian population. Thyroid. 11:171–175. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Farra C, Awwad J, Fadlallah A, Sebaly G,
Hage G, Souaid M, Ashkar H, Medlej R, Gannageh MH and Halaby G:
Genetics of autoimmune thyroid disease in the Lebanese population.
J Community Genet. 3:259–264. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Andreevskiĭ TV, Sudomoina MA, Gusev EI,
Boĭko AN, Alekseenkov AD and Favorova OO: Polymorphism A/G in
position +49 of CTLA4 exon 1 in multiple sclerosis in Russians. Mol
Biol (Mosk). 36:643–648. 2002.(In Russian). PubMed/NCBI
|
|
32
|
Knight AK, Serrano D, Tomer Y and
Cunningham-Rundles C: CTLA-4 gene exon-1 +49 A/G polymorphism: Lack
of association with autoimmune disease in patients with common
variable immune deficiency. J Clin Immunol. 27:95–100. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
van Gerven NM, de Boer YS, Zwiers A, van
Hoek B, van Erpecum KJ, Beuers U, van Buuren HR, Drenth JP, den
Ouden JW, Verdonk RC, et al: Dutch Autoimmune Hepatitis Study
group: Cytotoxic T lymphocyte antigen-4 +49A/G polymorphism does
not affect susceptibility to autoimmune hepatitis. Liver Int.
33:1039–1043. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Monne M, Piras G, Palmas A, Arru L,
Murineddu M, Latte G, Noli A and Gabbas A: Cytotoxic T-lymphocyte
antigen-4 (CTLA-4) gene polymorphism and susceptibility to
non-Hodgkin's lymphoma. Am J Hematol. 76:14–18. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Ghaderi A: CTLA4 gene variants in
autoimmunity and cancer: A comparative review. Iran J Immunol.
8:127–149. 2011.PubMed/NCBI
|
|
36
|
Balbi G, Ferrera F, Rizzi M, Piccioli P,
Morabito A, Cardamone L, Ghio M, Palmisano GL, Carrara P, Pedemonte
S, et al: Association of −318 C/T and +49 A/G cytotoxic T
lymphocyte antigen-4 (CTLA-4) gene polymorphisms with a clinical
subset of Italian patients with systemic sclerosis. Clin Exp
Immunol. 149:40–47. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Muñoz-Valle JF, Valle Y, Padilla-Gutiérrez
JR, Parra-Rojas I, Rangel-Villalobos H, del Mercado M Vázquez,
Ledezma-Lozano IY, Villafan-Bernal JR, Armendáriz-Borunda J and
Pereira-Suárez AL: The +49A>G CTLA-4 polymorphism is associated
with rheumatoid arthritis in Mexican population. Clin Chim Acta.
411:725–728. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Yeşilkaya E, Koç A, Bideci A, Camurdan O,
Boyraz M, Erkal O, Ergun MA and Cinaz P: CTLA4 gene polymorphisms
in children and adolescents with autoimmune thyroid diseases. Genet
Test. 12:461–464. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Pastuszak-Lewandoska D, Domańska D,
Rudzińska M, Bossowski A, Kucharska A, Sewerynek E, Czarnecka K,
Migdalska-Sęk M and Czarnocka B: CTLA-4 polymorphisms (+49 A/G and
−318 C/T) are important genetic determinants of AITD susceptibility
and predisposition to high levels of thyroid autoantibodies in
Polish children - preliminary study. Acta Biochim Pol. 60:641–646.
2013.PubMed/NCBI
|
|
40
|
Heidari A, Daloii MR Noori, Keramatipour
M, Rashikinezhad A, Sahmani AA and Amirzargar AA: CTLA-4 gene
polymorphisms (−318C/T, +49A/G, +6230A/G) in Iranian patients with
multiple sclerosis. Iran J Allergy Asthma Immunol. 9:219–223.
2010.PubMed/NCBI
|
|
41
|
Han SZ, Zhang SH, Li R, Zhang WY and Li Y:
The common −318C/T polymorphism in the promoter region of CTLA4
gene is associated with reduced risk of ophthalmopathy in Chinese
Graves' patients. Int J Immunogenet. 33:281–287. 2006. View Article : Google Scholar : PubMed/NCBI
|
