1
|
Wijffels MC, Kirchhof CJ, Dorland R and
Allessie MA: Atrial fibrillation begets atrial fibrillation. A
study in awake chronically instrumented goats. Circulation.
92:1954–1968. 1995. View Article : Google Scholar : PubMed/NCBI
|
2
|
Ma CS, Liu XP and Wang Y: Originating from
the pulmonary veins electrophysiological characteristics of focal
atrial fibrillation with radio frequency ablation. Zhonghua Xin Lu
Shi Chang Xue Za Zhi. 4:18–22. 2000.(In Chinese).
|
3
|
Gaborit N, Steenman M, Lamirault G, et al:
Human atrial ion channel and transporter subunit gene-expression
remodeling associated with valvular heart disease and atrial
fibrillation. Circulation. 112:471–481. 2005. View Article : Google Scholar : PubMed/NCBI
|
4
|
Baruscotti M, Bucchi A and Difrancesco D:
Physiology and pharmacology of the cardiac pacemaker (‘funny’)
current. Pharmacol Ther. 107:59–79. 2005. View Article : Google Scholar : PubMed/NCBI
|
5
|
Morillo CA, Klein GJ, Jones DL and
Guiraudon CM: Chronic rapid atrial pacing. Structural, functional,
and electrophysiological characteristics of a new model of
sustained atrial fibrillation. Circulation. 91:1588–1595. 1995.
View Article : Google Scholar : PubMed/NCBI
|
6
|
Herrmann S, Stieber J and Ludwig A:
Pathophysiology of HCN channels. Pflugers Arch. 454:517–522. 2007.
View Article : Google Scholar : PubMed/NCBI
|
7
|
Robinson RB and Siegelbaum SA:
Hyperpolarization-activated cation currents: from molecules to
physiological function. Annu Rev Physiol. 65:453–480. 2003.
View Article : Google Scholar : PubMed/NCBI
|
8
|
Schulze-Bahr E, Neu A, Friederich P, et
al: Pacemaker channel dysfunction in a patient with sinus node
disease. J Clin Invest. 111:1537–1545. 2003. View Article : Google Scholar : PubMed/NCBI
|
9
|
Ueda K, Nakamura K, Hayashi T, et al:
Functional characterization of a trafficking-defective HCN4
mutation, D553N, associated with cardiac arrhythmia. J Biol Chem.
279:27194–27198. 2004. View Article : Google Scholar : PubMed/NCBI
|
10
|
Milanesi R, Baruscotti M, Gnecchi-Ruscone
T and DiFrancesco D: Familial sinus bradycardia associated with a
mutation in the cardiac pacemaker channel. N Engl J Med.
354:151–157. 2006. View Article : Google Scholar : PubMed/NCBI
|
11
|
Nof E, Luria D, Brass D, et al: Point
mutation in the HCN4 cardiac ion channel pore affecting synthesis,
trafficking, and functional expression is associated with familial
asymptomatic sinus bradycardia. Circulation. 116:463–470. 2007.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Zorn-Pauly K, Schaffer P, Pelzmann B, et
al: If in left human atrium: a potential contributor to atrial
ectopy. Cardiovasc Res. 64:250–259. 2004. View Article : Google Scholar : PubMed/NCBI
|
13
|
Stillitano F, Lonardo G, Zicha S, et al:
Molecular basis of funny current (If) in normal and failing human
heart. J Mol Cell Cardiol. 45:289–299. 2008. View Article : Google Scholar : PubMed/NCBI
|
14
|
Stephen J, Emerson B, Fox KA and
Dransfield I: The uncoupling of monocyte-platelet interactions from
the induction of proinflammatory signaling in monocytes. J Immunol.
191:5677–5683. 2013. View Article : Google Scholar : PubMed/NCBI
|