1
|
Lopes AA: End-stage renal disease due to
diabetes in racial/ethnic minorities and disadvantaged populations.
Ethnic Dis. 19:(Suppl 1). S1-47–S1-51. 2009.
|
2
|
Kihm L: Hypertension and diabetic
nephropathy. Exp Clin Endocrinol Diabetes. 124:333–334. 2016.
View Article : Google Scholar : PubMed/NCBI
|
3
|
Glastras SJ, Tsang M, Teh R, Chen H,
McGrath RT, Zaky AA, Pollock CA and Saad S: Maternal obesity
promotes diabetic nephropathy in rodent offspring. Sci Rep.
6:277692016. View Article : Google Scholar : PubMed/NCBI
|
4
|
Kikkawa R, Koya D and Haneda M:
Progression of diabetic nephropathy. Am J Kidney Dis. 41:(Suppl 1).
S19–S21. 2003. View Article : Google Scholar : PubMed/NCBI
|
5
|
Wendt T, Tanji N, Guo J, Hudson BI,
Bierhaus A, Ramasamy R, Arnold B, Nawroth PP, Yan SF, D'Agati V and
Schmidt AM: Glucose, glycation and rage: Implications for
amplification of cellular dysfunction in diabetic nephropathy. J Am
Soc Nephrol. 14:1383–1395. 2003. View Article : Google Scholar : PubMed/NCBI
|
6
|
Forbes JM, Thallas V, Thomas MC, Founds
HW, Burns WC, Jerums G and Cooper ME: The breakdown of preexisting
advanced glycation end products is associated with reduced renal
fibrosis in experimental diabetes. FASEB J. 17:1762–1764.
2003.PubMed/NCBI
|
7
|
Kilpatrick ES, Rigby AS and Atkin SL: The
diabetes control and complications trial: The gift that keeps
giving. Nat Rev Endocrinol. 5:537–545. 2009. View Article : Google Scholar : PubMed/NCBI
|
8
|
Lewko B and Stepinski J: Hyperglycemia and
mechanical stress: Targeting the renal podocyte. J Cell Physiol.
221:288–295. 2009. View Article : Google Scholar : PubMed/NCBI
|
9
|
Batlle D: Clinical and cellular markers of
diabetic nephropathy. Kidney Int. 63:2319–2330. 2003. View Article : Google Scholar : PubMed/NCBI
|
10
|
Shah IM, Mackay SP and McKay GA:
Therapeutic strategies in the treatment of diabetic nephropathy - a
translational medicine approach. Curr Med Chem. 16:997–1016. 2009.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Brezniceanu ML, Liu F, Wei CC, Chénier I,
Godin N, Zhang SL, Filep JG, Ingelfinger JR and Chan JS:
Attenuation of interstitial fibrosis and tubular apoptosis in db/db
transgenic mice overexpressing catalase in renal proximal tubular
cells. Diabetes. 57:451–459. 2008. View Article : Google Scholar : PubMed/NCBI
|
12
|
Elmarakby AA and Sullivan JC: Relationship
between oxidative stress and inflammatory cytokines in diabetic
nephropathy. Cardiovasc Ther. 30:49–59. 2012. View Article : Google Scholar : PubMed/NCBI
|
13
|
Guarente L: Calorie restriction and
sirtuins revisited. Gene Dev. 27:2072–2085. 2013. View Article : Google Scholar : PubMed/NCBI
|
14
|
de Oliveira Saraiva A, Pontes LQ, Pinho
LG, Bezerra MR Lobo, Braga H Alencar, Lima NN Rolim, de Vasconcelos
CA Carvalho, Neto ML Rolim, de Lima Fihlo JL, dos Santos FA
Brayner, et al: Ultrastructural aspects of cranial and peripheric
nerves of cronically diabetic and malnourished rats: A short
biochemical panorama. Int Arch Med. 8:2015.
|
15
|
Chalkiadaki A and Guarente L: Sirtuins
mediate mammalian metabolic responses to nutrient availability. Nat
Rev Endocrinol. 8:287–296. 2012. View Article : Google Scholar : PubMed/NCBI
|
16
|
Michishita E, Park JY, Burneskis JM,
Barrett JC and Horikawa I: Evolutionarily conserved and
nonconserved cellular localizations and functions of human SIRT
proteins. Mol Biol Cell. 16:4623–4635. 2005. View Article : Google Scholar : PubMed/NCBI
|
17
|
North BJ, Marshall BL, Borra MT, Denu JM
and Verdin E: The human Sir2 ortholog, SIRT2, is an
NAD+-dependent tubulin deacetylase. Mol Cell.
11:437–444. 2003. View Article : Google Scholar : PubMed/NCBI
|
18
|
Kume S, Uzu T, Horiike K, Chin-Kanasaki M,
Isshiki K, Araki S, Sugimoto T, Haneda M, Kashiwagi A and Koya D:
Calorie restriction enhances cell adaptation to hypoxia through
Sirt1-dependent mitochondrial autophagy in mouse aged kidney. J
Clin Invest. 120:1043–1055. 2010. View
Article : Google Scholar : PubMed/NCBI
|
19
|
Shi JX, Wang QJ, Li H and Huang Q:
Silencing of USP22 suppresses high glucose-induced apoptosis, ROS
production and inflammation in podocytes. Mol Biosyst.
12:1445–1456. 2016. View Article : Google Scholar : PubMed/NCBI
|
20
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(−Delta Delta C(T)) Method. Methods. 25:402–408. 2001.
View Article : Google Scholar : PubMed/NCBI
|
21
|
Chatterjee S, Rhee YH and Ahn JC:
Sulforaphene-carboplatin combination synergistically enhances
apoptosis by disruption of mitochondrial membrane potential and
cell cycle arrest in human non-small cell lung carcinoma. J Med
Food. 19:860–869. 2016. View Article : Google Scholar : PubMed/NCBI
|
22
|
Zhao X, Ren X, Zhu R, Luo Z and Ren B:
Zinc oxide nanoparticles induce oxidative DNA damage and
ROS-triggered mitochondria-mediated apoptosis in zebrafish embryos.
Aquat Toxicol. 180:56–70. 2016. View Article : Google Scholar : PubMed/NCBI
|
23
|
Haigis MC, Mostoslavsky R, Haigis KM,
Fahie K, Christodoulou DC, Murphy AJ, Valenzuela DM, Yancopoulos
GD, Karow M, Blander G, et al: SIRT4 inhibits glutamate
dehydrogenase and opposes the effects of calorie restriction in
pancreatic beta cells. Cell. 126:941–954. 2006. View Article : Google Scholar : PubMed/NCBI
|
24
|
Chen YR, Fang SR, Fu YC, Zhou XH, Xu MY
and Xu WC: Calorie restriction on insulin resistance and expression
of SIRT1 and SIRT4 in rats. Biochem Cell Biol. 88:715–722. 2010.
View Article : Google Scholar : PubMed/NCBI
|
25
|
Mahlknecht U and Voelter-Mahlknecht S:
Fluorescence in situ hybridization and chromosomal organization of
the sirtuin 4 gene (Sirt4) in the mouse. Biochem Bioph Res Commun.
382:685–690. 2009. View Article : Google Scholar
|
26
|
Susztak K, Raff AC, Schiffer M and
Böttinger EP: Glucose-induced reactive oxygen species cause
apoptosis of podocytes and podocyte depletion at the onset of
diabetic nephropathy. Diabetes. 55:225–233. 2006. View Article : Google Scholar : PubMed/NCBI
|
27
|
Schiffer M, Mundel P, Shaw AS and
Böttinger EP: A novel role for the adaptor molecule CD2-associated
protein in transforming growth factor-beta-induced apoptosis. J
Biol Chem. 279:37004–37012. 2004. View Article : Google Scholar : PubMed/NCBI
|
28
|
Long J, Wang Y, Wang W, Chang BH and
Danesh FR: MicroRNA-29c is a signature microRNA under high glucose
conditions that targets sprouty homolog 1 and its in vivo knockdown
prevents progression of diabetic nephropathy. J Biol Chem.
286:11837–11848. 2011. View Article : Google Scholar : PubMed/NCBI
|
29
|
Nilsson L, Burlaka I, Brismar H, Aperia A
and Scott L: Glucose activation of the mitochondrial apoptotic
pathway in proximal tubular cells and protective effect of ouabain
signaling. FASEB J. 29:912–959. 2015.
|
30
|
Wang H, Madhusudhan T, He T, Hummel B,
Schmidt S, Vinnikov IA, Shahzad K, Kashif M, Muller-Krebs S,
Schwenger V, et al: Low but sustained coagulation activation
ameliorates glucose-induced podocyte apoptosis: Protective effect
of factor V Leiden in diabetic nephropathy. Blood. 117:5231–5242.
2011. View Article : Google Scholar : PubMed/NCBI
|
31
|
Bock F, Shahzad K, Wang H, Stoyanov S,
Wolter J, Dong W, Pelicci PG, Kashif M, Ranjan S, Schmidt S, et al:
Activated protein C ameliorates diabetic nephropathy by
epigenetically inhibiting the redox enzyme p66Shc. Pro Nat Acad Sci
USA. 110:648–653. 2013. View Article : Google Scholar
|
32
|
Ahmad A, Mondello S, Di Paola R, Mazzon E,
Esposito E, Catania MA, Italiano D, Mondello P, Aloisi C and
Cuzzocrea S: Protective effect of apocynin, a NADPH-oxidase
inhibitor, against contrast-induced nephropathy in the diabetic
rats: A comparison with N-acetylcysteine. Eur J Pharmacol.
674:397–406. 2012. View Article : Google Scholar : PubMed/NCBI
|
33
|
Koshikawa M, Mukoyama M, Mori K, Suganami
T, Sawai K, Yoshioka T, Nagae T, Yokoi H, Kawachi H, Shimizu F, et
al: Role of p38 mitogen-activated protein kinase activation in
podocyte injury and proteinuria in experimental nephrotic syndrome.
J Am Soc Nephrol. 16:2690–2701. 2005. View Article : Google Scholar : PubMed/NCBI
|
34
|
Galkina E and Ley K: Leukocyte recruitment
and vascular injury in diabetic nephropathy. J Am Soc Nephrol.
17:368–377. 2006. View Article : Google Scholar : PubMed/NCBI
|
35
|
Sun L and Kanwar YS: Relevance of TNF-α in
the context of other inflammatory cytokines in the progression of
diabetic nephropathy. Kidney Int. 88:662–665. 2015. View Article : Google Scholar : PubMed/NCBI
|
36
|
Elseweidy MM, Elswefy SE, Younis NN and
Zaghloul MS: Pyridoxamine, an inhibitor of protein glycation, in
relation to microalbuminuria and proinflammatory cytokines in
experimental diabetic nephropathy. Exp Biol Med (Maywood).
238:881–888. 2013. View Article : Google Scholar : PubMed/NCBI
|
37
|
Navarro JF, Milena FJ, Mora C, León C,
Claverie F, Flores C and García J: Tumor necrosis factor-alpha gene
expression in diabetic nephropathy: Relationship with urinary
albumin excretion and effect of angiotensin-converting enzyme
inhibition. Kidney Int Suppl. 99:S98–S102. 2005. View Article : Google Scholar
|
38
|
Navarro JF, Mora C, Muros M and García J:
Urinary tumour necrosis factor-alpha excretion independently
correlates with clinical markers of glomerular and
tubulointerstitial injury in type 2 diabetic patients. Nephrol Dial
Transplant. 21:3428–3434. 2006. View Article : Google Scholar : PubMed/NCBI
|
39
|
Balasubramanyam M, Aravind S,
Gokulakrishnan K, Prabu P, Sathishkumar C, Ranjani H and Mohan V:
Impaired miR-146a expression links subclinical inflammation and
insulin resistance in type 2 diabetes. Mol Cell Biochem.
351:197–205. 2011. View Article : Google Scholar : PubMed/NCBI
|
40
|
Wong CK, Ho AW, Tong PU, Yeung CY, Kong
AP, Lun SW, Chan JC and Lam CW: Aberrant activation profile of
cytokines and mitogen-activated protein kinases in type 2 diabetic
patients with nephropathy. Clin Exp Immunol. 149:123–131. 2007.
View Article : Google Scholar : PubMed/NCBI
|