|
1
|
Zaky A, Deem S, Bendjelid K and Treggiari
MM: Characterization of cardiac dysfunction in sepsis: An ongoing
challenge. Shock. 41:12–24. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Annane D, Bellissant E and Cavaillon JM:
Septic shock. Lancet. 365:63–78. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Angus DC, Linde-Zwirble WT, Lidicker J,
Clermont G, Carcillo J and Pinsky MR: Epidemiology of severe sepsis
in the United States: Analysis of incidence, outcome, and
associated costs of care. Crit Care Med. 29:1303–1310. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Merx MW and Weber C: Sepsis and the heart.
Circulation. 116:793–802. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Alvarez S, Vico T and Vanasco V: Cardiac
dysfunction, mitochondrial architecture, energy production, and
inflammatory pathways: Interrelated aspects in endotoxemia and
sepsis. Int J Biochem Cell Biol. 81:307–314. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Vikenes K, Farstad M and Nordrehaug JE:
Serotonin is associated with coronary artery disease and cardiac
events. Circulation. 100:483–489. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Liu MY, Ren YP, Zhang LJ and Ding JY:
Pretreatment with Ginseng Fruit Saponins affects serotonin
expression in an experimental comorbidity model of myocardial
infarction and depression. Aging Dis. 7:680–686. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Cowen P: Serotonin depression and
antidepressant action. S Afr J Psychiatr. 19:942013.
|
|
9
|
Selim AM, Sarswat N, Kelesidis I, Iqbal M,
Chandra R and Zolty R: Plasma aerotonin in heart failure: Possible
marker and potential treatment target. Heart Lung Circ. 26:442–449.
2017. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Villeneuve C, Guilbeau-Frugier C, Sicard
P, Lairez O, Ordener C, Duparc T, De Paulis D, Couderc B,
Spreux-Varoquaux O, Tortosa F, et al: p53-PGC-1α pathway mediates
oxidative mitochondrial damage and cardiomyocyte necrosis induced
by monoamine oxidase-A upregulation: Role in chronic left
ventricular dysfunction in mice. Antioxid Redox Signal. 18:5–18.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Rudiger A and Singer M: Mechanisms of
sepsis-induced cardiac dysfunction. Crit Care Med. 35:1599–1608.
2007. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Liu AL, Zhu HD, Yu XZ, Liu J, Ma S, Liu ZY
and Guo SB: The mechanisms of LPS-induced cardiac dysfunction in
septic mice. Chin J Emerg Med. 24:825–829. 2015.
|
|
13
|
Walley KR: Deeper understanding of
mechanisms contributing to sepsis-induced myocardial dysfunction.
Crit Care. 18:1372014. View
Article : Google Scholar : PubMed/NCBI
|
|
14
|
Kong HL, Li ZQ, Zhao YJ, Zhao SM, Zhu L,
Li T, Fu Y and Li HJ: Ginsenoside Rb1 protects cardiomyocytes
against CoCl2-induced apoptosis in neonatal rats by inhibiting
mitochondria permeability transition pore opening. Acta Pharmacol
Sin. 31:687–695. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Passarelli C, Tozzi G, Pastore A, Bertini
E and Piemonte F: GSSG-mediated complex I defect in isolated
cardiac mitochondria. Int J Mol Med. 26:95–99. 2010.PubMed/NCBI
|
|
16
|
Williams DL, Ha T, Li C, Kalbfleisch JH,
Schweitzer J, Vogt W and Browder IW: Modulation of tissue Toll-like
receptor 2 and 4 during the early phases of polymicrobial sepsis
correlates with mortality. Crit Care Med. 31:1808–1818. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Ha T, Hua F, Grant D, Xia Y, Ma J, Gao X,
Kelley J, Williams DL, Kalbfleisch J, Browder IW, et al: Glucan
phosphate attenuates cardiac dysfunction and inhibits cardiac MIF
expression and apoptosis in septic mice. Am J Physiol Heart Circ
Physiol. 291:H1910–H1918. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Liu Z, Liu H and Zeng ZH: Chronic
unpredictable mild stress causing cardiac and thoracic spinal cord
electrophysiological abnormalities may be associated with increased
cardiac expression of serotonin and growth-associated protein-43 in
rats. Biomed Res Int. 2018:86979132018.PubMed/NCBI
|
|
19
|
Makara MA, Hoang KV, Ganesan LP, Crouser
ED, Gunn JS, Turner J, Schlesinger LS, Mohler PJ and Rajaram MV:
Cardiac electrical and structural changes during bacterial
infection: An instructive model to study cardiac dysfunction in
sepsis. J Am Heart Assoc. 5(pii): e0038202016.PubMed/NCBI
|
|
20
|
Christ T, Rozmaritsa N, Engel A, Berk E,
Knaut M, Metzner K, Canteras M, Ravens U and Kaumann A:
Arrhythmias, elicited by catecholamines and serotonin, vanish in
human chronic atrial fibrillation. Proc Natl Acad Sci USA.
111:11193–11198. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Goette A, Kalman JM, Aguinaga L, Akar J,
Cabrera JA, Chen SA, Chugh SS, Corradi D, D'Avila A, Dobrev D, et
al: EHRA/HRS/APHRS/SOLAECE expert consensus on Atrial
cardiomyopathies: Definition, characterisation, and clinical
implication. J Arrhythm. 32:247–278. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Goette A, Bukowska A, Dobrev D,
Pfeiffenberger J, Morawietz H, Strugala D, Wiswedel I, Röhl FW,
Wolke C, Bergmann S, et al: Acute atrial tachyarrhythmia induces
angiotensin II type 1 receptor-mediated oxidative stress and
microvascular flow abnormalities in the ventricles. Eur Heart J.
30:1411–1420. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Tucker NR and Ellinor PT: Emerging
directions in the genetics of atrial fibrillation. Circ Res.
114:1469–1482. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Grün S, Schumm J, Greulich S, Wagner A,
Schneider S, Bruder O, Kispert EM, Hill S, Ong P, Klingel K, et al:
Long-term follow-up of biopsy-proven viral myocarditis: Predictors
of mortality and incomplete recovery. J Am Coll Cardiol.
59:1604–1615. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Liu Z, Zeng Z, Wu C and Liu H: Tropisetron
inhibits sepsis by repressing hyper-inflammation and regulating the
cardiac action potential in rat models. Biomed Pharmacother.
110:380–388. 2019. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Pennec JP, Talarmin H, Droguet M,
Giroux-Metgès MA, Gioux M and Dorange G: Characterization of the
voltage-activated currents in cultured atrial myocytes isolated
from the heart of the common oyster Crassostrea gigas. J Exp
Biol. 207:3935–3944. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Rahme MM, Cotter B, Leistad E, Wadhwa MK,
Mohabir R, Ford AP, Eglen RM and Feld GK: Electrophysiological and
antiarrhythmic effects of the atrial selective 5-HT(4) receptor
antagonist RS-100302 in experimental atrial flutter and
fibrillation. Circulation. 100:2010–2017. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Galindo-Tovar A, Vargas ML, Escudero E and
Kaumann AJ: Ontogenic changes of the control by phosphodiesterase-3
and −4 of 5-HT responses in porcine heart and relevance to human
atrial 5-HT(4) receptors. Br J Pharmacol. 156:237–249. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Singer M: Mitochondrial function in
sepsis: Acute phase versus multiple organ failure. Crit Care Med 35
(9 Suppl). S441–S448. 2007. View Article : Google Scholar
|
|
30
|
Wang X, Liu X, Kong R, Zhan R, Wang X,
Leng X, Gong J, Duan M, Wang L, Wu L and Qian L: NGFI-B targets
mitochondria and induces cardiomyocyte apoptosis in
restraint-stressed rats by mediating energy metabolism disorder.
Cell Stress Chaperones. 14:639–648. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Xinxing W, Hong F, Rui Z, Yun Z, Jingbo G
and Lingjia Q: Phosphorylated nerve growth factor-induced clone B
(NGFI-B) translocates from the nucleus to mitochondria of stressed
rat cardiomyocytes and induces apoptosis. Stress. 15:545–553. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Briasoulis A, Androulakis E, Christophides
T and Tousoulis D: The role of inflammation and cell death in the
pathogenesis, progression and treatment of heart failure. Heart
Fail Rev. 21:169–176. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Liu ZJ, Liu H, Xue KD and Wu S:
Experimental research of heart serotonin and myocardial action
potential change in the model of sepsis rats. Chin J Clinicians.
10:72–75. 2016.
|
|
34
|
Chung HY, Kollmey AS, Schrepper A, Kohl M,
Bläss MF, Stehr SN, Lupp A, Gräler MH and Claus RA: Adjustment of
dysregulated ceramide metabolism in a murine model of
sepsis-induced cardiac dysfunction. Int J Mol Sci. 18(pii):
E8392017. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Mössner R and Lesch KP: Role of serotonin
in the immune system and in neuroimmune interactions. Brain Behav
Immun. 12:249–271. 1998. View Article : Google Scholar : PubMed/NCBI
|
