|
1
|
Trautmann A, Schnaidt S,
Lipska-Ziętkiewicz BS, Bodria M, Ozaltin F, Emma F, Anarat A, Melk
A, Azocar M, Oh J, et al: Long-term outcome of steroid-resistant
nephrotic syndrome in children. J Am Soc Nephrol. 28:3055–3065.
2017.PubMed/NCBI View Article : Google Scholar
|
|
2
|
Kopp JB: Global glomerulosclerosis in
primary nephrotic syndrome: Including age as a variable to predict
renal outcomes. Kidney Int. 93:1043–1044. 2018.PubMed/NCBI View Article : Google Scholar
|
|
3
|
Levey AS, Eckardt KU, Tsukamoto Y, Levin
A, Coresh J, Rossert J, De Zeeuw D, Hostetter TH, Lameire N and
Eknoyan G: Definition and classification of chronic kidney disease:
A position statement from kidney disease: Improving Global Outcomes
(KDIGO). Kidney Int. 67:2089–2100. 2005.PubMed/NCBI View Article : Google Scholar
|
|
4
|
Levey AS, Andreoli SP, Dubose T,
Provenzano R and Collins AJ: Chronic kidney disease: Common,
harmful and treatable-World Kidney Day 2007. Am J Nephrol.
27:108–112. 2007.PubMed/NCBI View Article : Google Scholar
|
|
5
|
Bennett MR: Biomarkers of therapeutic
response in primary nephrotic syndrome: Response. Pediat Nephrol.
28:161–162. 2013.PubMed/NCBI View Article : Google Scholar
|
|
6
|
Chen W, Jiang Y, Han J, Hu J, He T, Yan T,
Huang N, Zhang Q, Mei H, Liao Y, et al: Atgl deficiency induces
podocyte apoptosis and leads to glomerular filtration barrier
damage. FEBS J. 284:1070–1081. 2017.PubMed/NCBI View Article : Google Scholar
|
|
7
|
Tian X, Kim JJ, Monkley SM, Gotoh N,
Nandez R, Soda K, Inoue K, Balkin DM, Hassan H, Son SH, et al:
Podocyte-associated talin1 is critical for glomerular filtration
barrier maintenance. J Clin Invest. 124:1098–1113. 2014.PubMed/NCBI View
Article : Google Scholar
|
|
8
|
Petermann A and Floege J: Podocyte damage
resulting in podocyturia: A potential diagnostic marker to assess
glomerular disease activity. Nephron Clin Pract.
106(c61-c66)2007.PubMed/NCBI View Article : Google Scholar
|
|
9
|
Perico L, Conti S, Benigni A and Remuzzi
G: Podocyte-actin dynamics in health and disease. Nat Rev Nephrol.
12(692)2016.PubMed/NCBI View Article : Google Scholar
|
|
10
|
Nagata M: Podocyte injury and its
consequences. Kidney Int. 89:1221–1230. 2016.PubMed/NCBI View Article : Google Scholar
|
|
11
|
Ivanova EA, Arcolino FO, Elmonem MA,
Rastaldi MP, Giardino L, Cornelissen EM, Van Den Heuvel LP and
Levtchenko EN: Cystinosin deficiency causes podocyte damage and
loss associated with increased cell motility. Kidney Int.
89:1037–1048. 2016.PubMed/NCBI View Article : Google Scholar
|
|
12
|
Korn T, Bettelli E, Oukka M and Kuchroo
VK: IL-17 and Th17 cells. Annu Rev Immunol. 8:485–517.
2009.PubMed/NCBI View Article : Google Scholar
|
|
13
|
Beringer A, Noack M and Miossec P: IL-17
in chronic inflammation: From discovery to targeting. Trends Mol
Med. 22:230–241. 2016.PubMed/NCBI View Article : Google Scholar
|
|
14
|
Ivanov II, Mckenzie BS, Zhou L, Tadokoro
CE, Lepelley A, Lafaille JJ, Cua DJ and Littman DR: The orphan
nuclear receptor RORgammat directs the differentiation program of
proinflammatory IL-17+ T helper cells. Cell.
126:1121–1133. 2006.PubMed/NCBI View Article : Google Scholar
|
|
15
|
Kuwabara T, Ishikawa F, Kondo M and
Kakiuchi T: The role of IL-17 and related cytokines in inflammatory
autoimmune diseases. Mediators Inflamm.
2017(3908061)2017.PubMed/NCBI View Article : Google Scholar
|
|
16
|
Paust HJ, Turner JE, Steinmetz OM, Peters
A, Heymann F, Hölscher C, Wolf G, Kurts C, Mittrücker HW, Stahl RA
and Panzer U: The IL-23/Th17 axis contributes to renal injury in
experimental glomerulonephritis. J Am Soc Nephrol.
20(969)2009.PubMed/NCBI View Article : Google Scholar
|
|
17
|
Zhang L, Yan J, Yang B, Zhang G, Wang M,
Dong S, Liu W, Yang H and Li Q: IL-23 actived γδ T cells affect
Th17 cells and regulatory T cells by secreting IL-21 in children
with primary nephrotic syndrome. Scand J Immunol.
87(36)2017.PubMed/NCBI View Article : Google Scholar
|
|
18
|
Okada H, Inoue T, Hashimoto K, Suzuki H
and Matsushita S: D1-like receptor antagonist inhibits IL-17
expression and attenuates crescent formation in nephrotoxic serum
nephritis. Am J Nephrol. 30:274–279. 2009.PubMed/NCBI View Article : Google Scholar
|
|
19
|
Jennette JC and Falk RJ: Diagnosis and
management of glomerular diseases. Med Clin North Am. 81:653–677.
1997.PubMed/NCBI View Article : Google Scholar
|
|
20
|
Ji Z, Hu Z and Xu Y: APPL1 acts as a
protective factor against podocytes injury in high glucose
environment. Int J Clin Exp Pathol. 8:6764–6771. 2015.PubMed/NCBI
|
|
21
|
Rops AL, van der Vlag J, Jacobs CW,
Dijkman HB, Lensen JF, Wijnhoven TJ, van den Heuvel LP, van
Kuppevelt TH and Berden JH: Isolation and characterization of
conditionally immortalized mouse glomerular endothelial cell lines.
Kidney Int. 66:2193–2201. 2004.PubMed/NCBI View Article : Google Scholar
|
|
22
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(-Delta Delta C(T)) method. Methods. 25:402–408.
2001.PubMed/NCBI View Article : Google Scholar
|
|
23
|
Zhang YM, You LF, Chen J and Mao CP:
Expression of kinesin family member 3B is associated with poor
prognosis in epithelial ovarian cancer patients. Int J Clin Exp
Pathol. 10:2834–2842. 2017.
|
|
24
|
Chen WQ, Zhang Y, Jiang H, Li H, Li XY,
Yang X, Feng S, Wang YC, Lin C, Shen XJ, et al: Podocyte-related
proteins in membranous nephropathy progression. Chinese J Med
(Engl). 126:3782–3783. 2013.PubMed/NCBI
|
|
25
|
Yu D, Petermann A, Kunter U, Rong S,
Shankland SJ and Floege J: Urinary podocyte loss is a more specific
marker of ongoing glomerular damage than proteinuria. J Am Soc
Nephrol. 16:1733–1741. 2005.PubMed/NCBI View Article : Google Scholar
|
|
26
|
Buss H, Dörrie A, Schmitz ML, Hoffmann E,
Resch K and Kracht M: Constitutive and interleukin-1-inducible
phosphorylation of p65 NF-{kappa}B at serine 536 is mediated by
multiple protein kinases including I{kappa}B kinase (IKK)-{alpha},
IKK{beta}, IKK{epsilon}, TRAF family member-associated
(TANK)-binding kinase 1 (TBK1), and an unknown kinase and couples
p65 to TATA-binding protein-associated factor II31-mediated
interleukin-8 transcription. J Biol Chem. 279:55633–55643.
2004.PubMed/NCBI View Article : Google Scholar
|
|
27
|
Monaco C, Andreakos E, Kiriakidis S, Mauri
C, Bicknell C, Foxwell B, Cheshire N, Paleolog E and Feldmann M:
Canonical pathway of nuclear factor κB activation selectively
regulates proinflammatory and prothrombotic responses in human
atherosclerosis. Proc Natl Acad Sci USA. 101:5634–5639.
2004.PubMed/NCBI View Article : Google Scholar
|
|
28
|
Yin XL, Zou MS, Zhang Y, Wang J, Liu TL,
Tang JH, Qiu LR, Chen Y, Yuan HQ and Zhou JH: Twenty-three-year
review of disease patterns from renal biopsies: An experience from
a pediatric renal center. J Nephrol. 26:699–707. 2013.PubMed/NCBI View Article : Google Scholar
|
|
29
|
Yuen LK, Lai WM, Lau SC, Tong PC, Tse KC
and Chiu MC: Ten-year review of disease pattern from percutaneous
renal biopsy: An experience from a paediatric tertiary renal centre
in Hong Kong. Hong Kong Med J. 14:348–355. 2008.PubMed/NCBI
|
|
30
|
Umemoto S, Okamoto T, Yoshimura K,
Sakumura T, Murata T, Fukai T, Yano M and Matsuzaki M: Toll-like
receptor 4 function is essential for increasing oxidative stress,
and promoting the inflammation and glomerular sclerosis by
angiotensin II. J Am Coll Cardiol. 67:2243. 2016. View Article : Google Scholar
|
|
31
|
Reggiani F and Ponticelli C: Focal
segmental glomerular sclerosis: Do not overlook the role of immune
response. J Nephrol. 29:525–534. 2016.PubMed/NCBI View Article : Google Scholar
|
|
32
|
Cousins DJ, Lee TH and Staynov DZ:
Cytokine coexpression during human Th1/Th2 cell differentiation:
Direct evidence for coordinated expression of Th2 cytokines. J
Immunol. 169:2498–2506. 2002.PubMed/NCBI View Article : Google Scholar
|
|
33
|
Egwuagu CE, Yu CR, Zhang M, Mahdi RM, Kim
SJ and Gery I: Suppressors of cytokine signaling proteins are
differentially expressed in Th1 and Th2 cells: Implications for Th
cell lineage commitment and maintenance. J Immunol. 168:3181–3187.
2002.PubMed/NCBI View Article : Google Scholar
|
|
34
|
Essa S, Pacsa A, Raghupathy R, Said T,
Nampoory MRN, Johny KV and Alnakib W: Low levels of Th1-type
cytokines and increased levels of Th2-type cytokines in kidney
transplant recipients with active cytomegalovirus infection.
Transplant Proc. 41:1643–1647. 2009.PubMed/NCBI View Article : Google Scholar
|
|
35
|
Hwang YJ, Yun MO, Jeong KT and Park JH:
Uremic toxin indoxyl 3-sulfate regulates the differentiation of Th2
but not of Th1 cells to lessen allergic asthma. Toxicol Lett.
225:130–138. 2014.PubMed/NCBI View Article : Google Scholar
|
|
36
|
Lama G, Luongo I, Tirino G, Borriello A,
Carangio C and Salsano ME: T-lymphocyte populations and cytokines
in childhood nephrotic syndrome. Am J Kidney Dis. 39:958–965.
2002.PubMed/NCBI View Article : Google Scholar
|
|
37
|
Kaneko K, Tuchiya K, Fujinaga S, Kawamura
R, Ohtomo Y, Shimizu T and Yamashiro Y: Th1/Th2 balance in
childhood idiopathic nephrotic syndrome. Clin Nephrol. 58:393–397.
2002.PubMed/NCBI View
Article : Google Scholar
|
|
38
|
Weaver CT, Harrington LE, Mangan PR,
Gavrieli M and Murphy KM: Th17: An effector CD4 T cell lineage with
regulatory T cell ties. Immunity. 24:677–688. 2006.PubMed/NCBI View Article : Google Scholar
|
|
39
|
Gong F, Liu Z, Liu J, Zhou P, Liu Y and Lu
X: The paradoxical role of IL-17 in atherosclerosis. Cell Immunol.
297:33–39. 2015.PubMed/NCBI View Article : Google Scholar
|
|
40
|
Potekhina AV, Pylaeva E, Provatorov S,
Ruleva N, Masenko V, Noeva E, Krasnikova T and Arefieva T:
Treg/Th17 balance in stable CAD patients with different stages of
coronary atherosclerosis. Atherosclerosis. 238:17–21.
2015.PubMed/NCBI View Article : Google Scholar
|
|
41
|
Babaloo Z, Aliparasti MR, Babaiea F,
Almasi S, Baradaran B and Farhoudi M: The role of Th17 cells in
patients with relapsing-remitting multiple sclerosis:
Interleukin-17A and interleukin-17F serum levels. Immunol Lett.
164:76–80. 2015.PubMed/NCBI View Article : Google Scholar
|
|
42
|
Matsumoto K and Kanmatsuse K: Increased
urinary excretion of interleukin-17 in nephrotic patients. Nephron.
91:243–249. 2002.PubMed/NCBI View Article : Google Scholar
|
|
43
|
Matsumoto K and Kanmatsuse K:
Interleukin-17 stimulates the release of pro-inflammatory cytokines
by blood monocytes in patients with IgA nephropathy. Scand J Urol
Nephrol. 37:164–171. 2003.PubMed/NCBI View Article : Google Scholar
|
|
44
|
Niranjan T, Bielesz B, Gruenwald A, Ponda
MP, Kopp JB, Thomas DB and Susztak K: The Notch pathway in
podocytes plays a role in the development of glomerular disease.
Nat Med. 14(290)2008.PubMed/NCBI View
Article : Google Scholar
|
|
45
|
Reiser J, von Gersdorff G, Loos M, Oh J,
Asanuma K, Giardino L, Rastaldi MP, Calvaresi N, Watanabe H,
Schwarz K, et al: Induction of B7-1 in podocytes is associated with
nephrotic syndrome. J Clin Invest. 113:1390–1397. 2004.PubMed/NCBI View Article : Google Scholar
|
|
46
|
Kavoura E, Gakiopoulou H, Paraskevakou H,
Marinaki S, Agrogiannis G, Stofas A, Boletis I, Patsouris E and
Lazaris AC: Immunohistochemical evaluation of podocalyxin
expression in glomerulopathies associated with nephrotic syndrome.
Hum Pathol. 42:227–235. 2011.PubMed/NCBI View Article : Google Scholar
|
|
47
|
Mollet G, Ratelade J, Boyer O, Muda AO,
Morisset L, Lavin TA, Kitzis D, Dallman MJ, Bugeon L, Hubner N, et
al: Podocin inactivation in mature kidneys causes focal segmental
glomerulosclerosis and nephrotic syndrome. J Am Soc Nephrol.
20:2181–2189. 2009.PubMed/NCBI View Article : Google Scholar
|
|
48
|
Sakurai N Kuroiwa T, Ikeuchi H, Hiramatsu
N, Takeuchi S, Tomioka M, Shigehara T, Maeshima A, Kaneko Y and
Hiromura K: Fluvastatin prevents podocyte injury in a murine model
of HIV-associated nephropathy. Nephrol Dial Transplant.
24:2378–2383. 2009.PubMed/NCBI View Article : Google Scholar
|
|
49
|
Wei P, Grimm PR, Settles DC, Balwanz CR,
Padanilam BJ and Sansom SC: Simvastatin reverses podocyte injury
but not mesangial expansion in early stage type 2 diabetes
mellitus. Ren Fail. 31:503–513. 2009.PubMed/NCBI View Article : Google Scholar
|
|
50
|
Kondo M, Murakawa Y, Harashima N,
Kobayashi S, Yamaguchi S and Harada M: Roles of proinflammatory
cytokines and the Fas/Fas ligand interaction in the pathogenesis of
inflammatory myopathies. Immunology. 128 (Suppl
1)(e589-e599)2009.PubMed/NCBI View Article : Google Scholar
|
|
51
|
Sun SC: The non-canonical NF-κB pathway in
immunity and inflammation. Nat Rev Immunol. 17:545–558.
2017.PubMed/NCBI View Article : Google Scholar
|
