|
1
|
Go AS, Chertow GM, Fan D, McCulloch CE and
Hsu CY: Chronic kidney disease and the risks of death,
cardiovascular events, and hospitalization. N Engl J Med.
351:1296–1305. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Amann K, Gross ML and Ritz E:
Pathophysiology underlying accelerated atherogenesis in renal
disease: Closing in on the target. J Am Soc Nephrol. 15:1664–1666.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Foley RN, Parfrey PS and Sarnak MJ:
Clinical epidemiology of cardiovascular disease in chronic renal
disease. Am J Kidney Dis. 32(Suppl 3): S112–S119. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Seliger SL, Gillen DL, Tirschwell D, Wasse
H, Kestenbaum BR and Stehman-Breen CO: Risk factors for incident
stroke among patients with end-stage renal disease. J Am Soc
Nephrol. 14:2623–2631. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Banerjee D, Recio-Mayoral A, Chitalia N
and Kaski JC: Insulin resistance, inflammation, and vascular
disease in nondiabetic predialysis chronic kidney disease patients.
Clin Cardiol. 34:360–365. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Pedersen TX, Binder CJ, Fredrikson GN,
Nilsson J, Bro S and Nielsen LB: The pro-inflammatory effect of
uraemia overrules the anti-atherogenic potential of immunization
with oxidized LDL in apoE−/− mice. Nephrol Dial
Transplant. 25:2486–2491. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Yilmaz MI, Stenvinkel P, Sonmez A, Saglam
M, Yaman H, Kilic S, Eyileten T, Caglar K, Oguz Y, Vural A, et al:
Vascular health, systemic inflammation and progressive reduction in
kidney function; clinical determinants and impact on cardiovascular
outcomes. Nephrol Dial Transplant. 26:3537–3543. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Pluquet O, Pourtier A and Abbadie C: The
unfolded protein response and cellular senescence. A review in the
theme: Cellular mechanisms of endoplasmic reticulum stress
signaling in health and disease. Am J Physiol Cell Physiol.
308:C415–C425. 2015. View Article : Google Scholar
|
|
9
|
Ron D and Walter P: Signal integration in
the endoplasmic reticulum unfolded protein response. Nat Rev Mol
Cell Biol. 8:519–529. 2007. View
Article : Google Scholar : PubMed/NCBI
|
|
10
|
Sozen E, Karademir B and Ozer NK: Basic
mechanisms in endoplasmic reticulum stress and relation to
cardiovascular diseases. Free Radic Biol Med. 78:30–41. 2015.
View Article : Google Scholar
|
|
11
|
Gotoh T, Endo M and Oike Y: Endoplasmic
reticulum stress-related inflammation and cardiovascular diseases.
Int J Inflamm. 2011:2594622011. View Article : Google Scholar
|
|
12
|
Tabas I: The role of endoplasmic reticulum
stress in the progression of atherosclerosis. Circ Res.
107:839–850. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Zhou J, Lhoták S, Hilditch BA and Austin
RC: Activation of the unfolded protein response occurs at all
stages of atherosclerotic lesion development in apolipoprotein
E-deficient mice. Circulation. 111:1814–1821. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Wang J, Wen Y, Lv LL, Liu H, Tang RN, Ma
KL and Liu BC: Involvement of endoplasmic reticulum stress in
angiotensin II-induced LRP3 inflammasome activation in human renal
proximal tubular cells in vitro. Acta Pharmacol Sin. 36:821–830.
2015. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Young CN, Li A, Dong FN, Horwath JA, Clark
CG and Davisson RL: Endoplasmic reticulum and oxidant stress
mediate nuclear factor-κB activation in the subfornical organ
during angiotensin II hypertension. Am J Physiol Cell Physiol.
308:C803–C812. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Bernardi S, Candido R, Toffoli B, Carretta
R and Fabris B: Prevention of accelerated atherosclerosis by AT1
receptor blockade in experimental renal failure. Nephrol Dial
Transplant. 26:832–838. 2011. View Article : Google Scholar
|
|
17
|
Bro S, Binder CJ, Witztum JL, Olgaard K
and Nielsen LB: Inhibition of the renin-angiotensin system
abolishes the proatherogenic effect of uremia in apolipoprotein
E-deficient mice. Arterioscler Thromb Vasc Biol. 27:1080–1086.
2007. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Yao S, Miao C, Tian H, Sang H, Yang N,
Jiao P, Han J, Zong C and Qin S: Endoplasmic reticulum stress
promotes macrophage-derived foam cell formation by up-regulating
cluster of differentiation 36 (CD36) expression. J Biol Chen.
289:4032–4042. 2014. View Article : Google Scholar
|
|
19
|
Berger AK, Duval S and Krumholz HM:
Aspirin, beta-blocker, and angiotensin-converting enzyme inhibitor
therapy in patients with end-stage renal disease and an acute
myocardial infarction. J Am Coll Cardiol. 42:201–208. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Mach F: The role of chemokines in
atherosclerosis. Curr Atheroscler Rep. 3:243–251. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Hansson GK: Inflammatory mechanisms in
atherosclerosis. J Thromb Haemost. 7(Suppl 1): 328–331. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Liang C, Wu ZG, Ding J, Jiang JF, Huang
GZ, Du RZ and Ge JB: Losartan inhibited expression of matrix
metalloproteinases in rat atherosclerotic lesions and angiotensin
II-stimulated macrophages. Acta Pharmacol Sin. 25:1426–1432.
2004.PubMed/NCBI
|
|
23
|
Kabour A, Henegar JR, Devineni VR and
Janicki JS: Prevention of angiotensin II induced myocyte necrosis
and coronary vascular damage by lisinopril and losartan in the rat.
Cardiovasc Res. 29:543–548. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Drogat B, Auguste P, Nguyen DT,
Bouchecareilh M, Pineau R, Nalbantoglu J, Kaufman RJ, Chevet E,
Bikfalvi A and Moenner M: IRE1 signaling is essential for
ischemia-induced vascular endothelial growth factor-A expression
and contributes to angiogenesis and tumor growth in vivo. Cancer
Res. 67:6700–6707. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Wang X, Bai YP, Hong D, Gao HC, Li LF, Li
CC, Zhu LP, Sun Q and Zhang GG: Ang II induces capillary formation
from endothelial cells via the AT1R-dependent inositol requiring
enzyme 1 pathway. Biochem Biophys Res Commun. 434:552–558. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Hu P, Han Z, Couvillon AD, Kaufman RJ and
Exton JH: Autocrine tumor necrosis factor alpha links endoplasmic
reticulum stress to the membrane death receptor pathway through
IRE1alpha-mediated NF-kappaB activation and downregulation of TRAF2
expression. Mol Cell Biol. 26:3071–3084. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Kaneko M, Niinuma Y and Nomura Y:
Activation signal of nuclear factor-kappa B in response to
endoplasmic reticulum stress is transduced via IRE1 and tumor
necrosis factor receptor-associated factor 2. Biol Pharm Bull.
26:931–935. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Wu L, Wang D, Xiao Y, Zhou X, Wang L, Chen
B, Li Q, Guo X and Huang Q: Endoplasmic reticulum stress plays a
role in the advanced glycation end product-induced inflammatory
response in endothelial cells. Life Sci. 110:44–51. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Urano F, Wang X, Bertolotti A, Zhang Y,
Chung P, Harding HP and Ron D: Coupling of stress in the ER to
activation of JNK protein kinases by transmembrane protein kinase
IRE1. Science. 287:664–666. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Miyazaki-Anzai S, Masuda M, Demos-Davies
KM, Keenan AL, Saunders SJ, Masuda R, Jablonski K, Cavasin MA,
Kendrick J, Chonchol M, et al: Endoplasmic reticulum stress
effector CCAAT/enhancer-binding protein homologous protein (CHOP)
regulates chronic kidney disease-induced vascular calcification. J
Am Heart Assoc. 3:e0009492014. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Galkina E and Ley K: Immune and
inflammatory mechanisms of atherosclerosis (*). Annu Rev Immunol.
27:165–197. 2009. View Article : Google Scholar
|
|
32
|
Libby P: Inflammation in atherosclerosis.
Nature. 420:868–874. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Guo F, Chen XL, Wang F, Liang X, Sun YX
and Wang YJ: Role of angiotensin II type 1 receptor in angiotensin
II-induced cytokine production in macrophages. J Interferon
Cytokine Res. 31:351–361. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Xanthoulea S, Curfs DM, Hofker MH and de
Winther MP: Nuclear factor kappa B signaling in macrophage function
and atherogenesis. Curr Opin Lipidol. 16:536–542. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
de Winther MPJ, Kanters E, Kraal G and
Hofker MH: Nuclear factor kappaB signaling in atherogenesis.
Arterioscler Thromb Vasc Biol. 25:904–914. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Martinon F, Chen X, Lee AH and Glimcher
LH: TLR activation of the transcription factor XBP1 regulates
innate immune responses in macrophages. Nat Immunol. 11:411–418.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Takeya M, Yoshimura T, Leonard EJ and
Takahashi K: Detection of monocyte chemoattractant protein-1 in
human atherosclerotic lesions by an anti-monocyte chemoattractant
protein-1 monoclonal antibody. Hum Pathol. 24:534–539. 1993.
View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Ylä-Herttuala S, Lipton BA, Rosenfeld ME,
Särkioja T, Yoshimura T, Leonard EJ, Witztum JL and Steinberg D:
Expression of monocyte chemoattractant protein 1 in macrophage-rich
areas of human and rabbit atherosclerotic lesions. Proc Natl Acad
Sci USA. 88:5252–5256. 1991. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Boring L, Gosling J, Cleary M and Charo
IF: Decreased lesion formation in CCR2−/− mice reveals a
role for chemokines in the initiation of atherosclerosis. Nature.
394:894–897. 1998. View
Article : Google Scholar : PubMed/NCBI
|
|
40
|
Gu L, Okada Y, Clinton SK, Gerard C,
Sukhova GK, Libby P and Rollins BJ: Absence of monocyte
chemoattractant protein-1 reduces atherosclerosis in low density
lipoprotein receptor-deficient mice. Mol Cell. 2:275–281. 1998.
View Article : Google Scholar : PubMed/NCBI
|
