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International Journal of Molecular Medicine
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Print ISSN: 1107-3756 Online ISSN: 1791-244X
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August-2023 Volume 52 Issue 2

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

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Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

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Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

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World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

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International Journal of Epigenetics

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Correction Open Access

[Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice

  • Authors:
    • Chuncha Bao
    • Zhong Yang
    • Qiyan Cai
    • Qian Li
    • Hongli Li
    • Bin Shu
  • View Affiliations / Copyright

    Affiliations: Department of Rehabilitation Medicine, University Town Hospital, Chongqing Medical University, Chongqing 401331, P.R. China, Department of Clinical Blood Teaching and Research, Army Medical University, Chongqing 400038, P.R. China, Department of Histology and Embryology, Army Medical University, Chongqing 400038, P.R. China
    Copyright: © Bao et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 71
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    Published online on: July 3, 2023
       https://doi.org/10.3892/ijmm.2023.5274
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Article

Int J Mol Med 44: [Related article:] 1677-1686, 2019; DOI: 10.3892/ijmm.2019.4344

Following the publication of the above article, an interested reader drew to the authors' attention that, for the immunofluorescence staining experiments shown in Fig. 3A on p. 1681, the 'E-cadherin / YC' and 'E-cadherin / OC' data panels were overlapping, such that they may have been derived from the same original source; moreover, with the transmission electron microscopic images shown in Fig. 6G on p. 1683, the 'OC' and 'OY' panels were similarly overlapping.

Figure 3

Effects of incremental load training on the expression of the E-cadherin and α-SMA in the kidney tissues of aged mice. (A) Expression of E-cadherin and α-SMA were identified by immunofluorescence staining (magnification, x400; scale bar=20 µm). The red color represents a positive signal; cell nuclei were counterstained with Hoechst (blue). Relative OD values of (B) E-cadherin and (C) α-SMA staining are shown in bar graphs. Values are expressed as the mean ± standard deviation (n=3). **P<0.01. YC, young control group; OC, elderly control group; OY, elderly exercise group; OD, optical density; α-SMA, α-smooth muscle actin.

Figure 6

Effects of incremental load training on autophagic markers Beclin 1 and LC3 in the kidney tissues of aged mice. (A) Comparison of the effect of incremental load training on the relative mRNA expression of Beclin 1 and LC3 in kidney tissues. Bar graphs show the relative quantification of (B) Beclin 1 and (C) LC3 mRNA. (D) Expression of Beclin 1 and LC3 was assessed by immunofluorescence staining (magnification, x200; scale bar=20 µm). The green color indicates the positive signal; cell nuclei were counterstained with DAPI (blue). Bar graphs show the relative OD values of (E) Beclin1 and (F) LC3 staining. Values are expressed as the mean ± standard deviation (n=3). *P<0.05, **P<0.01. (G) Assessment of autophagic activity by transmission electron microscopy (magnification, ×6,000 and ×15,000; scale bars, 2 and 1 µm). The arrows indicate autophagic vacuoles. YC, young control group; OC, elderly control group; OY, elderly exercise group; OD, optical density; LC3, light chain 3.

After having re-examined their figures, the authors have realized that the data shown for the 'E-cadherin / YC' experiment in Fig. 3A and the 'OC' experiment in Fig. 6G were selected incorrectly. The authors were able, however, to identify the correct data for both of these figures, and the revised versions of Figs. 3 and 6 are shown on the next page. Note that the errors made during the assembly of these figures did not affect the overall conclusions reported in the paper. All the authors agree with the publication of this corrigendum, and are grateful to the Editor of International Journal of Molecular Medicine for allowing them the opportunity to publish this. They also apologize to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Bao C, Yang Z, Cai Q, Li Q, Li H and Shu B: [Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice. Int J Mol Med 52: 71, 2023.
APA
Bao, C., Yang, Z., Cai, Q., Li, Q., Li, H., & Shu, B. (2023). [Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice. International Journal of Molecular Medicine, 52, 71. https://doi.org/10.3892/ijmm.2023.5274
MLA
Bao, C., Yang, Z., Cai, Q., Li, Q., Li, H., Shu, B."[Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice". International Journal of Molecular Medicine 52.2 (2023): 71.
Chicago
Bao, C., Yang, Z., Cai, Q., Li, Q., Li, H., Shu, B."[Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice". International Journal of Molecular Medicine 52, no. 2 (2023): 71. https://doi.org/10.3892/ijmm.2023.5274
Copy and paste a formatted citation
x
Spandidos Publications style
Bao C, Yang Z, Cai Q, Li Q, Li H and Shu B: [Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice. Int J Mol Med 52: 71, 2023.
APA
Bao, C., Yang, Z., Cai, Q., Li, Q., Li, H., & Shu, B. (2023). [Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice. International Journal of Molecular Medicine, 52, 71. https://doi.org/10.3892/ijmm.2023.5274
MLA
Bao, C., Yang, Z., Cai, Q., Li, Q., Li, H., Shu, B."[Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice". International Journal of Molecular Medicine 52.2 (2023): 71.
Chicago
Bao, C., Yang, Z., Cai, Q., Li, Q., Li, H., Shu, B."[Corrigendum] Incremental load training improves renal fibrosis by regulating the TGF‑β1/TAK1/MKK3/p38MAPK signaling pathway and inducing the activation of autophagy in aged mice". International Journal of Molecular Medicine 52, no. 2 (2023): 71. https://doi.org/10.3892/ijmm.2023.5274
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