[Corrigendum] Curcumin reverses benzidine-induced epithelial-mesenchymal transition via suppression of ERK5/AP-1 in SV-40 immortalized human urothelial cells

Liu,Zhiqi; Liu,Jie; Zhao,Li; Geng,Hao; Ma,Jiaxing; Zhang,Zhiqiang; Yu,Dexin; Zhong,Caiyun

doi:10.3892/ijo.2023.5533

July-2023 Volume 63 Issue 1

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[Corrigendum] Curcumin reverses benzidine-induced epithelial-mesenchymal transition via suppression of ERK5/AP-1 in SV-40 immortalized human urothelial cells

Authors:
- Zhiqi Liu
- Jie Liu
- Li Zhao
- Hao Geng
- Jiaxing Ma
- Zhiqiang Zhang
- Dexin Yu
- Caiyun Zhong
View Affiliations / Copyright

Affiliations: Department of urology, The Second affiliated Hospital of anhui Medical university, Hefei, Anhui 230032, P.R. China, Department of Nutrition and Food Safety, School of Public Health, Nanjing Medical university, Nanjing, Jiangsu 211166, P.R. China

Copyright: © Liu et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
Article Number: 85
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Published online on: June 12, 2023

https://doi.org/10.3892/ijo.2023.5533
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Int J Oncol 50: [Related article:] 1321-1329, 2017; DOI: 10.3892/ijo.2017.3887

Following the publication of the above article, an interested reader drew to the authors' attention that, for the Transwell invasion assays shown in Fig. 5D on p. 1326, the images selected for the '0 μM benzidine / 0 μM curcumin' and '0 μM benzidine / 1 μM curcumin' experiments were overlapping, such that these data appeared to have been derived from the same original source. After having consulted their original data, the authors have realized that the '0 μM benzidine / 1 μM curcumin' data panel was selected incorrectly.

Figure 5

Curcumin repairs benzidine-induced SV-HUC-1 EMT via suppression of ERK5/AP-1. (A) Detection of cell viability after treating with curcumin for 4 days, curcumin at concentrations <1 μM had no impact on cell viability. Thus, dose of 1 μM was chosen for consequent experiments. (B) Curcumin attenuated benzidine-induced cell morphology changes. (C) Curcumin reversed benzidine-elevated cell migratory capacity. (D) Treatment with benzidine weakened benzidine-enhanced cell invasive ability as detected by Transwell invasive assay. (E) Cultivation with curcumin leads to elevation of expression levels of E-cadherin and ZO-1 as well as decreased expression levels of vimentin and N-cadherin as determined by western blotting. (F) Cultivation with curcumin upregulated mRNA levels of E-cadherin and ZO-1 and downregulated vimentin and N-cadherin mRNA levels. (G) Curcumin suppressed ERK5/AP-1 activity. Benzidine-induced elevation of phosphorylated ERK5, phosphorylated c-Fos and phosphorylated c-Jun was reversed by curcumin. Data are expressed as mean ± SD. **P<0.01, benzidine groups with control groups. #P<0.05, ##P<0.01, curcumin groups with respective benzidine groups.

The revised version of Fig. 5, showing the correct data for the '0 μM benzidine / 1 μM curcumin' data panel in Fig. 5D, is shown on the next page. The authors regret that this error went unnoticed prior to the publication of this article, and thank the Editor of International Journal of Oncology for allowing them the opportunity to publish this corrigendum. All the authors agree with the publication of this corrigendum; furthermore, they also apologize to the readership of the journal for any inconvenience caused.

Journals

International Journal of Molecular Medicine

International Journal of Oncology

Molecular Medicine Reports

Oncology Reports

Experimental and Therapeutic Medicine

Oncology Letters

Biomedical Reports

Molecular and Clinical Oncology

World Academy of Sciences Journal

International Journal of Functional Nutrition

International Journal of Epigenetics

Medicine International

[Corrigendum] Curcumin reverses benzidine-induced epithelial-mesenchymal transition via suppression of ERK5/AP-1 in SV-40 immortalized human urothelial cells

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