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International Journal of Oncology
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Print ISSN: 1019-6439 Online ISSN: 1791-2423
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January-2024 Volume 64 Issue 1

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Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

Medicine International

Medicine International

An International Open Access Journal Devoted to General Medicine.

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Correction Open Access

[Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway

  • Authors:
    • Xianchao Sun
    • Shiyong Xin
    • Ying Zhang
    • Liang Jin
    • Xiang Liu
    • Jiaxin Zhang
    • Wangli Mei
    • Bihui Zhang
    • Weiguo Ma
    • Lin Ye
  • View Affiliations / Copyright

    Affiliations: Department of Urology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai 200120, P.R. China, Department of Urology, The Second Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230032, P.R. China, Department of Urology, Tongxin People's Hospital, Tongxin, Ningxia 751300, P.R. China
    Copyright: © Sun et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 5
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    Published online on: November 27, 2023
       https://doi.org/10.3892/ijo.2023.5593
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Article

Int J Oncol 61: [Related article:] 110, 2022; DOI: 10.3892/ijo.2022.5400

Subsequently to the publication of the above article, an interested reader drew to the authors' attention that, concerning the Transwell assay experiments shown in Fig. 3G and I on p. 8, the data panel showing the result of the 'LNCaP / sh-CASCS11-1' experiment in Fig. 3G appeared to be overlapping with the 'LNCaP / Vector' experiment in Fig. 3I, even though the data were intended to have shown the results from differently performed experiments.

Figure 3

CASC11 promotes migration of PCa cells. (A-C) CASC11 knockdown attenuated cell migration in LNCaP cells, whereas CASC11 overexpression exerted the opposite effect. (D-F) CASC11 knockdown attenuated cell migration in 22Rv1 cells, whereas CASC11 overexpression exerted the opposite effect. (G and H) Transwell assay demonstrated that CASC11 knockdown inhibited cell migration in LNCaP and 22Rv1 cells. (I and J) Transwell assay demonstrated that CASC11 overexpression promoted cell migration in LNCaP and 22Rv1 cells. Scale bar, 100 μm. *P<0.05, **P<0.01 vs. sh-NC or vector. CASC11, cancer susceptibility candidate 11; NC, negative control; OE-CASC11, CASC11 overexpression plasmid; PCa, prostate cancer; sh, short hairpin.

After having re-examined their original data, the authors have realized that Fig. 3G and I were inadvertently assembled incorrectly. The revised version of Fig. 3, showing the correct data for the 'LNCaP / Vector' experiment in Fig. 3I, is shown on on the next page. The authors are grateful to the Editor of International Journal of Oncology for allowing them this opportunity to publish a Corrigendum, and all the authors agree with its publication. Furthermore, the authors thank the interested reader for drawing this matter to their attention, and apologize to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Sun X, Xin S, Zhang Y, Jin L, Liu X, Zhang J, Mei W, Zhang B, Ma W, Ye L, Ye L, et al: [Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway. Int J Oncol 64: 5, 2024.
APA
Sun, X., Xin, S., Zhang, Y., Jin, L., Liu, X., Zhang, J. ... Ye, L. (2024). [Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway. International Journal of Oncology, 64, 5. https://doi.org/10.3892/ijo.2023.5593
MLA
Sun, X., Xin, S., Zhang, Y., Jin, L., Liu, X., Zhang, J., Mei, W., Zhang, B., Ma, W., Ye, L."[Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway". International Journal of Oncology 64.1 (2024): 5.
Chicago
Sun, X., Xin, S., Zhang, Y., Jin, L., Liu, X., Zhang, J., Mei, W., Zhang, B., Ma, W., Ye, L."[Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway". International Journal of Oncology 64, no. 1 (2024): 5. https://doi.org/10.3892/ijo.2023.5593
Copy and paste a formatted citation
x
Spandidos Publications style
Sun X, Xin S, Zhang Y, Jin L, Liu X, Zhang J, Mei W, Zhang B, Ma W, Ye L, Ye L, et al: [Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway. Int J Oncol 64: 5, 2024.
APA
Sun, X., Xin, S., Zhang, Y., Jin, L., Liu, X., Zhang, J. ... Ye, L. (2024). [Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway. International Journal of Oncology, 64, 5. https://doi.org/10.3892/ijo.2023.5593
MLA
Sun, X., Xin, S., Zhang, Y., Jin, L., Liu, X., Zhang, J., Mei, W., Zhang, B., Ma, W., Ye, L."[Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway". International Journal of Oncology 64.1 (2024): 5.
Chicago
Sun, X., Xin, S., Zhang, Y., Jin, L., Liu, X., Zhang, J., Mei, W., Zhang, B., Ma, W., Ye, L."[Corrigendum] Long non‑coding RNA CASC11 interacts with YBX1 to promote prostate cancer progression by suppressing the p53 pathway". International Journal of Oncology 64, no. 1 (2024): 5. https://doi.org/10.3892/ijo.2023.5593
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