|
1
|
Li N, Rivéra-Bermúdez MA, Zhang M, et al:
LXR modulation blocks prostaglandin E2 production and matrix
degradation in cartilage and alleviates pain in a rat
osteoarthritis model. Proc Natl Acad Sci USA. 107:3734–3739. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Mor A, Abramson SB and Pillinger MH: The
fibroblast-like synovial cell in rheumatoid arthritis: a key player
in inflammation and joint destruction. Clin Immunol. 115:118–128.
2005. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Shen PC, Wu CL, Jou IM, et al: T helper
cells promote disease progression of osteoarthritis by inducing
macrophage inflammatory protein-1γ. Osteoarthritis Cartilage.
19:728–736. 2011.PubMed/NCBI
|
|
4
|
Choy EH and Panayi GS: Cytokine pathways
and joint inflammation in rheumatoid arthritis. New Engl J Med.
344:907–916. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Sakkas LI and Platsoucas CD: The role of T
cells in the pathogenesis of osteoarthritis. Arthritis Rheum.
56:409–424. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Sucosky P, Balachandran K, Elhammali A, Jo
H and Yoganathan AP: Altered shear stress stimulates upregulation
of endothelial VCAM-1 and ICAM-1 in a BMP-4- and
TGF-beta1-dependent pathway. Arterioscler Thromb Vasc Biol.
29:254–260. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Qureshi MH, Cook-Mills J, Doherty DE and
Garvy BA: TNF-alpha-dependent ICAM-1- and VCAM-1-mediated
inflammatory responses are delayed in neonatal mice infected with
Pneumocystis carinii. J Immunol. 171:4700–4707. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Perbal B: CCN proteins: multifunctional
signalling regulators. Lancet. 363:62–64. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Kular L, Pakradouni J, Kitabgi P, Laurent
M and Martinerie C: The CCN family: a new class of inflammation
modulators? Biochimie. 93:377–388. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Omoto S, Nishida K, Yamaai Y, et al:
Expression and localization of connective tissue growth factor
(CTGF/Hcs24/CCN2) in osteoarthritic cartilage. Osteoarthritis
Cartilage. 12:771–778. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Blaney Davidson EN, Vitters EL, Mooren FM,
et al: Connective tissue growth factor/CCN2 overexpression in mouse
synovial lining results in transient fibrosis and cartilage damage.
Arthritis Rheum. 54:1653–1661. 2006.PubMed/NCBI
|
|
12
|
Altman R, Asch E, Bloch D, et al:
Development of criteria for the classification and reporting of
osteoarthritis. Classification of osteoarthritis of the knee
Diagnostic and Therapeutic Criteria Committee of the American
Rheumatism Association. Arthritis Rheum. 29:1039–1049. 1986.
View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Wang Y, Chang H, Zou J, Jin X and Qi Z:
The effect of atorvastatin on mRNA levels of inflammatory genes
expression in human peripheral blood lymphocytes by DNA microarray.
Biomed Pharmacother. 65:118–122. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Meier FM, Frommer KW, Peters MA, et al:
Visfatin/pre-B-cell colony-enhancing factor (PBEF), a
proinflammatory and cell motility-changing factor in rheumatoid
arthritis. J Biol Chem. 287:28378–28385. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Haywood L, McWilliams DF, Pearson CI, et
al: Inflammation and angiogenesis in osteoarthritis. Arthritis
Rheum. 48:2173–2177. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Nair A, Kanda V, Bush-Joseph C, et al:
Synovial fluid from patients with early osteoarthritis modulates
fibroblast-like synoviocyte responses to toll-like receptor 4 and
toll-like receptor 2 ligands via soluble CD14. Arthritis Rheum.
64:2268–2277. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Kloesch B, Liszt M, Krehan D, Broell J,
Kiener H and Steiner G: High concentrations of hydrogen sulphide
elevate the expression of a series of pro-inflammatory genes in
fibroblast-like synoviocytes derived from rheumatoid and
osteoarthritis patients. Immunol Lett. 141:197–203. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Fu Z, Liu P, Yang D, et al:
Interleukin-18-induced inflammatory responses in synoviocytes and
chondrocytes from osteoarthritic patients. Int J Mol Med.
30:805–810. 2012.PubMed/NCBI
|
|
19
|
Bondeson J, Wainwright SD, Lauder S, Amos
N and Hughes CE: The role of synovial macrophages and
macrophage-produced cytokines in driving aggrecanases, matrix
metalloproteinases, and other destructive and inflammatory
responses in osteoarthritis. Arthritis Res Ther. 8:R1872006.
View Article : Google Scholar
|
|
20
|
Liu SC, Hsu CJ, Chen HT, Tsou HK, Chuang
SM and Tang CH: CTGF increases IL-6 expression in human synovial
fibroblasts through integrin-dependent signaling pathway. PLoS One.
7:e510972012. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Liu SC, Hsu CJ, Fong YC, Chuang SM and
Tang CH: CTGF induces monocyte chemoattractant protein-1 expression
to enhance monocyte migration in human synovial fibroblasts.
Biochim Biophys Acta. 1833:114–1124. 2013.PubMed/NCBI
|
|
22
|
Hayashida K, Nanki T, Girschick H, Yavuz
S, Ochi T and Lipsky PE: Synovial stromal cells from rheumatoid
arthritis patients attract monocytes by producing MCP-1 and IL-8.
Arthritis Res. 3:118–126. 2001. View
Article : Google Scholar : PubMed/NCBI
|
|
23
|
Hatano Y, Kasama T, Iwabuchi H, et al:
Macrophage inflammatory protein 1 alpha expression by synovial
fluid neutrophils in rheumatoid arthritis. Ann Rheum Dis.
58:297–302. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Schutyser E, Struyf S and Van Damme J: The
CC chemokine CCL20 and its receptor CCR6. Cytokine Growth Factor
Rev. 14:409–426. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Chabaud M, Page G and Miossec P: Enhancing
effect of IL-1, IL-17, and TNF-alpha on macrophage inflammatory
protein-3alpha production in rheumatoid arthritis: regulation by
soluble receptors and Th2 cytokines. J Immunol. 167:6015–6020.
2001. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Zhang J, Wu L and Qu JM: Inhibited
proliferation of human lung fibroblasts by LPS is through IL-6 and
IL-8 release. Cytokine. 54:289–295. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
García-Vicuña R, Gómez-Gaviro MV,
Domínguez-Luis MJ, et al: CC and CXC chemokine receptors mediate
migration, proliferation, and matrix metalloproteinase production
by fibroblast-like synoviocytes from rheumatoid arthritis patients.
Arthritis Rheum. 50:3866–3877. 2004.
|
|
28
|
Poole AR, Nelson F, Dahlberg L, et al:
Proteolysis of the collagen fibril in osteoarthritis. Biochem Soc
Symp. 70:115–123. 2003.PubMed/NCBI
|
|
29
|
Wu W, Billinghurst RC, Pidoux I, et al:
Sites of collagenase cleavage and denaturation of type II collagen
in aging and osteoarthritic articular cartilage and their
relationship to the distribution of matrix metalloproteinase 1 and
matrix metalloproteinase 13. Arthritis Rheum. 46:2087–2094. 2002.
View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Amos N, Lauder S, Evans A, Feldmann M and
Bondeson J: Adenoviral gene transfer into osteoarthritis synovial
cells using the endogenous inhibitor IkappaBalpha reveals that
most, but not all, inflammatory and destructive mediators are
NFkappaB dependent. Rheumatology (Oxford). 45:1201–1209. 2006.
View Article : Google Scholar
|
|
31
|
Bondeson J, Lauder S, Wainwright S, et al:
Adenoviral gene transfer of the endogenous inhibitor IkappaBalpha
into human osteoarthritis synovial fibroblasts demonstrates that
several matrix metalloproteinases and aggrecanases are nuclear
factor-kappaB-dependent. J Rheumatol. 34:523–533. 2007.
|
|
32
|
Rasheed Z, Akhtar N and Haqqi TM:
Pomegranate extract inhibits the interleukin-1β-induced activation
of MKK-3, p38α-MAPK and transcription factor RUNX-2 in human
osteoarthritis chondrocytes. Arthritis Res Ther. 12:R1952010.
|
|
33
|
Miyazawa K, Mori A, Miyata H, Akahane M,
Ajisawa Y and Okudaira H: Regulation of interleukin-1beta-induced
interleukin-6 gene expression in human fibroblast-like synoviocytes
by p38 mitogen-activated protein kinase. J Biol Chem.
273:24832–24838. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Suzuki M, Tetsuka T, Yoshida S, et al: The
role of p38 mitogen-activated protein kinase in IL-6 and IL-8
production from the TNF-alpha- or IL-1beta-stimulated rheumatoid
synovial fibroblasts. FEBS Lett. 465:23–27. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Pillinger MH, Rosenthal PB, Tolani SN, et
al: Cyclooxygenase-2-derived E prostaglandins down-regulate matrix
metalloproteinase-1 expression in fibroblast-like synoviocytes via
inhibition of extracellular signal-regulated kinase activation. J
Immunol. 171:6080–6089. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Zhang HG, Wang Y, Xie JF, et al:
Regulation of tumor necrosis factor alpha-mediated apoptosis of
rheumatoid arthritis synovial fibroblasts by the protein kinase
Akt. Arthritis Rheum. 44:1555–1567. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
El Mabrouk M, Sylvester J and Zafarullah
M: Signaling pathways implicated in oncostatin M-induced
aggrecanase-1 and matrix metalloproteinase-13 expression in human
articular chondrocytes. Biochim Biophys Acta. 1773:309–320.
2007.PubMed/NCBI
|
|
38
|
Litherland GJ, Dixon C, Lakey RL, et al:
Synergistic collagenase expression and cartilage collagenolysis are
phosphatidylinositol 3-kinase/Akt signaling-dependent. J Biol Chem.
283:14221–14229. 2008. View Article : Google Scholar : PubMed/NCBI
|
