|
1
|
Peat G, McCarney R and Croft P: Knee pain
and osteoarthritis in older adults: a review of community burden
and current use of primary health care. Ann Rheum Dis. 60:91–97.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Clouet J, Vinatier C, Merceron C,
Pot-vaucel M, Maugars Y, Weiss P, Grimandi G and Guicheux J: From
osteoarthritis treatments to future regenerative therapies for
cartilage. Drug Discov Today. 14:913–925. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Wright AA, Cook C and Abbott JH: Variables
associated with the progression of hip osteoarthritis: a systematic
review. Arthritis Rheum. 61:925–936. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Pap T, Müller-Ladner U, Gay RE and Gay S:
Fibroblast biology. Role of synovial fibroblasts in the
pathogenesis of rheumatoid arthritis. Arthritis Res. 2:361–367.
2000. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Mor A, Abramson SB and Pillinger MH: The
fibroblast-like synovial cell in rheumatoid arthritis: a key player
in inflammation and joint destruction. Clin Immunol. 115:118–128.
2005. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Dinarello CA: A clinical perspective of
IL-1β as the gatekeeper of inflammation. Eur J Immunol.
41:1203–1217. 2011.
|
|
7
|
Jotanovic Z, Mihelic R, Sestan B and
Dembic Z: Role of interleukin-1 inhibitors in osteoarthritis: an
evidence-based review. Drugs Aging. 29:343–358. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Bauernfeind F, Ablasser A, Bartok E, Kim
S, Schmid-Burgk J, Cavlar T and Hornung V: Inflammasomes: current
understanding and open questions. Cell Mol Life Sci. 68:765–783.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Martinon F, Burns K and Tschopp J: The
inflammasome: a molecular platform triggering activation of
inflammatory caspases and processing of proIL-beta. Mol Cell.
10:417–426. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Ray K: Crystal arthritis: NLRP3
inflammasome mediates crystal-induced joint inflammation and
dysfunction. Nat Rev Rheumatol. 7:6842011. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Mathews RJ, Robinson JI, Battellino M,
Wong C and Taylor JC; Biologics in Rheumatoid Arthritis Genetics
and Genomics Study Syndicate (BRAGGSS). Eyre S, Churchman SM,
Wilson AG, Isaacs JD, et al: Evidence of NLRP3-inflammasome
activation in rheumatoid arthritis (RA); genetic variants within
the NLRP3-inflammasome complex in relation to susceptibility to RA
and response to anti-TNF treatment. Ann Rheum Dis. Aug
16–2013.(Epub ahead of print).
|
|
12
|
Sakaki H, Fujiwaki T, Tsukimoto M, Kawano
A, Harada H and Kojima S: P2X4 receptor regulates P2X7
receptor-dependent IL-1β and IL-18 release in mouse bone
marrow-derived dendritic cells. Biochem Biophys Res Commun.
432:406–411. 2013.PubMed/NCBI
|
|
13
|
Uzan B, Ea HK, Launay JM, Garel JM, Champy
R, Cressent M and Lioté F: A critical role for
adrenomedullin-calcitonin receptor-like receptor in regulating
rheumatoid fibroblast-like synoviocyte apoptosis. J Immunol.
176:5548–5558. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Lioté F, Champy R, Moenner M,
Boval-Boizard B and Badet J: Elevated angiogenin levels in synovial
fluid from patients with inflammatory arthritis and secretion of
angiogenin by cultured synovial fibroblasts. Clin Exp Immunol.
132:163–168. 2003.PubMed/NCBI
|
|
15
|
Alqallaf SM, Evans BA and Kidd EJ:
Atypical P2X receptor pharmacology in two human osteoblast-like
cell lines. Br J Pharmacol. 156:1124–1135. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Klein K, Aeschlimann A, Jordan S, Gay R,
Gay S and Sprott H: ATP induced brain-derived neurotrophic factor
expression and release from osteoarthritis synovial fibroblasts is
mediated by purinergic receptor P2X4. PLoS One. 7:e366932012.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Ulmann L, Hirbec H and Rassendren F: P2X4
receptors mediate PGE2 release by tissue-resident macrophages and
initiate inflammatory pain. EMBO J. 29:2290–2300. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Troeberg L and Nagase H: Proteases
involved in cartilage matrix degradation in osteoarthritis. Biochim
Biophys Acta. 1824:133–145. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Li H, Li L, Min J, Yang H, Xu X, Yuan Y
and Wang D: Levels of metalloproteinase (MMP-3, MMP-9), NF-kappaB
ligand (RANKL), and nitric oxide (NO) in peripheral blood of
osteoarthritis (OA) patients. Clin Lab. 58:755–762. 2012.PubMed/NCBI
|
|
20
|
Levandowski CB, Mailloux CM, Ferrara TM,
Gowan K, Ben S, Jin Y, McFann KK, Holland PJ, Fain PR, Dinarello CA
and Spritz RA: NLRP1 haplotypes associated with vitiligo and
autoimmunity increase interleukin-1β processing via the NLRP1
inflammasome. Proc Natl Acad Sci USA. 110:2952–2956.
2013.PubMed/NCBI
|
|
21
|
van de Veerdonk FL, Netea MG, Dinarello CA
and Joosten LA: Inflammasome activation and IL-1β and IL-18
processing during infection. Trends Immunol. 32:110–116. 2011.
|
|
22
|
Masters SL, Gerlic M, Metcalf D, Preston
S, Pellegrini M, O’Donnell JA, McArthur K, Baldwin TM, Chevrier S,
Nowell CJ, et al: NLRP1 inflammasome activation induces pyroptosis
of hematopoietic progenitor cells. Immunity. 37:1009–1023. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Pontillo A, Girardelli M, Kamada AJ,
Pancotto JA, Donadi EA, Crovella S and Sandrin-Garcia P:
Polimorphisms in inflammasome genes are involved in the
predisposition to systemic lupus erythematosus. Autoimmunity.
45:271–278. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Sui J, Li H, Fang Y, Liu Y, Li M, Zhong B,
Yang F, Zou Q and Wu Y: NLRP1 gene polymorphism influences gene
transcription and is a risk factor for rheumatoid arthritis in han
chinese. Arthritis Rheum. 64:647–654. 2012. View Article : Google Scholar : PubMed/NCBI
|
