|
1
|
Yellon DM and Hausenloy DJ: Myocardial
reperfusion injury. N Engl J Med. 357:1121–1135. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Andrassy M, Volz HC, Igwe JC, Funke B,
Eichberger SN, Kaya Z, Buss S, Autschbach F, Pleger ST, Lukic IK,
et al: High-mobility group box-1 in ischemia-reperfusion injury of
the heart. Circulation. 117:3216–3226. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Xiong J, Xue FS, Yuan YJ, Wang Q, Liao X
and Wang WL: Cholinergic anti-inflammatory pathway: A possible
approach to protect against myocardial ischemia reperfusion injury.
Chin Med J (Engl). 123:2720–2726. 2010.PubMed/NCBI
|
|
4
|
Yang M, Chen J, Zhao J and Meng M:
Etanercept attenuates myocardial ischemia/reperfusion injury by
decreasing inflammation and oxidative stress. PLoS One.
9:e1080242014. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Frangogiannis NG, Smith CW and Entman ML:
The inflammatory response in myocardial infarction. Cardiovasc Res.
53:31–47. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Oppmann B, Lesley R, Blom B, Timans JC, Xu
Y, Hunte B, Vega F, Yu N, Wang J, Singh K, et al: Novel p19 protein
engages IL-12p40 to form a cytokine, IL-23, with biological
activities similar as well as distinct from IL-12. Immunity.
13:715–725. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Yannam GR, Gutti T and Poluektova LY:
IL-23 in infections, inflammation, autoimmunity and cancer:
Possible role in HIV-1 and AIDS. J Neuroimmune Pharmacol. 7:95–112.
2012. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Langrish CL, Chen Y, Blumenschein WM,
Mattson J, Basham B, Sedgwick JD, McClanahan T, Kastelein RA and
Cua DJ: IL-23 drives a pathogenic T cell population that induces
autoimmune inflammation. J Exp Med. 201:233–240. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Morrison PJ, Ballantyne SJ and Kullberg
MC: Interleukin-23 and T helper 17-type responses in intestinal
inflammation: From cytokines to T-cell plasticity. Immunology.
133:397–408. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Yao J, Liu L and Yang M: Interleukin-23
receptor genetic variants contribute to susceptibility of multiple
cancers. Gene. 533:21–25. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Zhu H, Li J, Wang S, Liu K, Wang L and
Huang L: Hmgb1-TLR4-IL-23-IL-17A axis promote ischemia-reperfusion
injury in a cardiac transplantation model. Transplantation.
95:1448–1454. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Liao YH, Xia N, Zhou SF, Tang TT, Yan XX,
Lv BJ, Nie SF, Wang J, Iwakura Y, Xiao H, et al: Interleukin-17A
contributes to myocardial ischemia/reperfusion injury by regulating
cardiomyocyte apoptosis and neutrophil infiltration. J Am Coll
Cardiol. 59:420–429. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Research NRCU: Guide for the care and use
of laboratory animals. National academies press (US); Washington
(DC): 1996
|
|
14
|
Hu X, Cui B, Zhou X, Xu C, Lu Z and Jiang
H: Ethyl pyruvate reduces myocardial ischemia and reperfusion
injury by inhibiting high mobility group box 1 protein in rats. Mol
Biol Rep. 39:227–231. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Xu H, Su Z, Wu J, Yang M, Penninger JM,
Martin CM, Kvietys PR and Rui T: The alarmin cytokine, high
mobility group box 1, is produced by viable cardiomyocytes and
mediates the lipopolysaccharide-induced myocardial dysfunction via
a TLR4/phosphatidylinositol 3-kinase gamma pathway. J Immunol.
184:1492–1498. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Ni J, Hu G, Xiong J, Shen J, Shen J, Yang
L, Tang M, Zhao Y, Ying G, Yu G, et al: Involvement of
interleukin-17A in pancreatic damage in rat experimental acute
necrotizing pancreatitis. Inflammation. 36:53–65. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Steffens S, Montecucco F and Mach F: The
inflammatory response as a target to reduce myocardial ischaemia
and reperfusion injury. Thromb Haemost. 102:240–247.
2009.PubMed/NCBI
|
|
18
|
Linfert D, Chowdhry T and Rabb H:
Lymphocytes and ischemia-reperfusion injury. Transplant Rev
(Orlando). 23:1–10. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Sun J, Walsh M, Villarino AV, Cervi L,
Hunter CA, Choi Y and Pearce EJ: TLR ligands can activate dendritic
cells to provide a MyD88-dependent negative signal for Th2 cell
development. J Immunol. 174:742–751. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Mosser DM and Edwards JP: Exploring the
full spectrum of macrophage activation. Nat Rev Immunol. 8:958–969.
2008. View
Article : Google Scholar : PubMed/NCBI
|
|
21
|
Wang X, Sun R, Wei H and Tian Z:
High-mobility group box 1 (HMGB1)-Toll-like receptor
(TLR)4-interleukin (IL)-23-IL-17A axis in drug-induced
damage-associated lethal hepatitis: Interaction of γδ T cells with
macrophages. Hepatology. 57:373–384. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
McAllister F, Henry A, Kreindler JL, Dubin
PJ, Ulrich L, Steele C, Finder JD, Pilewski JM, Carreno BM, Goldman
SJ, et al: Role of IL-17A, IL-17F, and the IL-17 receptor in
regulating growth-related oncogene-alpha and granulocyte
colony-stimulating factor in bronchial epithelium: Implications for
airway inflammation in cystic fibrosis. J Immunol. 175:404–412.
2005. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Stark MA, Huo Y, Burcin TL, Morris MA,
Olson TS and Ley K: Phagocytosis of apoptotic neutrophils regulates
granulopoiesis via IL-23 and IL-17. Immunity. 22:285–294. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Vanden ES, Goriely S, De Wit D, Willems F
and Goldman M: IL-23 up-regulates IL-10 and induces IL-17 synthesis
by polyclonally activated naive T cells in human. Eur J Immunol.
35:469–475. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Li L, Huang L, Vergis AL, Ye H, Bajwa A,
Narayan V, Strieter RM, Rosin DL and Okusa MD: IL-17 produced by
neutrophils regulates IFN-gamma-mediated neutrophil migration in
mouse kidney ischemia-reperfusion injury. J Clin Invest.
120:331–342. 2010. View
Article : Google Scholar : PubMed/NCBI
|
|
26
|
Cho ML, Kang JW, Moon YM, Nam HJ, Jhun JY,
Heo SB, Jin HT, Min SY, Ju JH, Park KS, et al: STAT3 and NF-kappaB
signal pathway is required for IL-23-mediated IL-17 production in
spontaneous arthritis animal model IL-1 receptor
antagonist-deficient mice. J Immunol. 176:5652–5661. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Herndon TM, Pirone DM, Tsokos GC and Chen
CS: T cell-to-T cell clustering enhances NF-kappaB activity by a
PI3K signal mediated by Cbl-b and Rho. Biochem Biophys Res Commun.
332:1133–1139. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Hunter CA: New IL-12-family members: IL-23
and IL-27, cytokines with divergent functions. Nat Rev Immunol.
5:521–531. 2005. View
Article : Google Scholar : PubMed/NCBI
|
|
29
|
Kim KW, Cho ML, Park MK, Yoon CH, Park SH,
Lee SH and Kim HY: Increased interleukin-17 production via a
phosphoinositide 3-kinase/Akt and nuclear factor kappaB-dependent
pathway in patients with rheumatoid arthritis. Arthritis Res Ther.
7:R139–R148. 2005. View
Article : Google Scholar : PubMed/NCBI
|
|
30
|
Xie L, Pi X, Wang Z, He J, Willis MS and
Patterson C: Depletion of PHD3 protects heart from
ischemia/reperfusion injury by inhibiting cardiomyocyte apoptosis.
J Mol Cell Cardiol. 80:156–165. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Hillyer P, Larché MJ, Bowman EP,
McClanahan TK, de Waal Malefyt R, Schewitz LP, Giddins G, Feldmann
M, Kastelein RA and Brennan FM: Investigating the role of the
interleukin-23/-17A axis in rheumatoid arthritis. Rheumatology
(Oxford). 48:1581–1589. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Shichita T, Sugiyama Y, Ooboshi H,
Sugimori H, Nakagawa R, Takada I, Iwaki T, Okada Y, Iida M, Cua DJ,
et al: Pivotal role of cerebral interleukin-17-producing
gammadeltaT cells in the delayed phase of ischemic brain injury.
Nat Med. 15:946–950. 2009. View
Article : Google Scholar : PubMed/NCBI
|
|
33
|
Boengler K, Hilfiker-Kleiner D, Drexler H,
Heusch G and Schulz R: The myocardial JAK/STAT pathway: From
protection to failure. Pharmacol Ther. 120:172–185. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Knight RA, Scarabelli TM and Stephanou A:
STAT transcription in the ischemic heart. Jakstat. 1:111–117.
2012.PubMed/NCBI
|
|
35
|
Jacoby JJ, Kalinowski A, Liu MG, Zhang SS,
Gao Q, Chai GX, Ji L, Iwamoto Y, Li E, Schneider M, et al:
Cardiomyocyte-restricted knockout of STAT3 results in higher
sensitivity to inflammation, cardiac fibrosis, and heart failure
with advanced age. Proc Natl Acad Sci USA. 100:pp. 12929–12934.
2003; View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Mathur AN, Chang HC, Zisoulis DG,
Stritesky GL, Yu Q, O'Malley JT, Kapur R, Levy DE, Kansas GS and
Kaplan MH: Stat3 and Stat4 direct development of IL-17-secreting Th
cells. J Immunol. 178:4901–4907. 2007. View Article : Google Scholar : PubMed/NCBI
|
