|
1
|
Jensen TS, Baron R, Haanpää M, Kalso E,
Loeser JD, Rice AS and Treede RD: A new definition of neuropathic
pain. Pain. 152:2204–2205. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Bowsher D: Neurogenic pain syndromes and
their management. Br Med Bull. 47:644–666. 1991. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Carter GT and Galer BS: Advances in the
management of neuropathic pain. Phys Med Rehabil Clin N Am.
12:447–459. 2001.PubMed/NCBI
|
|
4
|
Toth C, Lander J and Wiebe S: The
prevalence and impact of chronic pain with neuropathic pain
symptoms in the general population. Pain Med. 10:918–929. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Mcdermott AM, Toelle TR, Rowbotham DJ,
Schaefer CP and Dukes EM: The burden of neuropathic pain: Results
from a cross-sectional survey. Eur J Pain. 10:127–135. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
O'Connor AB: Neuropathic pain:
Quality-of-life impact, costs and cost effectiveness of therapy.
Pharmacoeconomics. 27:95–112. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Park GH, Jeon SJ, Ryu JR, Choi MS, Han SH,
Yang SI, Ryu JH, Cheong JH, Shin CY and Ko KH: Essential role of
mitogen-activated protein kinase pathways in protease activated
receptor 2-mediated nitric-oxide production from rat primary
astrocytes. Nitric Oxide. 21:110–119. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Bao Y, Hou W and Hua B: Protease-activated
receptor 2 signalling pathways: A role in pain processing. Expert
Opin Ther Targets. 18:15–27. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Huang ZJ, Li HC, Cowan AA, Liu S, Zhang YK
and Song XJ: Chronic compression or acute dissociation of dorsal
root ganglion induces cAMP-dependent neuronal hyperexcitability
through activation of PAR2. Pain. 153:1426–1437. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Lee B, Cho H, Jung J, Yang YD, Yang DJ and
Oh U: Anoctamin 1 contributes to inflammatory and nerve-injury
induced hypersensitivity. Mol Pain. 10:52014. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
García G, Martínezrojas VA, Rochagonzález
HI, Granadossoto V and Murbartián J: Evidence for the participation
of Ca(2+)-activated chloride channels in formalin-induced acute and
chronic nociception. Brain Res. 1579:35–44. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Yang YD, Cho H, Koo JY, Tak MH, Cho Y,
Shim WS, Park SP, Lee J, Lee B, Kim BM, et al: TMEM16A confers
receptor-activated calcium-dependent chloride conductance. Nature.
455:1210–1215. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Cho H, Yang YD, Lee J, Lee B, Kim T, Jang
Y, Back SK, Na HS, Harfe BD, Wang F, et al: The calcium-activated
chloride channel anoctamin 1 acts as a heat sensor in nociceptive
neurons. Nat Neurosci. 15:1015–1021. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Matsuba S, Niwa S, Muraki K, Kanatsuka S,
Nakazono Y, Hatano N, Fujii M, Zhan P, Suzuki T and Ohya S:
Downregulation of Ca2+-activated Cl-channel TMEM16A by the
inhibition of histone deacetylase in TMEM16A-expressing cancer
cells. J Pharmacol Exp Ther. 351:510–518. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Zhao L, Li LI, MA KT, Wang Y, Li J, Shi
WY, Zhu HE, Zhang ZS and Si JQ: NSAIDs modulate GABA-activated
currents via Ca2+-activated Cl-channels in rat dorsal root ganglion
neurons. Exp Ther Med. 11:1755–1761. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Li L, Zhao L, Wang Y, Ma KT, Shi WY, Wang
YZ and Si JQ: PKCε mediates substance P inhibition of GABAA
receptors-mediated current in rat dorsal root ganglion. J Huazhong
Univ Sci Technolog Med Sci. 35:1–9. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Han YY, Huang Z, Wang YP, et al: The study
of the expression of potassium sodium chloride transporter on DRG
neurons in CCI model rat. Chinese Pain Med. 22:583–90. 2016.(In
Chinese).
|
|
18
|
Miao XR, Gao XF, Wu JX, Lu ZJ, Huang ZX,
Li XQ, He C and Yu WF: Bilateral downregulation of Nav1.8 in dorsal
root ganglia of rats with bone cancer pain induced by inoculation
with Walker 256 breast tumor cells. BMC Cancer. 10:2162010.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Yang W, Si-yuan L, Ke-tao M, Jun-qiang S,
Lei Z, Zhong-Shuang Z, He Z and Li L: Changes in presynaptic
inhibition and the second message system of neuropathic pain model
in rats. China J Mod Med. 22:9–14. 2012.(In Chinese).
|
|
20
|
Mcmahon SB, Bennett DLH and Bevan S:
Inflammatory mediators and modulators of pain. Churchill
Livingstone (Elsevier Health Sciences). 1–72. 2006.
|
|
21
|
Bennett GJ and Xie YK: A peripheral
mononeuropathy in rat that produces disorders of pain sensation
like those seen in man. Pain. 33:87–107. 1988. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Striggow F, Riekburchardt M, Kiesel A,
Schmidt W, Henrich-Noack P, Breder J, Krug M, Reymann KG and Reiser
G: Four different types of protease-activated receptors are widely
expressed in the brain and are up-regulated in hippocampus by
severe ischemia. Eur J Neurosci. 14:595–608. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Chen Y, Yang C and Wang ZJ:
Proteinase-activated receptor 2 sensitizes transient receptor
potential vanilloid 1, transient receptor potential vanilloid 4 and
transient receptor potential ankyrin 1 in paclitaxel-induced
neuropathic pain. Neuroscience. 193:440–451. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Fan Y, Chen J, Ye J, Yan H and Cai Y:
Proteinase-activated receptor 2 modulates corticotropin releasing
hormone-induced brain-derived neurotrophic factor release from
microglial cells. Cell Biol Int. 38:92–96. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Spicarova D, Nerandzic V and Palecek J:
Update on the role of spinal cord TRPV1 receptors in pain
modulation. Physiol Res. 63 Suppl 1:S225–S236. 2014.PubMed/NCBI
|
|
26
|
Spicarova D, Adamek P, Kalynovska N,
Mrozkova P and Palecek J: TRPV1 receptor inhibition decreases
CCL2-induced hyperalgesia. Neuropharmacology. 81:75–84. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Uchytilova E, Spicarova D and Palecek J:
TRPV1 antagonist attenuates postoperative hypersensitivity by
central and peripheral mechanisms. Mol Pain. 10:672014. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Lam DK, Dang D, Zhang J, Dolan JC and
Schmidt BL: Novel animal models of acute and chronic cancer pain: a
pivotal role for PAR2. J Neurosci. 32:14178–14183. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Liu S, Liu YP, Yue DM and Liu GJ:
Protease-activated receptor 2 in dorsal root ganglion contributes
to peripheral sensitization of bone cancer pain. Eur J Pain.
18:326–337. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Stephan AB, Shum EY, Hirsh S, Cygnar KD,
Reisert J and Zhao H: ANO2 is the cilial calcium-activated chloride
channel that may mediate olfactory amplification. Proc Natl Acad
Sci USA. 106:pp. 11776–11781. 2009; View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Liu B, Linley JE, Du X, Zhang X, Ooi L,
Zhang H and Gamper N: The acute nociceptive signals induced by
bradykinin in rat sensory neurons are mediated by inhibition of
M-type K+ channels and activation of Ca2+-activated Cl-channels. J
Clin Invest. 120:1240–1252. 2010. View
Article : Google Scholar : PubMed/NCBI
|
|
32
|
Deba F and Bessac BF: Anoctamin-1 Cl(−)
channels in nociception: Activation by an N-aroylaminothiazole and
capsaicin and inhibition by T16A (inh)-A01. Mol Pain. 11:552015.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Ferrera L, Caputo A and Galietta LJ:
TMEM16A protein: A new identity for Ca(2+)-dependent Cl(−)
channels. Physiology (Bethesda). 25:357–363. 2010.PubMed/NCBI
|
|
34
|
Bulley S, Neeb ZP, Burris SK, Bannister
JP, Thomas-Gatewood CM, Jangsangthong W and Jaggar JH: TMEM16A
channels contribute to myogenic constriction in cerebral arteries.
Circ Res. 111:1027–1036. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Chun H, Cho H, Choi J, Lee J, Kim SM, Kim
H and Oh U: Protons inhibit anoctamin 1 by competing with calcium.
Cell Calcium. 58:431–441. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Sones WR, Davis AJ, Leblanc N and
Greenwood IA: Cholesterol depletion alters amplitude and
pharmacology of vascular calcium-activated chloride channels.
Cardiovasc Res. 87:476–484. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Cipriani G, Serboiu CS, Gherghiceanu M,
Faussone-Pellegrini MS and Vannucchi MG: NK receptors, Substance
PAno1 expression and ultrastructural features of the muscle coat in
Cav-1(−/-) mouse ileum. J Cell Mol Med. 15:2411–2420. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Tian Y, Kongsuphol P, Hug M, Ousingsawat
J, Witzgall R, Schreiber R and Kunzelmann K: Calmodulin-dependent
activation of the epithelial calcium-dependent chloride channel
TMEM16A. FASEB J. 25:1058–1068. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Jung J, Nam JH, Park HW, Oh U, Yoon JH and
Lee MG: Dynamic modulation of ANO1/TMEM16A HCO3(−) permeability by
Ca2+/calmodulin. Proc Natl Acad Sci USA. 110:pp. 360–365. 2013;
View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Vocke K, Dauner K, Hahn A, Ulbrich A,
Broecker J, Keller S, Frings S and Möhrlen F: Calmodulin-dependent
activation and inactivation of anoctamin calcium-gated chloride
channels. J Gen Physiol. 142:381–404. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Tian Y, Schreiber R, Wanitchakool P,
Kongsuphol P, Sousa M, Uliyakina I, Palma M, Faria D,
Traynor-Kaplan AE, Fragata JI, et al: Control of TMEM16A by
INO-4995 and other inositolphosphates. Br J Pharmacol. 168:253–265.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Pritchard HA, Leblanc N, Albert AP and
Greenwood IA: Inhibitory role of phosphatidylinositol
4,5-bisphosphate on TMEM16A-encoded calcium-activated chloride
channels in rat pulmonary artery. Br J Pharmacol. 171:4311–4321.
2014. View Article : Google Scholar : PubMed/NCBI
|
