|
1
|
Salvesen GS and Dixit VM: Caspase
activation: The induced-proximity model. Proc Natl Acad Sci USA.
96:pp. 10964–10967. 1999; View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Narula J, Haider N, Virmani R, DiSalvo TG,
Kolodgie FD, Hajjar RJ, Schmidt U, Semigran MJ, Dec GW and Khaw BA:
Apoptosis in myocytes in end-stage heart failure. N Engl J Med.
335:1182–1189. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Kerr JF, Wyllie AH and Currie AR:
Apoptosis: A basic biological phenomenon with wide-ranging
implications in tissue kinetics. Br J Cancer. 26:239–257. 1972.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Colucci WS: Apoptosis in the heart. N Engl
J Med. 335:1224–1226. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Wencker D, Chandra M, Nguyen K, Miao W,
Garantziotis S, Factor SM, Shirani J, Armstrong RC and Kitsis RN: A
mechanistic role for cardiac myocyte apoptosis in heart failure. J
Clin Invest. 111:1497–1504. 2003. View
Article : Google Scholar : PubMed/NCBI
|
|
6
|
Ma TK, Kam KK, Yan BP and Lam YY:
Renin-angiotensin-aldosterone system blockade for cardiovascular
diseases: Current status. Br J Pharmacol. 160:1273–1292. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Suzuki J, Iwai M, Nakagami H, Wu L, Chen
R, Sugaya T, Hamada M, Hiwada K and Horiuchi M: Role of angiotensin
II-regulated apoptosis through distinct AT1 and AT2 receptors in
neointimal formation. Circulation. 106:847–853. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Demers C, Mody A, Teo KK and McKelvie RS:
ACE inhibitors in heart failure: What more do we need to know? Am J
Cardiovasc Drugs. 5:351–359. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Smith WH and Ball SG: ACE inhibitors in
heart failure: An update. Basic Res Cardiol. 95 Suppl 1:I8–I14.
2000. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Zhao H, Qi G, Han Y, Shen X, Yao F, Xuan
C, Gu Y, Qian SY, Zeng Q, O'Rourke ST, et al:
20-hydroxyeicosatetraenoic acid is a key mediator of angiotensin
II-induced apoptosis in cardiac myocytes. J Cardiovasc Pharmacol.
66:86–95. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Schröder D, Heger J, Piper HM and Euler G:
Angiotensin II stimulates apoptosis via TGF-beta1 signaling in
ventricular cardiomyocytes of rat. J Mol Med (Berl). 84:975–983.
2006. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Grishko V, Pastukh V, Solodushko V,
Gillespie M, Azuma J and Schaffer S: Apoptotic cascade initiated by
angiotensin II in neonatal cardiomyocytes: Role of DNA damage. Am J
Physiol Heart Circ Physiol. 285:H2364–H2372. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Cakmak HA, Coskunpinar E, Ikitimur B,
Barman HA, Karadag B, Tiryakioglu NO, Kahraman K and Vural VA: The
prognostic value of circulating microRNAs in heart failure:
Preliminary results from a genome-wide expression study. J
Cardiovasc Med (Hagerstown). 16:431–437. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Cheng Y, Liu X, Zhang S, Lin Y, Yang J and
Zhang C: MicroRNA-21 protects against the H(2)O(2)-induced injury
on cardiac myocytes via its target gene PDCD4. J Mol Cell Cardiol.
47:5–14. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Flynt AS, Li N, Thatcher EJ,
Solnica-Krezel L and Patton JG: Zebrafish miR-214 modulates
Hedgehog signaling to specify muscle cell fate. Nat Genet.
39:259–263. 2007. View
Article : Google Scholar : PubMed/NCBI
|
|
16
|
Lv G, Shao S, Dong H, Bian X, Yang X and
Dong S: MicroRNA-214 protects cardiac myocytes against H2O2-induced
injury. J Cell Biochem. 115:93–101. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Valastyan S, Chang A, Benaich N, Reinhardt
F and Weinberg RA: Activation of miR-31 function in
already-established metastases elicits metastatic regression. Genes
Dev. 25:646–659. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Pacurari M and Tchounwou PB: Role of
MicroRNAs in renin-angiotensin-aldosterone system-mediated
cardiovascular inflammation and remodeling. Int J Inflam.
2015:1015272015. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Rayner KJ: MicroRNA-155 in the heart: The
right time at the right place in the right cell. Circulation.
131:1533–1535. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Elton TS, Selemon H, Elton SM and
Parinandi NL: Regulation of the MIR155 host gene in physiological
and pathological processes. Gene. 532:1–12. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Jeppesen PL, Christensen GL, Schneider M,
Nossent AY, Jensen HB, Andersen DC, Eskildsen T, Gammeltoft S,
Hansen JL and Sheikh SP: Angiotensin II type 1 receptor signalling
regulates microRNA differentially in cardiac fibroblasts and
myocytes. Br J Pharmacol. 164:394–404. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Eskildsen TV, Schneider M, Sandberg MB,
Skov V, Brønnum H, Thomassen M, Kruse TA, Andersen DC and Sheikh
SP: The microRNA-132/212 family fine-tunes multiple targets in
Angiotensin II signalling in cardiac fibroblasts. J Renin
Angiotensin Aldosterone Syst. 16:1288–1297. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Mlcochova J, Faltejskova-Vychytilova P,
Ferracin M, Zagatti B, Radova L, Svoboda M, Nemecek R, John S, Kiss
I, Vyzula R, et al: MicroRNA expression profiling identifies
miR-31-5p/3p as associated with time to progression in wild-type
RAS metastatic colorectal cancer treated with cetuximab.
Oncotarget. 6:38695–38704. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Liu X, Meng H, Jiang C, Yang S, Cui F and
Yang P: Differential microRNA expression and regulation in the rat
model of post-infarction heart failure. PLoS One. 11:e01609202016.
View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Cigola E, Kajstura J, Li B, Meggs LG and
Anversa P: Angiotensin II activates programmed myocyte cell death
in vitro. Exp Cell Res. 231:363–371. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Kajstura J, Mansukhani M, Cheng W, Reiss
K, Krajewski S, Reed JC, Quaini F, Sonnenblick EH and Anversa P:
Programmed cell death and expression of the protooncogene bcl-2 in
myocytes during postnatal maturation of the heart. Exp Cell Res.
219:110–121. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Sadoshima J and Izumo S: Molecular
characterization of angiotensin II-induced hypertrophy of cardiac
myocytes and hyperplasia of cardiac fibroblasts. Critical role of
the AT1 receptor subtype. Circ Res. 73:413–423. 1993. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Kajstura J, Cigola E, Malhotra A, Li P,
Cheng W, Meggs LG and Anversa P: Angiotensin II induces apoptosis
of adult ventricular myocytes in vitro. J Mol Cell Cardiol.
29:859–870. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Goussev A, Sharov VG, Shimoyama H,
Tanimura M, Lesch M, Goldstein S and Sabbah HN: Effects of ACE
inhibition on cardiomyocyte apoptosis in dogs with heart failure.
Am J Physiol. 275:H626–H631. 1998.PubMed/NCBI
|
|
30
|
Bäcklund T, Palojoki E, Saraste A,
Grönholm T, Eriksson A, Lakkisto P, Vuolteenaho O, Nieminen MS,
Voipio-Pulkki LM, Laine M and Tikkanen I: Effect of vasopeptidase
inhibitor omapatrilat on cardiomyocyte apoptosis and ventricular
remodeling in rat myocardial infarction. Cardiovasc Res.
57:727–737. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Liu CH, Gong Z, Liang ZL, Liu ZX, Yang F,
Sun YJ, Ma ML, Wang YJ, Ji CR, Wang YH, et al: Arrestin-biased AT1R
agonism induces acute catecholamine secretion through TRPC3
coupling. Nat Commun. 8:143352017. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Prech M, Marszałek A, Schröder J, Filas V,
Lesiak M, Jemielity M, Araszkiewicz A and Grajek S: Apoptosis as a
mechanism for the elimination of cardiomyocytes after acute
myocardial infarction. Am J Cardiol. 105:1240–1245. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Kim NH and Kang PM: Apoptosis in
cardiovascular diseases: Mechanism and clinical implications.
Korean Circ J. 40:299–305. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Thornberry NA and Lazebnik Y: Caspases:
Enemies within. Science. 281:1312–1316. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Sadowski-Debbing K, Coy JF, Mier W, Hug H
and Los M: Caspases-their role in apoptosis and other physiological
processes as revealed by knock-out studies. Arch Immunol Ther Exp
(Warsz). 50:19–34. 2002.PubMed/NCBI
|
|
36
|
Crow MT, Mani K, Nam YJ and Kitsis RN: The
mitochondrial death pathway and cardiac myocyte apoptosis. Circ
Res. 95:957–970. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Hünten S, Siemens H, Kaller M and
Hermeking H: The p53/microRNA network in cancer: Experimental and
bioinformatics approaches. Adv Exp Med Biol. 774:77–101. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Feng Z, Zhang C, Wu R and Hu W: Tumor
suppressor p53 meets microRNAs. J Mol Cell Biol. 3:44–50. 2011.
View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Tazawa H, Tsuchiya N, Izumiya M and
Nakagama H: Tumor-suppressive miR-34a induces senescence-like
growth arrest through modulation of the E2F pathway in human colon
cancer cells. Proc Natl Acad Sci USA. 104:pp. 15472–15477. 2007;
View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Raver-Shapira N, Marciano E, Meiri E,
Spector Y, Rosenfeld N, Moskovits N, Bentwich Z and Oren M:
Transcriptional activation of miR-34a contributes to p53-mediated
apoptosis. Mol Cell. 26:731–743. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Hu W, Chan CS, Wu R, Zhang C, Sun Y, Song
JS, Tang LH, Levine AJ and Feng Z: Negative regulation of tumor
suppressor p53 by microRNA miR-504. Mol Cell. 38:689–699. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Le MT, Teh C, Shyh-Chang N, Xie H, Zhou B,
Korzh V, Lodish HF and Lim B: MicroRNA-125b is a novel negative
regulator of p53. Genes Dev. 23:862–876. 2009. View Article : Google Scholar : PubMed/NCBI
|
