|
1
|
Ashley NT, Weil ZM and Nelson RJ:
Inflammation: mechanisms, costs, and natural variation. Ann Rev
Ecol Evol Syst. 43:385–406. 2012. View Article : Google Scholar
|
|
2
|
Medzhitov R: Origin and physiological
roles of inflammation. Nature. 454:428–435. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Graham BS: Biological challenges and
technological opportunities for respiratory syncytial virus vaccine
development. Immunol Rev. 239:149–166. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Martinon F, Mayor A and Tschopp J: The
inflammasomes: guardians of the body. Annu Rev Immunol. 27:229–265.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Nathan C: Points of control in
inflammation. Nature. 420:846–852. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Adefuye A and Sales K: Regulation of
inflammatory pathways in cancer and infectious disease of the
cervix. Scientifica (Cairo). 2012:5481502012.
|
|
7
|
Coussens LM and Werb Z: Inflammation and
cancer. Nature. 420:860–867. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Schottenfeld D and Beebe-Dimmer JL:
Advances in cancer epidemiology: understanding causal mechanisms
and the evidence for implementing interventions. Ann Rev Public
Health. 26:37–60. 2005. View Article : Google Scholar
|
|
9
|
Akagi K, Li J, Broutian TR, Padilla-Nash
H, et al: Genome-wide analysis of HPV integration in human cancers
reveals recurrent, focal genomic instability. Genome Res.
24:185–199. 2014. View Article : Google Scholar :
|
|
10
|
Trottier H and Franco EL: The epidemiology
of genital human papillomavirus infection. Vaccine. 24(Suppl 1):
S1–S15. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Muñoz N, Castellsagué X, de González AB
and Gissmann L: Chapter 1: HPV in the etiology of human cancer.
Vaccine. 24(Suppl 3): S1–S10. 2006. View Article : Google Scholar
|
|
12
|
Mazibrada J, Rittà M, Mondini M, et al:
Interaction between inflammation and angiogenesis during different
stages of cervical carcinogenesis. Gynecol Oncol. 108:112–120.
2008. View Article : Google Scholar
|
|
13
|
Paghini BC, Abdalla DR, Barcelos AC, et
al: Local cytokine profiles of patients with cervical
intraepithelial and invasive neoplasia. Hum Immunol. 73:920–926.
2012. View Article : Google Scholar
|
|
14
|
Graham CS, Wells A, Liu T, et al:
Antigen-specific immune responses and liver histology in HIV and
hepatitis C coinfection. AIDS. 19:767–773. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Tang T, Wong HK, Gu W, et al: MicroRNA-182
plays an onco-miRNA role in cervical cancer. Gynecol Oncol.
129:199–208. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Viviani B, Bartesaghi S, Gardoni F, et al:
Interleukin-1beta enhances NMDA receptor-mediated intracellular
calcium increase through activation of the Src family of kinases. J
Neurosci. 23:8692–8700. 2003.PubMed/NCBI
|
|
17
|
Pasparakis M: Regulation of tissue
homeostasis by NF-kappaB signaling: implications for inflammatory
diseases. Nat Rev Immunol. 9:778–788. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Lou JL, Maeda S, Hsu LC, et al: Inhibition
of NF-kappaB in cancer cells converts inflammation-induced tumor
growth mediated by TNFalpha to TRAIL-mediated tumor regression.
Cancer Cell. 6:297–305. 2004. View Article : Google Scholar
|
|
19
|
Hussain SP, Hofseth LJ and Harris CC:
Radical causes of cancer. Nat Rev Cancer. 3:276–285. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Chen WL and Konkel JE: TGF-beta and
‘adaptive’ Foxp3(+) regulatory T cells. J Mol Cell Biol. 2:30–36.
2010. View Article : Google Scholar :
|
|
21
|
Derynck R, Akhurst RJ and Balmain A:
TGF-beta signaling in tumor suppression and cancer progression. Nat
Genet. 29:117–129. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Ishihara K and Hirano T: Il-6 autoimmune
disease and chronic inflammatory proliferative disease. Cytokine
Growth Factor Rev. 13:357–368. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Jenkins BJ, Roberts AW, Greenhill CJ, et
al: Pathologic consequences of STAT3 hyperactivation by IL-6 and
IL-11 during hematopoiesis and lymphopoiesis. Blood. 109:2380–2388.
2007. View Article : Google Scholar
|
|
24
|
Vicari AP and Trinchieri G: Interleukin-10
in viral diseases and cancer: exiting the labyrinth? Immunol Rev.
202:223–236. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Mocellin S, Marincola FM and Young HA:
Interleukin-10 and the immune response against cancer: a
counterpoint. J Leukoc Biol. 78:1043–1051. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Sadler AJ and Williams BR:
Interferon-inducible antiviral effectors. Nature Rev Immunol.
8:559–568. 2008. View Article : Google Scholar
|
|
27
|
González-Navajas JM, Lee J, David M and
Raz E: Immunomodulatory functions of type I interferons. Nat Rev
Immunol. 12:125–135. 2012.PubMed/NCBI
|
|
28
|
Guarda G, Braun M, Staehli F, et al: Type
I interferon inhibits interleukin-1 production and inflammasome
activation. Immunity. 34:213–223. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Schroder K, Hertzog PJ, Ravasi T and Hume
DA: Interferon-gamma: an overview of signals, mechanisms and
functions. J Leukoc Biol. 75:163–189. 2004. View Article : Google Scholar
|
|
30
|
Schoenborn JR and Wilson CB: Regulation of
Interferon-gamma during innate and adaptive immune responses. Adv
Immunol. 96:41–101. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Ruddy MJ, Wong GC, Liu XK, et al:
Functional cooperation between interleukin-17 and tumor necrosis
factor-alpha is mediated by CCAAT/enhancer-binding protein family
members. J Biol Chem. 279:2559–2567. 2004. View Article : Google Scholar
|
|
32
|
Zhang Y, Ma D, Zhang Y, et al: The
imbalance of Th17/Treg in patients with uterine cervical cancer.
Clin Chim Acta. 412:894–900. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Chen Z, Ding J, Pang N, et al: The
Th17/Treg balance and the expression of related cytokines in Uygur
cervical cancer patients. Diagn Pathol. 8:612013. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Cho ML, Kang JW, Moon YM, et al: STAT3 and
NF-kappaB signal pathway is required for IL-23-mediated IL-17
production in spontaneous arthritis animal model IL-1 receptor
antagonist-deficient mice. J Immunol. 176:5652–5661. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Hao JS and Shan BE: Immune enhancement and
anti-tumour activity of IL-23. Cancer Immunol Immunother.
55:1426–1431. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Biet F, Locht C and Kremer L:
Immunoregulatory functions of interleukin 18 and its role in
defense against bacterial pathogens. J Mol Med (Berl). 80:147–162.
2002. View Article : Google Scholar
|
|
37
|
Nakanishi K: Regulation of Th1 and Th2
immune responses by IL-18. Kekkaku. 77:87–93. 2002.(Japanese).
PubMed/NCBI
|
|
38
|
Reddy P and Ferrara JL: Role of
interleukin-18 in acute graft-vs-host disease. J Lab Clin Med.
141:365–371. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Medzhitov R: Origin and physiological
roles of inflammation. Nature. 454:428–435. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Serhan CN and Savill J: Resolution of
inflammation: the beginning programs the end. Nat Immunol.
6:1191–1197. 2005. View
Article : Google Scholar : PubMed/NCBI
|
|
41
|
Akira S, Uematsu S and Takeuchi O:
Pathogen recognition and innate immunity. Cell. 124:783–801. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Mogensen TH: Pathogen recognition and
inflammatory signaling in innate immune defenses. Clin Microbiol
Rev. 22:240–273. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
43
|
Roach JC, Glusman G, Rowen L, et al: The
evolution of vertebrate Toll-like receptors. Proc Natl Acad Sci
USA. 102:9577–9582. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
44
|
Proell M, Riedl SJ, Fritz JH, et al: The
Nod-like receptor (NLR) family: a tale of similarities and
differences. PLoS One. 3:e21192008. View Article : Google Scholar : PubMed/NCBI
|
|
45
|
Friedman R and Hughes AL: Molecular
evolution of the NF-kappaB signaling system. Immunogenetics.
53:964–974. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
46
|
Ahmed AU: An overview of inflammation:
mechanism and consequences. Front Biol. 6:274–281. 2011.
|
|
47
|
Welsh RM, Bahl K, Marshall HD and Urban
SL: Type 1 interferons and antiviral CD8 T-Cell responses. PLoS
Pathog. 8:e10023522012. View Article : Google Scholar : PubMed/NCBI
|
|
48
|
Sica A and Bronte V: Altered macrophage
differentiation and immune dysfunction in tumor development. J Clin
Invest. 117:1155–1166. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
49
|
Liu CY, Xu JY, Shi XY, et al: M2-polarized
tumor-associated macrophages promoted epithelial-mesenchymal
transition in pancreatic cancer cells, partially through TLR4/IL-10
signaling pathway. Lab Invest. 93:844–854. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
50
|
Lin WW and Karin M: A cytokine-mediated
link between innate immunity, inflammation and cancer. J Clin
Invest. 117:1175–1183. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
51
|
Sieve AN, Meeks KD, Lee S and Berg RE: A
novel immunoregulatory function for IL-23: inhibition of
IL-12-dependent IFN-γ production. Eur J Immunol. 40:2236–2247.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
52
|
Rubtsov YP, Rasmussen JP, Chi EY, et al:
Regulatory T cell-derived interleukin-10 limits inflammation at
environmental interfaces. Immunity. 28:546–558. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
53
|
Ouyang W, Kolls JK and Zheng Y: The
biological functions of T helper 17 cell effector cytokines in
inflammation. Immunity. 28:454–467. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
54
|
Koch MA, Trucker-Heard G, Perduc NR, et
al: The transcription factor T-bet controls regulatory T cell
homeostasis and function during type 1 inflammation. Nat Immunol.
10:595–602. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
55
|
Anthony MN, Gardner L, Marks G, et al:
Factors associated with use of HIV primary care among persons
recently diagnosed with HIV: examination of variables from the
behavioural model of health-care utilization. AIDS Care.
19:195–202. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
56
|
Anthony RM, Rutitzky LI, Urban JF Jr,
Stadecker MJ and Gause WC: Protective immune mechanisms in helminth
infection. Nat Rev Immunol. 7:975–987. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
57
|
Graham AL: When T-helper cells don’t help:
immunopathology during concomitant infection. Q Rev Biol.
77:409–434. 2002. View
Article : Google Scholar
|
|
58
|
Suvas S, Azkur AK, Kim BS, et al:
CD4+CD25+ regulatory T cells control the severity of viral
immunoinflammatory lesions. J Immunol. 172:4123–4132. 2004.
View Article : Google Scholar : PubMed/NCBI
|
|
59
|
Belkaid Y and Tarbell K: Regulatory T
Cells in the Control of host-microorganism interactions. Ann Rev
Immunol. 27:551–589. 2009. View Article : Google Scholar
|
|
60
|
Korn T, Bettelli E, Oukka M and Kuchroo
VK: IL-17 and Th17 cells. Ann Rev Immunol. 27:485–517. 2009.
View Article : Google Scholar
|
|
61
|
Jabbour HN, Sales KJ, Catalano RD and
Norman JE: Inflammatory pathways in female reproductive health and
disease. Reproduction. 138:903–919. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
62
|
Maeda K, Yoshida K, Ichimiya I and Suzuki
M: Dexamethasone inhibits tumor necrosis factor-alpha-induced
cytokine secretion from spiral ligament fibrocytes. Hear Res.
202:154–160. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
63
|
Sakurai F, Nakamura S, Akitomo K, Shibata
H, et al: Transduction properties of adenovirus serotype 35 vectors
after intravenous administration into nonhuman primates. Mol Ther.
16:726–733. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
64
|
Mantovani A, Allavena P, Sica A and
Balkwill F: Cancer-related inflammation. Nature. 454:436–444. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
65
|
Karin M and Greten FR: NF-kappaB: linking
inflammation and immunity to cancer development and progression.
Nat Rev Immunol. 5:749–759. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
66
|
Karin M: Nuclear factor-kappaB in cancer
development and progression. Nature. 441:431–436. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
67
|
Karin M: NF-kappaB and cancer: mechanisms
and targets. Mol Carcinog. 45:355–361. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
68
|
Karin M, Cao Y, Greten FR and Li ZW:
NF-kappaB in cancer: from innocent bystander to major culprit. Nat
Rev Cancer. 2:301–310. 2002. View
Article : Google Scholar : PubMed/NCBI
|
|
69
|
Lou L, Li DQ, Corrales RM and Pflugfelder
SC: Hyperosmolar saline is a proinflammatory stress on the mouse
ocular surface. Eye Contact Lens. 31:186–193. 2005. View Article : Google Scholar
|
|
70
|
Parkin DM and Bray F: Chapter 2: The
burden of HPV-related cancers. Vaccine. 24(Suppl 3): S11–S25. 2006.
View Article : Google Scholar
|
|
71
|
Shukla S, Bharti AC, Mahata S, et al:
Infection of human papillomavirus in cancers of different human
organ sites. Indian J Med Res. 130:222–233. 2009.PubMed/NCBI
|
|
72
|
Moody CA and Laimins LA: Human
papillomavirus oncoproteins: pathways to transformation. Nat Rev
Cancer. 10:550–560. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
73
|
Fernandes JV, Araújo JMG and Fernandes
TAAM: Biology and natural history of human papillomavirus
infection. Open Access J Clin Trials. 5:1–12. 2013. View Article : Google Scholar
|
|
74
|
Hernádi Z, Gazdag L, Szoke K, Sápy T,
Krasznai ZT and Kónya J: Duration of HPV-associated risk for
high-grade cervical intraepithelial neoplasia. Eur J Obstet Gynecol
Reprod Biol. 125:114–119. 2006. View Article : Google Scholar
|
|
75
|
Fernandes JV, Fernandes TAAM and de Araújo
JMG: Human papillomavirus infection in adolescents. Handbook on
Human Papillomavirus: Prevalence, Detection & Management. Smith
HB: Nova Science Publishers; New York, NY: pp. 181–212. 2013
|
|
76
|
Jemal A, Bray F, Center MM, et al: Global
cancer statistics. CA Cancer J Clin. 61:69–90. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
77
|
Stanley M: Immune responses to human
papillomavirus. Vaccine. 24(Suppl 1): S16–S22. 2006. View Article : Google Scholar
|
|
78
|
Smith JS, Herrero R, Bosetti C, et al:
Herpes simplex virus-2 as a human papillomavirus cofactor in the
etiology of invasive cervical cancer. J Natl Cancer Inst.
94:1604–1613. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
79
|
Smith JS, Muñoz N, Herrero R, et al:
Evidence for Chlamydia trachomatis as a human papillomavirus
cofactor in the etiology of invasive cervical cancer in Brazil and
the Philippines. J Infect Dis. 185:324–331. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
80
|
de Abreu AL, Nogara PR, Souza RP, et al:
Molecular detection of HPV and Chlamydia trachomatis infections in
Brazilian women with abnormal cervical cytology. Am J Trop Med Hyg.
87:1149–1151. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
81
|
Scott M, Stites DP and Mosciki AB: Th1
cytokine patterns in cervical human papillomavirus infection. Clin
Diagn Lab Immunol. 6:751–755. 1999.PubMed/NCBI
|
|
82
|
Adefuye A and Sales K: Regulation of
inflammatory pathways in cancer and infectious disease of the
cervix. Scientifica (Cairo). 2012:5481502012.
|
|
83
|
Ambros V and Chen X: The regulation of
genes and genomes by small RNAs. Development. 134:1635–1641. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
84
|
Niwa R and Slack FJ: The evolution of
animal microRNA function. Curr Opin Genet Dev. 17:145–150. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
85
|
Croce CM: Causes and consequences of
microRNA dysregulation in cancer. Nat Rev Genet. 10:704–714. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
86
|
Tili E, Michaillle JJ and Croce CM:
MicroRNAs play a central role in molecular dysfunctions linking
inflammation with cancer. Immunol Rev. 253:167–184. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
87
|
Trotta R, Chen L, Ciarlariello D, et al:
miR-155 regulates IFN-γ production in natural killer cells. Blood.
119:3478–3485. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
88
|
Schetter AJ, Okayama H and Harris CC: The
role of microRNAs in colorectal cancer. Cancer J. 18:244–252. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
89
|
de Visser KE, Korets LV and Coussens LM:
De novo carcinogenesis promoted by chronic inflammation is B
lymphocyte dependent. Cancer Cell. 7:411–423. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
90
|
Viau M and Zouali M: B-lymphocytes, innate
immunity, and autoimmunity. Clin Immunol. 114:17–26. 2005.
View Article : Google Scholar
|
|
91
|
Balkwill F, Charies KA and Mantovani A:
Smoldering and polarized inflammation in the initiation and
promotion of malignant disease. Cancer Cell. 7:211–217. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
92
|
Hawes SE and Kiviat NB: Are genital
infections and inflammation cofactors in the pathogenesis of
invasive cervical cancer? J Natl Cancer Inst. 94:1592–1593. 2002.
View Article : Google Scholar : PubMed/NCBI
|
|
93
|
Schwebke JR and Zajackowski ME: Effect of
concurrent lower genital tract infections on cervical cancer
screening. Genitourin Med. 73:383–386. 1997.
|
|
94
|
Castle PE, Hillier SL, Rabe LK, et al: An
association of cervical inflammation with high-grade cervical
neoplasia in women infected with oncogenic human papillomavirus
(HPV). Cancer Epidemiol Biomarkers Prev. 10:1021–1027.
2001.PubMed/NCBI
|
|
95
|
Yang YC, Chang CL, Huang YW and Wang DY:
Possible cofactor in cervical carcinogenesis: proliferation index
of the transformation zone in cervicitis. Chang Gung Med J.
24:615–620. 2001.
|
|
96
|
Hammes LS, Tekmal RR, Naud P, et al:
Macrophages, inflammation and risk of cervical intraepithelial
neoplasia (CIN) progression - clinicopathological correlation.
Gynecol Oncol. 105:157–165. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
97
|
Kovacic MB, Katki HA, Kreimer AR and
Sherman ME: Epidemiologic analysis of histologic cervical
inflammation: relationship to human papillomavirus infections.
Human Pathol. 39:1088–1095. 2008. View Article : Google Scholar
|
|
98
|
Sharkey DJ, Tremellen KP, Jasper MJ, et
al: Seminal fluid Induces leukocyte recruitment and cytokine and
chemokine mRNA expression in the human cervix after coitus. J
Immunol. 188:2445–2454. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
99
|
Manhart LS and Koutsky LZ: Do condoms
prevent genital HPV infection, external genital warts, or cervical
neoplasia? A meta-analysis. Sex Transm Dis. 29:725–735. 2002.
View Article : Google Scholar : PubMed/NCBI
|
|
100
|
Sales KJ and Jabbour HN: Cyclooxygenase
enzymes and prostaglandins in reproductive tract physiology and
pathology. Prostaglandins Other Lipid Mediat. 71:97–117. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
101
|
Silva J: Cerqueira F and Medeiros R:
Chlamydia trachomatis infection: implications for HPV status and
cervical cancer. Arch Gynecol Obstet. 289:715–23. 2014. View Article : Google Scholar
|
|
102
|
Niebler M, Quian X, Höfler D, et al:
Post-translational control of IL-1β via the human papillomavirus
type 16 E6 oncoprotein: a novel mechanism of innate immune escape
mediated by the E3-ubiquitin ligase E6-AP and p53. PLOS Pathog.
9:e10035362013. View Article : Google Scholar
|
|
103
|
Kemp TJ, Hildesheim A, Garcia-Piñeres A,
et al: Elevated systemic levels of inflammatory cytokines in older
women with persistent cervical HPV infection. Cancer Epidemiol
Biomarkers Prev. 19:1954–1959. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
104
|
Kaul R and Hirbod T: Editorial: Genital
epithelial cells: foot soldiers or fashion leaders? J Leuk Biol.
88:427–429. 2010. View Article : Google Scholar
|
|
105
|
Stanley M: Pathology and epidemiology of
HPV infection in females. Gynecol Oncol. 117(Suppl 2): S5–S10.
2010. View Article : Google Scholar
|
|
106
|
Karim R, Tummers B, Meyers C, et al: Human
papillomavirus (HPV) upregulates t6he cellular deubiquitinase UCHL1
to suppress the keratinocyte’s innate immune response. PLOS Pathog.
9:e10033842013. View Article : Google Scholar
|
|
107
|
Senba M and Mori N: Mechanisms of virus
immune evasion lead to development from chronic inflammation to
cancer formation associated with human papillomavirus infection.
Oncol Rev. 6:135–144. 2012. View Article : Google Scholar
|
|
108
|
Senba M, Buziba N, Mori N, et al: Human
papillomavirus infection induces NF-κB activation in cervical
cancer: a comparison with penile cancer. Oncol Lett. 2:65–68.
2011.PubMed/NCBI
|
|
109
|
James MA, Lee JH and Klingelhutz AJ: Human
papillomavirus type 16 E6 activates NF-kappaB, induces cIAP-2
expression, and protects against apoptosis in a PDZ binding
motif-dependent manner. J Virol. 80:5301–5307. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
110
|
Scott ME, Shvetsov YB, Thompson PJ, et al:
Cervical cytokines and clearance of incident human papillomavirus
infection: Hawaii HPV cohort study. Int J Cancer. 133:1187–1196.
2013. View Article : Google Scholar : PubMed/NCBI
|
|
111
|
Broccardo E, Lepique AP and Villa LL: The
role of inflammation in HPV carcinogenesis. Carcinogenesis.
31:1905–1912. 2010. View Article : Google Scholar
|
|
112
|
Balkwill F: Tumor necrosis factor or tumor
promoting factor? Cytokine Growth Factor Rev. 13:135–41. 2002.
View Article : Google Scholar : PubMed/NCBI
|
|
113
|
Stanley MA, Pett MR and Coleman N: HPV:
from infection to cancer. Bioch Soc Trans. 35:1456–1460. 2007.
View Article : Google Scholar
|
|
114
|
Tindle RW: Immune evasion in human
papillomavirus-associated cervical cancer. Nature Rev Cancer.
2:59–65. 2002. View
Article : Google Scholar
|
|
115
|
zur Hausen H: Papillomaviruses in the
causation of human cancers - a brief historical account. Virology.
384:260–265. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
116
|
Oh JM, Kim SH, Lee YI, et al: Human
papillomavirus E5 protein induces expression of the EP4 subtype of
prostaglandin E2 receptor in cyclic AMP response element-dependent
pathways in cervical cancer cells. Carcinogenesis. 30:144–149.
2009.
|
|
117
|
Bodily J and Laimins LA: Persistence of
human papillomavirus infection: keys to malignant progression.
Trends Microbiol. 19:33–39. 2011. View Article : Google Scholar :
|
|
118
|
Trinchieri G: Innate inflammation and
cancer: is it time for cancer prevention? F1000 Med Rep. 3:112011.
View Article : Google Scholar : PubMed/NCBI
|
|
119
|
Sales KJ, Katz AA, Howard B, et al:
Cyclooxygenase-1 is up-regulated in cervical carcinomas:
autocrine/paracrine regulation of cyclooxygenase-2, prostaglandin e
receptors, and angiogenic factors by cyclooxygenase-1. Cancer Res.
62:424–432. 2002.PubMed/NCBI
|
|
120
|
Santin AD, Zhan F, Bignotti E, et al: Gene
expression profiles of primary HPV16- and HPV18-infected early
stage cervical cancers and normal cervical epithelium:
identification of novel candidate molecular markers for cervical
cancer diagnosis and therapy. Virol. 331:269–291. 2005. View Article : Google Scholar
|