|
1
|
Ohgaki H and Kleihues P: Epidemiology and
etiology of gliomas. Acta Neuropathol. 109:93–108. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Lu KP, Hanes SD and Hunter T: A human
peptidyl-prolyl isomerase essential for regulation of mitosis.
Nature. 380:544–547. 1996. View
Article : Google Scholar : PubMed/NCBI
|
|
3
|
Bao L, Kimzey A, Sauter G, Sowadski JM, Lu
KP and Wang DG: Prevalent overexpression of prolyl isomerase Pin1
in human cancers. Am J Pathol. 164:1727–1737. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Göthel SF and Marahiel MA: Peptidyl-prolyl
cis-trans isomerases, a superfamily of ubiquitous folding
catalysts. Cell Mol Life Sci. 55:423–436. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Hunter T: Prolyl isomerases and nuclear
function. Cell. 92:141–143. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Kim MR, Choi HS, Heo TH, Hwang SW and Kang
KW: Induction of vascular endothelial growth factor by
peptidyl-prolyl isomerase Pin1 in breast cancer cells. Biochem
Biophys Res Commun. 369:547–553. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Ryo A, Uemura H, Ishiguro H, Saitoh T,
Yamaguchi A, Perrem K, Kubota Y, Lu KP and Aoki I: Stable
suppression of tumorigenicity by Pin1-targeted RNA interference in
prostate cancer. Clin Cancer Res. 11:7523–7531. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Hennig L, Christner C, Kipping M,
Schelbert B, Rücknagel KP, Grabley S, Küllertz G and Fischer G:
Selective inactivation of parvulin-like peptidyl-prolyl cis/trans
isomerases by juglone. Biochemistry. 37:5953–5960. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Kesavapany S, Patel V, Zheng YL, Pareek
TK, Bjelogrlic M, Albers W, Amin N, Jaffe H, Gutkind JS, Strong MJ,
Grant P and Pant HC: Inhibition of Pin1 reduces glutamate-induced
perikaryal accumulation of phosphorylated neurofilament-H in
neurons. Mol Biol Cell. 18:3645–3655. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Mathur R, Chandna S, Kapoor NP and
Dwarakanath SB: Peptidyl prolyl isomerase, Pin1 is a potential
target for enhancing the therapeutic efficacy of etoposide. Curr
Cancer Drug Targets. 11:380–392. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Mironova EV, Evstratova AA and Antonov SM:
A fluorescence vital assay for the recognition and quantification
of excitotoxic cell death by necrosis and apoptosis using confocal
microscopy on neurons in culture. J Neurosci Methods. 163:1–8.
2007. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Ongusaha PP, Kwak JC, Zwible AJ, Macip S,
Higashiyama S, Taniguchi N, Fang L and Lee SW: HB-EGF is a potent
inducer of tumor growth and angiogenesis. Cancer Res. 64:5283–5290.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Qin L, Li M, Ren W, Zhang D, Zhang J,
Zhang Y and Cheng N: Silencing Pin1 suppresses the expression and
bioactivity of MMP-9 through NF-κB in colorectal carcinoma SW480
cells. Clin Oncol Cancer Res. 7:12–17. 2010. View Article : Google Scholar
|
|
14
|
Lu KP: Prolyl isomerase Pin1 as a
molecular target for cancer diagnostics and therapeutics. Cancer
Cell. 4:175–180. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Atkinson GP, Nozell SE, Harrison DK,
Stonecypher MS, Chen D and Benveniste EN: The prolyl isomerase Pin1
regulates the NF-kappaB signaling pathway and interleukin-8
expression in glioblastoma. Oncogene. 28:3735–3745. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Ayala G, Wang D, Wulf G, Frolov A, Li R,
Sowadski J, Wheeler TM, Lu KP and Bao L: The prolyl isomerase Pin1
is a novel prognostic marker in human prostate cancer. Cancer Res.
63:6244–6251. 2003.PubMed/NCBI
|
|
17
|
Ryo A, Nakamura M, Wulf G, Liou YC and Lu
KP: Pin1 regulates turnover and subcellular localization of
beta-catenin by inhibiting its interaction with APC. Nat Cell Biol.
3:793–801. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Wulf GM, Ryo A, Wulf GG, Lee SW, Niu T,
Petkova V and Lu KP: Pin1 is overexpressed in breast cancer and
cooperates with Ras signaling in increasing the transcriptional
activity of c-Jun towards cyclin D1. EMBO J. 20:3459–3472. 2001.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Liou YC, Ryo A, Huang HK, Lu PJ, Bronson
R, Fujimori F, Uchida T, Hunter T and Lu KP: Loss of Pin1 function
in the mouse causes phenotypes resembling cyclin D1-null
phenotypes. Proc Natl Acad Sci USA. 99:1335–1340. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Ryo A, Liou YC, Lu KP and Wulf G: Prolyl
isomerase Pin1: a catalyst for oncogenesis and a potential
therapeutic target in cancer. J Cell Sci. 116:773–783. 2003.
View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Daum S, Erdmann F, Fischer G, Féaux de
Lacroix B, Hessamian-Alinejad A, Houben S, Frank W and Braun M:
Aryl indanyl ketones: efficient inhibitors of the human peptidyl
prolyl cis/trans isomerase Pin1. Angew Chem Int Ed Engl.
45:7454–7458. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Zhang Y, Daum S, Wildemann D, Zhou XZ,
Verdecia MA, Bowman ME, Lücke C, Hunter T, Lu KP, Fischer G and
Noel JP: Structural basis for high-affinity peptide inhibition of
human Pin1. ACS Chem Biol. 2:320–328. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Mori T, Hidaka M, Lin YC, Yoshizawa I,
Okabe T, Egashira S, Kojima H, Nagano T, Koketsu M, Takamiya M and
Uchida T: A dual inhibitor against prolyl isomerase Pin1 and
cyclophilin discovered by a novel real-time fluorescence detection
method. Biochem Biophys Res Commun. 406:439–443. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Lee TH, Pastorino L and Lu KP:
Peptidyl-prolyl cis-trans isomerase Pin1 in ageing, cancer and
Alzheimer disease. Expert Rev Mol Med. 13:e212011. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Atabay KD and Karabay A: Pin1 inhibition
activates cyclin D and produces neurodegenerative pathology. J
Neurochem. 120:430–439. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Sun J, Blaskovich MA, Jain RK, et al:
Blocking angiogenesis and tumorigenesis with GFA-116, a synthetic
molecule that inhibits binding of vascular endothelial growth
factor to its receptor. Cancer Res. 64:3586–3592. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Xie K, Wei D, Shi Q and Huang S:
Constitutive and inducible expression and regulation of vascular
endothelial growth factor. Cytokine Growth Factor Rev. 15:297–324.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Flügel D, Görlach A, Michiels C and
Kietzmann T: Glycogen synthase kinase 3 phosphorylates
hypoxia-inducible factor 1alpha and mediates its destabilization in
a VHL-independent manner. Mol Cell Biol. 27:3253–3265. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Chabannes E, Fauconnet S, Bernardini S,
Wallerand H, Adessi G and Bittard H: Protein kinase C signalling
pathway is involved in the regulation of vascular endothelial
growth factor expression in human bladder transitional carcinoma
cells. Cell Signal. 13:585–591. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Choe G, Park JK, JoubenSteele L, Kremen
TJ, Liau LM, Vinters HV, Cloughesy TF and Mischel PS: Active matrix
metalloproteinase 9 expression is associated with primary
glioblastoma subtype. Clin Cancer Res. 8:2894–2901. 2002.PubMed/NCBI
|
|
31
|
Zhong S, Salomoni P, Ronchetti S, Guo A,
Ruggero D and Pandolfi PP: Promyelocytic leukemia protein (PML) and
Daxx participate in a novel nuclear pathway for apoptosis. J Exp
Med. 191:631–640. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Perlman R, Schiemann W, Brooks MW, Lodish
HF and Weinberg RA: TGF-beta-induced apoptosis is mediated by the
adapter protein Daxx that facilitates JNK activation. Nat Cell
Biol. 3:708–714. 2001. View
Article : Google Scholar : PubMed/NCBI
|
|
33
|
Salomoni P and Khelifi A: Daxx: death or
survival protein? Trends Cell Biol. 16:97–104. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Ryo A, Hirai A, Nishi M, Liou YC, Perrem
K, Lin SC, Hirano H, Lee SW and Aoki I: A suppressive role of the
prolyl isomerase Pin1 in cellular apoptosis mediated by the
death-associated protein Daxx. J Biol Chem. 282:36671–36681. 2007.
View Article : Google Scholar : PubMed/NCBI
|
