|
1
|
Pui CH, Gajjar AJ, Kane JR, Qaddoumi IA
and Pappo AS: Challenging issues in pediatric oncology. Nat Rev
Clin Oncol. 8:540–549. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Mueller S and Chang S: Pediatric brain
tumors: Current treatment strategies and future therapeutic
approaches. Neurotherapeutics. 6:570–586. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Packer RJ and Vezina G: Management of and
prognosis with medulloblastoma: Therapy at a crossroads. Arch
Neurol. 65:1419–1424. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Northcott PA, Jones DT, Kool M, Robinson
GW, Gilbertson RJ, Cho YJ, Pomeroy SL, Korshunov A, Lichter P,
Taylor MD and Pfister SM: Medulloblastomics: The end of the
beginning. Nat Rev Cancer. 12:818–834. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Spiegler BJ, Bouffet E, Greenberg ML,
Rutka JT and Mabbott DJ: Change in neurocognitive functioning after
treatment with cranial radiation in childhood. J Clin Oncol.
22:706–713. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Taylor MD, Northcott PA, Korshunov A,
Remke M, Cho YJ, Clifford SC, Eberhart CG, Parsons DW, Rutkowski S,
Gajjar A, et al: Molecular subgroups of medulloblastoma: The
current consensus. Acta Neuropathol. 123:465–472. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Samkari A, White JC and Packer RJ:
Medulloblastoma: Toward biologically based management. Semin
Pediatr Neurol. 22:6–13. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Zhuang M, Calabrese MF, Liu J, Waddell MB,
Nourse A, Hammel M, Miller DJ, Walden H, Duda DM, Seyedin SN, et
al: Structures of SPOP-substrate complexes: Insights into molecular
architectures of BTB-Cul3 ubiquitin ligases. Mol Cell. 36:39–50.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Berger MF, Lawrence MS, Demichelis F,
Drier Y, Cibulskis K, Sivachenko AY, Sboner A, Esgueva R, Pflueger
D, Sougnez C, et al: The genomic complexity of primary human
prostate cancer. Nature. 470:214–220. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Kan Z, Jaiswal BS, Stinson J, Janakiraman
V, Bhatt D, Stern HM, Yue P, Haverty PM, Bourgon R, Zheng J, et al:
Diverse somatic mutation patterns and pathway alterations in human
cancers. Nature. 466:869–873. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Hernández-Muñoz I, Lund AH, van der Stoop
P, Boutsma E, Muijrers I, Verhoeven E, Nusinow DA, Panning B,
Marahrens Y and van Lohuizen M: Stable X chromosome inactivation
involves the PRC1 Polycomb complex and requires histone MACROH2A1
and the CULLIN3/SPOP ubiquitin E3 ligase. Proc Natl Acad Sci USA.
102:pp. 7635–7640. 2005; View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Liu A, Desai BM and Stoffers DA:
Identification of PCIF1, a POZ domain protein that inhibits PDX-1
(MODY4) transcriptional activity. Mol Cell Biol. 24:4372–4383.
2004. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Kwon JE, La M, Oh KH, Oh YM, Kim GR, Seol
JH, Baek SH, Chiba T, Tanaka K, Bang OS, et al: BTB
domain-containing speckle-type POZ protein (SPOP) serves as an
adaptor of Daxx for ubiquitination by Cul3-based ubiquitin ligase.
J Biol Chem. 281:12664–12672. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Wang C, Pan Y and Wang B: Suppressor of
fused and Spop regulate the stability, processing and function of
Gli2 and Gli3 full-length activators but not their repressors.
Development. 137:2001–2009. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Choo KB, Chuang TJ, Lin WY, Chang CM, Tsai
YH and Huang CJ: Evolutionary expansion of SPOP and associated
TD/POZ gene family: Impact of evolutionary route on gene expression
pattern. Gene. 460:39–47. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Errington WJ, Khan MQ, Bueler SA,
Rubinstein JL, Chakrabartty A and Privé GG: Adaptor protein
self-assembly drives the control of a cullin-RING ubiquitin ligase.
Structure. 20:1141–1153. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Mulatinho MV, de Carvalho Serao CL, Scalco
F, Hardekopf D, Pekova S, Mrasek K, Liehr T, Weise A, Rao N and
Llerena JC Jr: Severe intellectual disability, omphalocele,
hypospadia and high blood pressure associated to a deletion at
2q22.1q22.3: Case report. Mol Cytogenet. 5:302012. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Ellison DW, Clifford SC, Gajjar A and
Gilbertson RJ: What's new in neuro-oncology? Recent advances in
medulloblastoma. Eur J Paediatr Neurol. 7:53–66. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Louis DN, Ohgaki H, Wiestler OD, Cavenee
WK, Burger PC, Jouvet A, Scheithauer BW and Kleihues P: The 2007
WHO classification of tumours of the central nervous system. Acta
Neuropathol. 114:97–109. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
Song LB, Liao WT, Mai HQ, Zhang HZ, Zhang
L, Li MZ, Hou JH, Fu LW, Huang WL, Zeng YX and Zeng MS: The
clinical significance of twist expression in nasopharyngeal
carcinoma. Cancer Lett. 242:258–265. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Fukuoka J, Fujii T, Shih JH, Dracheva T,
Meerzaman D, Player A, Hong K, Settnek S, Gupta A, Buetow K, et al:
Chromatin remodeling factors and BRM/BRG1 expression as prognostic
indicators in non-small cell lung cancer. Clin Cancer Res.
10:4314–4324. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression using real time quantitative PCR and the
2(−Delta Delta C(T)) method. Methods. 25:402–408. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Zhang D, Wang H, Sun M, Yang J, Zhang W,
Han S and Xu B: Speckle-type POZ protein, SPOP, is involved in the
DNA damage response. Carcinogenesis. 35:1691–1697. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Mani RS: The emerging role of speckle-type
POZ protein (SPOP) in cancer development. Drug Discov Today.
19:1498–1502. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Kim MS, Je EM, Oh JE, Yoo NJ and Lee SH:
Mutational and expressional analyses of SPOP, a candidate tumor
suppressor gene, in prostate, gastric and colorectal cancers.
APMIS. 121:626–633. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Kim MS, Kim MS, Yoo NJ and Lee SH: Somatic
mutation of SPOP tumor suppressor gene is rare in breast, lung,
liver cancers, and acute leukemias. APMIS. 122:164–166. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Ding D, Song T, Jun W, Tan Z and Fang J:
Decreased expression of the SPOP gene is associated with poor
prognosis in glioma. Int J Oncol. 46:333–341. 2015.PubMed/NCBI
|
