|
1
|
Franceschi C and Campisi J: Chronic
inflammation (inflammaging) and its potential contribution to
age-associated diseases. J Gerontol A Biol Sci Med Sci. 69 Suppl
1:S4–S9. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Izano M, Wei EK, Tai C, Swede H, Gregorich
S, Harris TB, Klepin H, Satterfield S, Murphy R, Newman AB, et al:
Chronic inflammation and risk of colorectal and other
obesity-related cancers: The health, aging and body composition
study. Int J Cancer. 138:1118–1128. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Diller ML, Kudchadkar RR, Delman KA,
Lawson DH and Ford ML: Balancing inflammation: The link between
Th17 and rtegulatory T cells. Mediators Inflamm. 2016:63092192016.
View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Eyerich S, Eyerich K, Pennino D, Carbone
T, Nasorri F, Pallotta S, Cianfarani F, Odorisio T, Traidl-Hoffmann
C, Behrendt H, et al: Th22 cells represent a distinct human T cell
subset involved in epidermal immunity and remodeling. J Clin
Invest. 119:3573–3585. 2009.PubMed/NCBI
|
|
5
|
Trifari S, Kaplan CD, Tran EH, Crellin NK
and Spits H: Identification of a human helper T cell population
that has abundant production of interleukin 22 and is distinct from
T(H)-17, T(H)1 and T(H)2 cells. Nat Immunol. 10:864–871. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Wolk K, Witte E, Witte K, Warszawska K and
Sabat R: Biology of interleukin-22. Semin Immunopathol. 32:17–31.
2010. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Lim C and Savan R: The role of the
IL-22/IL-22R1 axis in cancer. Cytokine Growth Factor Rev.
25:257–271. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Lanfranca Perusina M, Lin Y, Fang J, Zou W
and Frankel T: Biological and pathological activities of
interleukin-22. J Mol Med (Berl). 94:523–534. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Zhang L, Wang T, Wang XQ, Du RZ, Zhang KN,
Liu XG, Ma DX, Yu S, Su GH, Li ZH, et al: Elevated frequencies of
circulating Th22 cell in addition to Th17 cell and Th17/Th1 cell in
patients with acute coronary syndrome. PLoS One. 8:e714662013.
View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Anuradha R, George PJ, Chandrasekaran V,
Kumaran PP, Nutman TB and Babu S: Interleukin 1 (IL-1)- and
IL-23-mediated expansion of filarial antigen-specific Th17 and Th22
cells in filarial lymphedema. Clin Vaccine Immunol. 21:960–965.
2014. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Miossec P, Korn T and Kuchroo VK:
Interleukin-17 and type 17 helper T cells. N Engl J Med.
361:888–898. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Li X, Wang Y, Han C, Li P and Zhang H:
Colorectal cancer progression is associated with accumulation of
Th17 lymphocytes in tumor tissues and increased serum levels of
interleukin-6. Tohoku J Exp Med. 233:175–182. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Wu IC, Lin CC and Hsiung CA: Emerging
roles of frailty and inflammaging in risk assessment of age-related
chronic diseases in older adults: The intersection between aging
biology and personalized medicine. Biomedicine (Taipei). 5:12015.
View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Duhen T, Geiger R, Jarrossay D,
Lanzavecchia A and Sallusto F: Production of interleukin 22 but not
interleukin 17 by a subset of human skin-homing memory T cells. Nat
Immunol. 10:857–863. 2009. View
Article : Google Scholar : PubMed/NCBI
|
|
15
|
Parker KH, Beury DW and Ostrand-Rosenberg
S: Myeloid-derived suppressor cells: Critical cells driving immune
suppression in the tumor microenvironment. Adv Cancer Res.
128:95–139. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Bueno V, Sant'Anna OA and Lord JM: Ageing
and myeloid-derived suppressor cells: possible involvement in
immunosenescence and age-related disease. Age (Dordr). 36:97292014.
View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Solito S, Falisi E, Diaz-Montero CM, Doni
A, Pinton L, Rosato A, Francescato S, Basso G, Zanovello P,
Onicescu G, et al: A human promyelocytic-like population is
responsible for the immune suppression mediated by myeloid-derived
suppressor cells. Blood. 118:2254–2265. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
Lechner MG, Liebertz DJ and Epstein AL:
Characterization of cytokine-induced myeloid-derived suppressor
cells from normal human peripheral blood mononuclear cells. J
Immunol. 185:2273–2284. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Chatterjee S, Das S, Chakraborty P, Manna
A, Chatterjee M and Choudhuri SK: Myeloid derived suppressor cells
(MDSCs) can induce the generation of Th17 response from naive CD4+
T cells. Immunobiology. 218:718–724. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
20
|
McGuire S: World cancer report 2014.
Geneva, Switzerland: World health organization, international
agency for research on cancer, WHO Press, 2015. Adv Nutr.
7:418–419. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Sobin LH, Gospodarowicz MK and Wittekind
C: International union against cancer (UICC) TNM classification of
malignant tumours. 7th edition. New York: Wiley-Liss; 2010
|
|
22
|
Zhang L, Li JM, Liu XG, Ma DX, Hu NW, Li
YG, Li W, Hu Y, Yu S, Qu X, et al: Elevated Th22 cells correlated
with Th17 cells in patients with rheumatoid arthritis. J Clin
Immunol. 31:606–614. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Cetin Aktas E, Cosan F, Cefle A and Deniz
G: IL-22-secreting Th22 and IFN-γ-secreting Th17 cells in Behcet's
disease. Mod Rheumatol. 24:802–807. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Truchetet ME, Brembilla NC, Montanari E,
Allanore Y and Chizzolini C: Increased frequency of circulating
Th22 in addition to Th17 and Th2 lymphocytes in systemic sclerosis:
Association with interstitial lung disease. Arthritis Res Ther.
13:R1662011. View
Article : Google Scholar : PubMed/NCBI
|
|
25
|
Huang YH, Cao YF, Jiang ZY, Zhang S and
Gao F: Th22 cell accumulation is associated with colorectal cancer
development. World J Gastroenterol. 21:4216–4224. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Kuang DM, Xiao X, Zhao Q, Chen MM, Li XF,
Liu RX, Wei Y, Ouyang FZ, Chen DP, Wu Y, et al: B7-H1-expressing
antigen-presenting cells mediate polarization of protumorigenic
Th22 subsets. J Clin Invest. 124:4657–4667. 2014. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Tian T, Sun Y, Li M, He N, Yuan C, Yu S,
Wang M, Ji C and Ma D: Increased Th22 cells as well as Th17 cells
in patients with adult T-cell acute lymphoblastic leukemia. Clin
Chim Acta. 426:108–113. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Xu X, Tang Y, Guo S, Zhang Y, Tian Y, Ni B
and Wang H: Increased intratumoral interleukin 22 levels and
frequencies of interleukin 22-producing CD4+ T cells correlate with
pancreatic cancer progression. Pancreas. 43:470–477. 2014.
View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Zhuang Y, Peng LS, Zhao YL, Shi Y, Mao XH,
Guo G, Chen W, Liu XF, Zhang JY, Liu T, et al: Increased
intratumoral IL-22-producing CD4(+) T cells and Th22 cells
correlate with gastric cancer progression and predict poor patient
survival. Cancer Immunol Immunother. 61:1965–1975. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Axelrad JE, Lichtiger S and Yajnik V:
Inflammatory bowel disease and cancer: The role of inflammation,
immunosuppression, and cancer treatment. World J Gastroenterol.
22:4794–4801. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Sommer A and Fabri M: Vitamin D regulates
cytokine patterns secreted by dendritic cells to promote
differentiation of IL-22-producing T cells. PLoS One.
10:e01303952015. View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Gorelick PB: Role of inflammation in
cognitive impairment: Results of observational epidemiological
studies and clinical trials. Ann N Y Acad Sci. 1207:155–162. 2010.
View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Satizabal CL, Zhu YC, Mazoyer B, Dufouil C
and Tzourio C: Circulating IL-6 and CRP are associated with MRI
findings in the elderly: The 3C-Dijon study. Neurology. 78:720–727.
2012. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Zhao G, Zhu G, Huang Y, Zheng W, Hua J,
Yang S, Zhuang J and Ye J: IL-6 mediates the signal pathway of
JAK-STAT3-VEGF-C promoting growth, invasion and lymphangiogenesis
in gastric cancer. Oncol Rep. 35:1787–1795. 2016. View Article : Google Scholar : PubMed/NCBI
|
|
35
|
Guo L, Ou JL, Zhang T, Ma L and Qu LF:
Effect of expressions of tumor necrosis factor alpha and
interleukin 1B on peritoneal metastasis of gastric cancer. Tumour
Biol. 36:8853–8860. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Highfill SL, Rodriguez PC, Zhou Q, Goetz
CA, Koehn BH, Veenstra R, Taylor PA, Panoskaltsis-Mortari A, Serody
JS, Munn DH, et al: Bone marrow myeloid-derived suppressor cells
(MDSCs) inhibit graft-vs.-host disease (GVHD) via an
arginase-1-dependent mechanism that is up-regulated by
interleukin-13. Blood. 116:5738–5747. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
37
|
Greten TF, Manns MP and Korangy F: Myeloid
derived suppressor cells in human diseases. Int Immunopharmacol.
11:802–807. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
38
|
Gerber HP, Olazoglu E and Grewal IS:
Targeting inflammatory cells to improve anti-VEGF therapies in
oncology. Recent Results Cancer Res. 180:185–200. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
39
|
Melani C, Sangaletti S, Barazzetta FM,
Werb Z and Colombo MP: Amino-biphosphonate-mediated MMP-9
inhibition breaks the tumor-bone marrow axis responsible for
myeloid-derived suppressor cell expansion and macrophage
infiltration in tumor stroma. Cancer Res. 67:11438–11446. 2007.
View Article : Google Scholar : PubMed/NCBI
|
|
40
|
Zhuang Y, Cheng P, Liu XF, Peng LS, Li BS,
Wang TT, Chen N, Li WH, Shi Y, Chen W, et al: A pro-inflammatory
role for Th22 cells in Helicobacter pylori-associated
gastritis. Gut. 64:1368–1378. 2015. View Article : Google Scholar : PubMed/NCBI
|
|
41
|
Zhang F, Shang D, Zhang Y and Tian Y:
Interleukin-22 suppresses the growth of A498 renal cell carcinoma
cells via regulation of STAT1 pathway. PLoS One. 6:e203822011.
View Article : Google Scholar : PubMed/NCBI
|
|
42
|
Dixon BR, Radin JN, Piazuelo MB, Contreras
DC and Algood HM: IL-17a and IL-22 induce expression of
antimicrobials in gastrointestinal epithelial cells and may
contribute to epithelial cell defense against Helicobacter
pylori. PLoS One. 11:e01485142016. View Article : Google Scholar : PubMed/NCBI
|
