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Oncology Letters
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Print ISSN: 1792-1074 Online ISSN: 1792-1082
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March-2019 Volume 17 Issue 3

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Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

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Correction Open Access

[Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells

  • Authors:
    • Mi Suk Choi
    • Sung‑Min Moon
    • Seul Ah Lee
    • Bo‑Ram Park
    • Jae‑Sung Kim
    • Do Kyung Kim
    • Yong Hwan Kim
    • Chun Sung Kim
  • View Affiliations / Copyright

    Affiliations: Department of Dental Hygiene, Chodang University, Muan‑ro, Muan‑eup, Muan 534‑701, Republic of Korea, CStech Research Institute, Gwangju, South Jeolla 61007, Republic of Korea, Department of Oral Biochemistry, College of Dentistry, Chosun University, Gwangju, South Jeolla 501‑759, Republic of Korea, Pre‑Dentistry, College of Dentistry, Chosun University, Gwangju, South Jeolla 501‑759, Republic of Korea, Department of Oral Physiology, College of Dentistry, Chosun University, Gwangju, South Jeolla 501‑759, Republic of Korea, Department of Crop Science and Biotechnology, College of Life and Resource Science, Cheonan, Chungnam 31116, Republic of Korea
    Copyright: © Choi et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Pages: 3615-3615
    |
    Published online on: February 4, 2019
       https://doi.org/10.3892/ol.2019.10014
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Article

Oncol Lett 15: [Related article:] 6489-6496 2018; DOI: 10.3892/ol.2018.8089

An interested reader drew to the authors' attention that, in the published version of the above article, the data shown in Fig. 4A for p-Akt and total phosphoinositide 3-kinase (PI3K) were strikingly similar. After having re-examined their source data, the authors were able to confirm that the data correctly shown for total PI3K had also inadvertently been included in the Figure as the data for p-Akt.

Figure 4.

Adenosine treatment of FaDu cells suppresses the PI3K/Akt/mTOR signaling pathway. FaDu cells were treated with 3 mM adenosine for 24 h prior to collection of whole-cell lysates. Samples were separated using 8–15% SDS-PAGE, and resolved by incubation with primary antibodies against (A) phospho-PI3K, total PI3K, phospho-Akt, total Akt, phospho-mTOR, total mTOR, and (B) phospho-S6K1, phospho-4EBP1, and phospho-EIF4 G. β-actin was used as an internal control for the western blot analysis. Data are representative of three experiments that produced similar results. Sample bands (n=3) were densitometrically evaluated. *P<0.05 and **P<0.01. PI3K, phosphoinositide 3-kinase; Akt, RAC serine/threonine-protein kinase; mTOR, mechanistic target of rapamycin; 4EBP1, eukaryotic translation initiation factor 4E-binding protein 1; EIF4 G, eukaryotic translation initiation factor 4 γ1.

A corrected version of Fig. 4, including the correct data for p-Akt for Fig. 4A, is shown opposite. Note that this change does not affect the results or the conclusions reported in this paper, and all the authors agree to this correction. The authors thank the reader for drawing this error to their attention, and apologize to the Editor and to the readership of the Journal for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Choi MS, Moon SM, Lee SA, Park BR, Kim JS, Kim DK, Kim YH and Kim CS: [Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells. Oncol Lett 17: 3615-3615, 2019.
APA
Choi, M.S., Moon, S., Lee, S.A., Park, B., Kim, J., Kim, D.K. ... Kim, C.S. (2019). [Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells. Oncology Letters, 17, 3615-3615. https://doi.org/10.3892/ol.2019.10014
MLA
Choi, M. S., Moon, S., Lee, S. A., Park, B., Kim, J., Kim, D. K., Kim, Y. H., Kim, C. S."[Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells". Oncology Letters 17.3 (2019): 3615-3615.
Chicago
Choi, M. S., Moon, S., Lee, S. A., Park, B., Kim, J., Kim, D. K., Kim, Y. H., Kim, C. S."[Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells". Oncology Letters 17, no. 3 (2019): 3615-3615. https://doi.org/10.3892/ol.2019.10014
Copy and paste a formatted citation
x
Spandidos Publications style
Choi MS, Moon SM, Lee SA, Park BR, Kim JS, Kim DK, Kim YH and Kim CS: [Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells. Oncol Lett 17: 3615-3615, 2019.
APA
Choi, M.S., Moon, S., Lee, S.A., Park, B., Kim, J., Kim, D.K. ... Kim, C.S. (2019). [Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells. Oncology Letters, 17, 3615-3615. https://doi.org/10.3892/ol.2019.10014
MLA
Choi, M. S., Moon, S., Lee, S. A., Park, B., Kim, J., Kim, D. K., Kim, Y. H., Kim, C. S."[Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells". Oncology Letters 17.3 (2019): 3615-3615.
Chicago
Choi, M. S., Moon, S., Lee, S. A., Park, B., Kim, J., Kim, D. K., Kim, Y. H., Kim, C. S."[Corrigendum] Adenosine induces intrinsic apoptosis via the PI3K/Akt/mTOR signaling pathway in human pharyngeal squamous carcinoma FaDu cells". Oncology Letters 17, no. 3 (2019): 3615-3615. https://doi.org/10.3892/ol.2019.10014
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