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Oncology Letters
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Print ISSN: 1792-1074 Online ISSN: 1792-1082
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April-2026 Volume 31 Issue 4

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

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Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

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Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

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International Journal of Epigenetics

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Correction Open Access

[Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma

  • Authors:
    • Yang Yang
    • Jia-Xin Luo
    • Yuan-Yang Li
    • Ke-Hong Guo
    • Lin Ye
    • Dan Zhao
  • View Affiliations / Copyright

    Affiliations: Department of Oral and Maxillofacial Surgery, The Affiliated Stomatological Hospital, Southwest Medical University, Luzhou, Sichuan 646000, P.R. China
    Copyright: © Yang et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 136
    |
    Published online on: February 12, 2026
       https://doi.org/10.3892/ol.2026.15489
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Oncol Lett 30: [Related article:] 597, 2025; DOI: 10.3892/ol.2025.15343

Subsequently to the publication of the above paper, an interested reader drew to the Editor's attention that a pair of data panels shown for the scratch-wound assay experiments in Figs. 1 and 7 were apparently overlapping; in addition, another pair of data panels within Fig. 7 itself were also found to be overlapping.

S100A4 OE rescues the inhibitory
effects of ILK knockdown on the migration and invasion of adenoid
cystic carcinoma. (A) Wound healing assays (10X magnification)
demonstrate that S100A4 OE restores migratory ability of SACC
cells, with sh + OE cells showing closure rates comparable with NC.
(B) Transwell assays revealed restoration of invasive capacity in
sh + OE cells suggests a reversal of ILK knockdown-induced
inhibition. ***P<0.001, ****P<0.0001. ns, not significant;
OE, overexpression; ILK, integrin-linked kinase; SACC, salivary
adenoid cystic carcinoma; sh, short hairpin; NC, negative control;
CON, blank control.

Figure 7.

S100A4 OE rescues the inhibitory effects of ILK knockdown on the migration and invasion of adenoid cystic carcinoma. (A) Wound healing assays (10X magnification) demonstrate that S100A4 OE restores migratory ability of SACC cells, with sh + OE cells showing closure rates comparable with NC. (B) Transwell assays revealed restoration of invasive capacity in sh + OE cells suggests a reversal of ILK knockdown-induced inhibition. ***P<0.001, ****P<0.0001. ns, not significant; OE, overexpression; ILK, integrin-linked kinase; SACC, salivary adenoid cystic carcinoma; sh, short hairpin; NC, negative control; CON, blank control.

Upon examining their original raw data (which were also presented to the Editorial Office for our inspection), the authors realized that several inadvertent errors had been made in the presentation of Figs. 2 and 7 in the published paper. Specifically, the image for the 24 h, SACC-LM Control (CON) group in Fig. 7 was inadvertently duplicated and used for the 24 h, negative control (NC-S100A4) group of the same cell line. Secondly, the image intended for the 24 h, SACC-LM sh-ILK group in Fig. 7 had mistakenly been replaced by an image from a separate sh-ILK experiment performed with the SACC-83 cells. Thirdly, the western blot data depicting ILK and Snail expression in the SACC-83 cell line in Fig. 2 were inadvertently re-used to represent different experimental endpoints in Figs. 3 and 6.

Western blot analysis of the effects
of ILK knockdown on EMT markers and signaling. Results are shown as
the mean ± SD of three independent assays. *P<0.05, **P<0.01,
***P<0.001, ****P<0.0001 vs. control group. ns, not
significant.

Figure 2.

Western blot analysis of the effects of ILK knockdown on EMT markers and signaling. Results are shown as the mean ± SD of three independent assays. *P<0.05, **P<0.01, ***P<0.001, ****P<0.0001 vs. control group. ns, not significant.

The revised versions of Figs. 2 and 7, featuring the correct data for the ILK and Snail blots for the SACC-83 cell line in Fig. 2, and the correct data for the 24 h, NC-S100A4 and 24 h, sh-ILK experiments for the SACC-LM cell line in Fig. 7, are shown on the next two pages. The authors regret these errors were made in compiling these figures, which resulted from administrative oversights during the image selection and integration process, although note that these errors did not grossly affect either the results or the conclusions reported in this study. The authors are grateful to the editor of Oncology Letters for allowing them the opportunity to publish a Corrigendum; furthermore, they apologize to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Yang Y, Luo J, Li Y, Guo K, Ye L and Zhao D: [Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma. Oncol Lett 31: 136, 2026.
APA
Yang, Y., Luo, J., Li, Y., Guo, K., Ye, L., & Zhao, D. (2026). [Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma. Oncology Letters, 31, 136. https://doi.org/10.3892/ol.2026.15489
MLA
Yang, Y., Luo, J., Li, Y., Guo, K., Ye, L., Zhao, D."[Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma". Oncology Letters 31.4 (2026): 136.
Chicago
Yang, Y., Luo, J., Li, Y., Guo, K., Ye, L., Zhao, D."[Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma". Oncology Letters 31, no. 4 (2026): 136. https://doi.org/10.3892/ol.2026.15489
Copy and paste a formatted citation
x
Spandidos Publications style
Yang Y, Luo J, Li Y, Guo K, Ye L and Zhao D: [Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma. Oncol Lett 31: 136, 2026.
APA
Yang, Y., Luo, J., Li, Y., Guo, K., Ye, L., & Zhao, D. (2026). [Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma. Oncology Letters, 31, 136. https://doi.org/10.3892/ol.2026.15489
MLA
Yang, Y., Luo, J., Li, Y., Guo, K., Ye, L., Zhao, D."[Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma". Oncology Letters 31.4 (2026): 136.
Chicago
Yang, Y., Luo, J., Li, Y., Guo, K., Ye, L., Zhao, D."[Corrigendum] Mechanistic role of the ILK‑S100A4 axis in modulating invasion and EMT in salivary adenoid cystic carcinoma". Oncology Letters 31, no. 4 (2026): 136. https://doi.org/10.3892/ol.2026.15489
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