[Corrigendum] TERT promoter regulating melittin expression induces apoptosis and G<sub>0</sub>/G<sub>1</sub> cell cycle arrest in esophageal carcinoma cells

Zhou,Chao; Ma,Jie; Lu,Yuanhua; Zhao,Wan; Xu,Bingxue; Lin,Jian; Ma,Yongjun; Tian,Yafei; Zhang,Qi; Wang,Wei; Yan,Weiqun; Jiao,Ping

doi:10.3892/ol.2026.15651

July-2026 Volume 32 Issue 1

Full Size Image

Journals

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

Biomedical Reports

Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

Medicine International

An International Open Access Journal Devoted to General Medicine.

July-2026 Volume 32 Issue 1

Full Size Image

Correction Open Access

[Corrigendum] TERT promoter regulating melittin expression induces apoptosis and G₀/G₁ cell cycle arrest in esophageal carcinoma cells

Authors:
- Chao Zhou
- Jie Ma
- Yuanhua Lu
- Wan Zhao
- Bingxue Xu
- Jian Lin
- Yongjun Ma
- Yafei Tian
- Qi Zhang
- Wei Wang
- Weiqun Yan
- Ping Jiao
View Affiliations / Copyright

Affiliations: Department of Regenerative Medicine, School of Pharmaceutical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China

Copyright: © Zhou et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
Article Number: 296
|
Published online on: May 15, 2026

https://doi.org/10.3892/ol.2026.15651
Expand metrics +

Article

Oncol Lett 21: [Related article:] 16, 2021; DOI: 10.3892/ol.2020.12277

Subsequently to the publication of the above article, an interested reader drew to the authors’ attention that, concerning the cellular images shown in Fig. 3A on p. 5, the ‘Blank’ and ‘Con’ data panels showed an overlapping section, such that these were apparently derived from the same original source where the results of differently performed experiments were intended to have been portrayed.

Figure 3.

Transfection of pcTERT-Mel decreases mitochondrial membrane potential and increases ROS production in TE1 cells, leading to apoptosis. (A) Cells were stained with tetraethylbenzimidazolylcarbocyanine iodide and visualized using a fluorescence microscope at 24 h post-transfection. pcTERT treated cells and untreated cells stained red suggested normal high membrane potentials. pcTERT-Mel treatment caused a significant loss of red fluorescence and an increase of green fluorescence, indicating the loss of mitochondrial membrane potential, which was associated with apoptosis (original magnification, ×200). (B) ROS production was detected with a ROS assay kit. Increased ROS production was observed in pcTERT-melittin treated cells with a fluorescence microplate at excitation and emission wavelengths of 488 and 525 nm, respectively. (C) Quantification of the pcTERT-melittin transfection-induced apoptosis of TE1 cells, as assessed via flow cytometry using Annexin-V and PI staining at 24, 48 and 72 h post-transfection. The percentage of apoptotic cells was presented as the mean ± SEM. Results are an average of three independent experiments. *P<0.05 vs. Con group. TERT, telomerase reverse transcriptase; Con, control; Mel, melittin; ROS, reactive oxygen species.

After re-examining their original data, the authors have realized that some of the data in Fig. 3A were inadvertently assembled incorrectly. The revised version of Fig. 3, now showing the correct data for the ‘Blank’ panel in Fig. 3A, is shown on the next page. The authors are grateful to the Editor of Oncology Letters for allowing them this opportunity to publish a Corrigendum, and all the authors agree with its publication. Furthermore, the authors apologize to the readership for any inconvenience caused.

Journals