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Oncology Reports
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Print ISSN: 1021-335X Online ISSN: 1791-2431
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August-2021 Volume 46 Issue 2

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

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Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

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Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

World Academy of Sciences Journal

World Academy of Sciences Journal

Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

International Journal of Functional Nutrition

International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

Publishes open-access research on using epigenetics to advance understanding and treatment of human disease.

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Correction Open Access

[Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3

  • Authors:
    • Shu Liu
    • Xiaomeng Zhang
    • Chunmei Hu
    • Yingxue Wang
    • Chunling Xu

  • View Affiliations / Copyright

    Affiliations: Department of Ophthalmology, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China, Department of Tumor and Hematology, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China, Department of Electrical Diagnosis, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China
    Copyright: © Liu et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 175
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    Published online on: June 28, 2021
       https://doi.org/10.3892/or.2021.8126
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This article is mentioned in:



Article

Oncol Rep 39: [Related article:] 739-746, 2018; DOI: 10.3892/or.2017.6144

Following the publication of this article, an interested reader drew to the authors attention that some western blotting data bands had apparently been duplicated in Fig. 4D. The authors have re-examined their original data, and realized that this figure was assembled incorrectly.

Figure 4.

STAT3 is a potential direct target of miR-29a in RB. (A) The predicted binding sites and mutation sites for miR-29a in the 3′-UTR of STAT3 are shown. (B) miR-29a overexpression significantly decreased the luciferase activity of the WT-STAT3-3’-UTR but not that of the mutant STAT3-3′-UTR in Y79 and SO-RB50 cells. (C) Overexpression of miR-29a reduced the STAT3 mRNA expression levels in Y79 and SO-RB50 cells. GAPDH was used as an internal control. (D) Overexpression of miR-29a reduced the STAT3, cyclin D1, Bcl-2 and MMP-9 protein levels in Y79 and SO-RB50 cells. GAPDH was used as an internal control. **P<0.01..

The corrected version of Fig. 4 is shown below. The authors sincerely apologize for the errors that were introduced during the preparation of this figure, and thank the Editor for allowing them the opportunity to publish a Corrigendum. Furthermore, they regret any inconvenience caused to the readership.

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Copy and paste a formatted citation
Spandidos Publications style
Liu S, Zhang X, Hu C, Wang Y and Xu C: [Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3. Oncol Rep 46: 175, 2021.
APA
Liu, S., Zhang, X., Hu, C., Wang, Y., & Xu, C. (2021). [Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3. Oncology Reports, 46, 175. https://doi.org/10.3892/or.2021.8126
MLA
Liu, S., Zhang, X., Hu, C., Wang, Y., Xu, C."[Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3". Oncology Reports 46.2 (2021): 175.
Chicago
Liu, S., Zhang, X., Hu, C., Wang, Y., Xu, C."[Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3". Oncology Reports 46, no. 2 (2021): 175. https://doi.org/10.3892/or.2021.8126
Copy and paste a formatted citation
x
Spandidos Publications style
Liu S, Zhang X, Hu C, Wang Y and Xu C: [Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3. Oncol Rep 46: 175, 2021.
APA
Liu, S., Zhang, X., Hu, C., Wang, Y., & Xu, C. (2021). [Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3. Oncology Reports, 46, 175. https://doi.org/10.3892/or.2021.8126
MLA
Liu, S., Zhang, X., Hu, C., Wang, Y., Xu, C."[Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3". Oncology Reports 46.2 (2021): 175.
Chicago
Liu, S., Zhang, X., Hu, C., Wang, Y., Xu, C."[Corrigendum] miR‑29a inhibits human retinoblastoma progression by targeting STAT3". Oncology Reports 46, no. 2 (2021): 175. https://doi.org/10.3892/or.2021.8126
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