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Oncology Reports
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Print ISSN: 1021-335X Online ISSN: 1791-2431
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May-2023 Volume 49 Issue 5

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

International Journal of Oncology

International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

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Molecular Medicine Reports

Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

Oncology Reports

Oncology Reports

Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine

Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

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Oncology Letters

Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

Molecular and Clinical Oncology

Molecular and Clinical Oncology

International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

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Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

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International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

International Journal of Epigenetics

International Journal of Epigenetics

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Correction Open Access

[Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling

  • Authors:
    • Yan-Hong Liu
    • Man Zhu
    • Pan-Pan Lei
    • Xiao-Yan Pan
    • Wei-Na Ma
  • View Affiliations / Copyright

    Affiliations: School of Pharmacy, Health Science Center, Xi'an Jiaotong University, Xi'an, Shaanxi 710061, P.R. China
    Copyright: © Liu et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].
  • Article Number: 81
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    Published online on: March 3, 2023
       https://doi.org/10.3892/or.2023.8518
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Article

Oncol Rep 46: [Related article:] 136, 2021; DOI: 10.3892/or.2021.8087

Subsequently to the publication of this paper, the authors have realized that an error was made during the compilation of Fig. 2A as it appeared on p. 4. Essentially, the partial Q2-3 images of the ‘1.56 µm’ group were inadvertently copied across to the Q2-3 images of the ‘3.12 µm’ group, leading to the cell number of the Q2-3 quadrant being the same for both the 1.56 µm and the 3.12 µm groups, and also leading the total cell number of the 3.12 µm group being calculated as 106.97%, which was clearly incorrect (the total percentage should have added up to 100%).

Figure 2.

Effect of ND-09 treatment on cell apoptosis. The proportion of apoptotic cells was determined by double staining with Annexin V/FITC and PI in (A) K562, (B) JURKAT, and (C) HUT78 cells after treatment with ND-09 (0, 1.56, 3.12, and 6.25 µM). (D) Effect of ND-09 on mitochondrial membrane potential (Δψm). Δψm was assessed through flow cytometry following treatment of K562 cells with ND-09 (0, 1.56, 3.12, and 6.25 µΜ) for 48 h. (E) Quantitative analysis of flow cytometry data. (F) Effects of ND-09 on apoptosis-related protein expression in K562 cells. All results were quantified by densitometric analysis of the bands and were normalized to GAPDH (internal control). Samples were derived from the same experiment, and blots were processed in parallel. Values represent the average of three independent experiments. Data are presented as the mean ± SEM (n=3). *P<0.05, **P<0.01 compared with the untreated control group.

The corrected version of Fig. 2, showing the correct data for the Q2-3 images in the ‘3.12 µm’ group, is shown on the next page. Note that this error did not significantly affect the results or the conclusions reported in this paper, and all the authors agree with the publication of this Corrigendum. The authors are grateful to the Editor of Oncology Reports for allowing them this opportunity to publish a corrigendum, and apologize to the readership for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Liu Y, Zhu M, Lei P, Pan X and Ma W: [Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling. Oncol Rep 49: 81, 2023.
APA
Liu, Y., Zhu, M., Lei, P., Pan, X., & Ma, W. (2023). [Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling. Oncology Reports, 49, 81. https://doi.org/10.3892/or.2023.8518
MLA
Liu, Y., Zhu, M., Lei, P., Pan, X., Ma, W."[Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling". Oncology Reports 49.5 (2023): 81.
Chicago
Liu, Y., Zhu, M., Lei, P., Pan, X., Ma, W."[Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling". Oncology Reports 49, no. 5 (2023): 81. https://doi.org/10.3892/or.2023.8518
Copy and paste a formatted citation
x
Spandidos Publications style
Liu Y, Zhu M, Lei P, Pan X and Ma W: [Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling. Oncol Rep 49: 81, 2023.
APA
Liu, Y., Zhu, M., Lei, P., Pan, X., & Ma, W. (2023). [Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling. Oncology Reports, 49, 81. https://doi.org/10.3892/or.2023.8518
MLA
Liu, Y., Zhu, M., Lei, P., Pan, X., Ma, W."[Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling". Oncology Reports 49.5 (2023): 81.
Chicago
Liu, Y., Zhu, M., Lei, P., Pan, X., Ma, W."[Corrigendum] ND‑09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR‑ABL signaling". Oncology Reports 49, no. 5 (2023): 81. https://doi.org/10.3892/or.2023.8518
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