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Molecular Medicine Reports
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Print ISSN: 1791-2997 Online ISSN: 1791-3004
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November-2015 Volume 12 Issue 5

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International Journal of Molecular Medicine

International Journal of Molecular Medicine

International Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.

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International Journal of Oncology

International Journal of Oncology is an international journal devoted to oncology research and cancer treatment.

Molecular Medicine Reports

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Covers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.

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Oncology Reports is an international journal devoted to fundamental and applied research in Oncology.

Experimental and Therapeutic Medicine

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Experimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.

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Oncology Letters is an international journal devoted to Experimental and Clinical Oncology.

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Explores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.

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International journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.

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Multidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.

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International Journal of Functional Nutrition

Open-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.

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Correction Open Access

[Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma

  • Authors:
    • Liang Chang
    • Dan Zhao
    • Hui‑Bin Liu
    • Qiu‑Shi Wang
    • Ping Zhang
    • Chen‑Long Li
    • Wen‑Zhong Du
    • Hong‑Jun Wang
    • Xing Liu
    • Zhi‑Ren Zhang
    • Chuan‑Lu Jiang
  • View Affiliations / Copyright

    Affiliations: Department of Neurosurgery, The Second Affiliated Hospital, Harbin Medical University, Harbin, Heilongjiang 150086, P.R. China, Department of Clinical Pharmacy, The Second Affiliated Hospital, Harbin Medical University, Harbin, Heilongjiang 150086, P.R. China
    Copyright: © Chang et al. This is an open access article distributed under the terms of Creative Commons Attribution License.
  • Pages: 7815-7815
    |
    Published online on: October 1, 2015
       https://doi.org/10.3892/mmr.2015.4414
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Article

Mol Med Rep 12: [Related article:] 6702–6710, 2015; DOI: 10.3892/mmr.2015.4229

After the publication of the article, it has been brought to the authors' attention by an interested reader that we had made an error regarding the presentation of certain data in the manuscript. The error relates to the presentation of Figs. 1 and 2 in the paper: The control panels for Fig. 1C [labelled 'cyclopamine (µM)'] and Figs. 2B and C [labelled 'rhSSH (µg/ml)'] were derived from the same image. The control U251 cells, featured in Fig. 1 and Figs. 2B and C, were treated without cyclopamine and rhSHH. Therefore, the U251 cells treated without cyclopamine and rhSHH were considered as a control group compared with U251 cells that were separately treated with cyclopamine or rhSHH, and these were photographed randomly. A new Fig. 2 is provided, which contains the correct data for the control panels for Figs. 2B and C.

Figure 2

rhSHH enhances the adhesion, invasion and migration of U251 cells. (A) U251 cells were incubated for 24 h with various concentrations of rhSHH. Cells were seeded into 96-well plates coated with fibronectin. After 1 h, the adhered cells were analyzed by MTT assay. The adhesion rate was expressed as a percentage of the control (0 µg/ml). (B) U251 cells were seeded in the upper wells without matrigel coating and treated with various concentrations of rhSHH. After 12 h, cells on the bottom side of the filter were fixed, stained and counted (magnification, ×400). The migration rate was expressed as a percentage of the control (0 µg/ml). (C) U251 cells were seeded in the upper wells with matrigel coating and treated with various concentrations of rhSHH. After 24 h, cells on the bottom side of the filter were fixed, stained and counted (magnification, x400). The invasion rate was expressed as a percentage of the control (0 µg/ml). Values are expressed as the mean ± standard deviation of three independent experiments. *P<0.001, compared with controls. rhSHH, recombinant human sonic hedgehog N-terminal peptide.

The selection of the same image for the control panels in Figs. 1 and 2 do not affect the overall conclusions in the paper. We would like to thank the reader who pointed out this error, and to apologize for any inconvenience caused.

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Copy and paste a formatted citation
Spandidos Publications style
Chang L, Zhao D, Liu HB, Wang QS, Zhang P, Li CL, Du WZ, Wang HJ, Liu X, Zhang ZR, Zhang ZR, et al: [Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma. Mol Med Rep 12: 7815-7815, 2015.
APA
Chang, L., Zhao, D., Liu, H., Wang, Q., Zhang, P., Li, C. ... Jiang, C. (2015). [Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma. Molecular Medicine Reports, 12, 7815-7815. https://doi.org/10.3892/mmr.2015.4414
MLA
Chang, L., Zhao, D., Liu, H., Wang, Q., Zhang, P., Li, C., Du, W., Wang, H., Liu, X., Zhang, Z., Jiang, C."[Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma". Molecular Medicine Reports 12.5 (2015): 7815-7815.
Chicago
Chang, L., Zhao, D., Liu, H., Wang, Q., Zhang, P., Li, C., Du, W., Wang, H., Liu, X., Zhang, Z., Jiang, C."[Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma". Molecular Medicine Reports 12, no. 5 (2015): 7815-7815. https://doi.org/10.3892/mmr.2015.4414
Copy and paste a formatted citation
x
Spandidos Publications style
Chang L, Zhao D, Liu HB, Wang QS, Zhang P, Li CL, Du WZ, Wang HJ, Liu X, Zhang ZR, Zhang ZR, et al: [Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma. Mol Med Rep 12: 7815-7815, 2015.
APA
Chang, L., Zhao, D., Liu, H., Wang, Q., Zhang, P., Li, C. ... Jiang, C. (2015). [Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma. Molecular Medicine Reports, 12, 7815-7815. https://doi.org/10.3892/mmr.2015.4414
MLA
Chang, L., Zhao, D., Liu, H., Wang, Q., Zhang, P., Li, C., Du, W., Wang, H., Liu, X., Zhang, Z., Jiang, C."[Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma". Molecular Medicine Reports 12.5 (2015): 7815-7815.
Chicago
Chang, L., Zhao, D., Liu, H., Wang, Q., Zhang, P., Li, C., Du, W., Wang, H., Liu, X., Zhang, Z., Jiang, C."[Corrigendum] Activation of sonic hedgehog signaling enhances cell migration and invasion by induction of matrix metalloproteinase-2 and -9 via the phosphoinositide-3 kinase/AKT signaling pathway in glioblastoma". Molecular Medicine Reports 12, no. 5 (2015): 7815-7815. https://doi.org/10.3892/mmr.2015.4414
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