|
1
|
Gilden D, Mahalingam R, Nagel MA,
Pugazhenthi S and Cohrs RJ: Review: The neurobiology of varicella
zoster virus infection. Neuropathol Appl Neurobiol. 37:441–463.
2011. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Yoleri O, Olmez N, Oztura I, Sengül I,
Günaydin R and Memiş A: Segmental zoster paresis of the upper
extremity: a case report. Arch Phys Med Rehabil. 86:1492–1494.
2005. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Oliver SL, Brady JJ, Sommer MH, et al: An
immunoreceptor tyrosine-based inhibition motif in varicella-zoster
virus glycoprotein B regulates cell fusion and skin pathogenesis.
Proc Natl Acad Sci USA. 110:1911–1916. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Arvin AM, Moffat JF, Sommer M, et al:
Varicella-zoster virus T cell tropism and the pathogenesis of skin
infection. Curr Top Microbiol Immunol. 342:189–209. 2010.PubMed/NCBI
|
|
5
|
Zerboni L, Ku CC, Jones CD, Zehnder JL and
Arvin AM: Varicella-zoster virus infection of human dorsal root
ganglia in vivo. Proc Natl Acad Sci USA. 102:6490–6495. 2005.
View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Berkhout B and Jeang KT: RISCy business:
MicroRNAs, pathogenesis, and viruses. J Biol Chem. 282:26641–26645.
2007. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Grassmann R and Jeang KT: The roles of
microRNAs in mammalian virus infection. Biochim Biophys Acta.
1779:706–711. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Bartels CL and Tsongalis GJ: MicroRNAs:
novel biomarkers for human cancer. Clin Chem. 55:623–631. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
9
|
Huang J, Wang F, Argyris E, et al:
Cellular microRNAs contribute to HIV-1 latency in resting primary
CD4+ T lymphocytes. Nat Med. 13:1241–1247. 2007.PubMed/NCBI
|
|
10
|
Gao L, Guo XK, Wang L, et al: MicroRNA 181
suppresses porcine reproductive and respiratory syndrome virus
(PRRSV) infection by targeting PRRSV receptor CD163. J Virol.
87:8808–8812. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Jung YJ, Kim JW, Park SJ, et al:
c-Myc-mediated overexpression of miR-17-92 suppresses replication
of hepatitis B virus in human hepatoma cells. J Med Virol.
85:969–978. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Jin J, Tang S, Xia L, et al: MicroRNA-501
promotes HBV replication by targeting HBXIP. Biochem Biophys Res
Commun. 430:1228–1233. 2013. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Ye X, Hemida MG, Qiu Y, Hanson PJ, Zhang
HM and Yang D: MiR-126 promotes coxsackievirus replication by
mediating cross-talk of ERK1/2 and Wnt/β-catenin signal pathways.
Cell Mol Life Sci. 70:4631–4644. 2013.PubMed/NCBI
|
|
14
|
Kumarswamy R, Volkmann I and Thum T:
Regulation and function of miRNA-21 in health and disease. RNA
Biol. 8:706–713. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Chen Y, Chen J, Wang H, et al: HCV-induced
miR-21 contributes to evasion of host immune system by targeting
MyD88 and IRAK1. PLoS Pathog. 9:e10032482013. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Keck K, Volper EM, Spengler RM, et al:
Rational design leads to more potent RNA interference against
hepatitis B virus: factors affecting silencing efficiency. Mol
Ther. 17:538–547. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Rosato P, Anastasiadou E, Garg N, et al:
Differential regulation of miR-21 and miR-146a by Epstein-Barr
virus-encoded EBNA2. Leukemia. 26:2343–2352. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
18
|
van der Fits L, van Kester MS, Qin Y, et
al: MicroRNA-21 expression in CD4+ T cells is regulated by STAT3
and is pathologically involved in Sézary syndrome. J Invest
Dermatol. 131:762–768. 2011.
|
|
19
|
Jarnicki A, Putoczki T and Ernst M: Stat3:
linking inflammation to epithelial cancer - more than a ‘gut’
feeling? Cell Div. 5:142010.PubMed/NCBI
|
|
20
|
Al Zaid Siddiquee K and Turkson J: STAT3
as a target for inducing apoptosis in solid and hematological
tumors. Cell Res. 18:254–267. 2008.PubMed/NCBI
|
|
21
|
Wang Z, Luo F, Li L, et al: STAT3
activation induced by Epstein-Barr virus latent membrane protein1
causes vascular endothelial growth factor expression and cellular
invasiveness via JAK3 and ERK signaling. Eur J Cancer.
46:2996–3006. 2010. View Article : Google Scholar
|
|
22
|
Punjabi AS, Carroll PA, Chen L and
Lagunoff M: Persistent activation of STAT3 by latent Kaposi’s
sarcoma-associated herpesvirus infection of endothelial cells. J
Virol. 81:2449–2458. 2007.PubMed/NCBI
|
|
23
|
Sen N, Che X, Rajamani J, et al: Signal
transducer and activator of transcription 3 (STAT3) and survivin
induction by varicella-zoster virus promote replication and skin
pathogenesis. Proc Natl Acad Sci USA. 109:600–605. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Holtick U, Vockerodt M, Pinkert D, et al:
STAT3 is essential for Hodgkin lymphoma cell proliferation and is a
target of tyrphostin AG17 which confers sensitization for
apoptosis. Leukemia. 19:936–944. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
25
|
Kohanbash G and Okada H: MicroRNAs and
STAT interplay. Semin Cancer Biol. 22:70–75. 2012. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Ventura A, Young AG, Winslow MM, et al:
Targeted deletion reveals essential and overlapping functions of
the miR-17 through 92 family of miRNA clusters. Cell. 132:875–886.
2008. View Article : Google Scholar : PubMed/NCBI
|
|
27
|
Carraro G, El-Hashash A, Guidolin D, et
al: miR-17 family of microRNAs controls FGF10-mediated embryonic
lung epithelial branching morphogenesis through MAPK14 and STAT3
regulation of E-Cadherin distribution. Dev Biol. 333:238–250. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Löffler D, Brocke-Heidrich K, Pfeifer G,
et al: Interleukin-6 dependent survival of multiple myeloma cells
involves the Stat3-mediated induction of microRNA-21 through a
highly conserved enhancer. Blood. 110:1330–1333. 2007.PubMed/NCBI
|
|
29
|
Ohno M, Natsume A, Kondo Y, et al: The
modulation of microRNAs by type I IFN through the activation of
signal transducers and activators of transcription 3 in human
glioma. Mol Cancer Res. 7:2022–2030. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Cullen BR: MicroRNAs as mediators of viral
evasion of the immune system. Nat Immunol. 14:205–210. 2013.
View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Sen N, Sommer M, Che X, White K, Ruyechan
WT and Arvin AM: Varicella-zoster virus immediate-early protein 62
blocks interferon regulatory factor 3 (IRF3) phosphorylation at key
serine residues: a novel mechanism of IRF3 inhibition among
herpesviruses. J Virol. 84:9240–9253. 2010. View Article : Google Scholar
|
|
32
|
Zhu H, Zheng C, Xing J, et al:
Varicella-zoster virus immediate-early protein ORF61 abrogates the
IRF3-mediated innate immune response through degradation of
activated IRF3. J Virol. 85:11079–11089. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Sloan E, Henriquez R, Kinchington PR,
Slobedman B and Abendroth A: Varicella-zoster virus inhibition of
the NF-κB pathway during infection of human dendritic cells: role
for open reading frame 61 as a modulator of NF-κB activity. J
Virol. 86:1193–1202. 2012.
|
