1
|
Hulsebosch CE, Hains BC, Crown ED and
Carlton SM: Mechanisms of chronic central neuropathic pain after
spinal cord injury. Brain Res Rev. 60:202–213. 2009. View Article : Google Scholar : PubMed/NCBI
|
2
|
Xu XJ, Hao JX, Aldskogius H, Seiger A and
Wiesenfeld-Hallin Z: Chronic pain-related syndrome in rats after
ischemic spinal cord lesion: a possible animal model for pain in
patients with spinal cord injury. Pain. 48:279–290. 1992.
View Article : Google Scholar : PubMed/NCBI
|
3
|
Gwak YS, Crown ED, Unabia GC and
Hulsebosch CE: Propentofylline attenuates allodynia, glial
activation and modulates GABAergic tone after spinal cord injury in
the rat. Pain. 138:410–422. 2008. View Article : Google Scholar : PubMed/NCBI
|
4
|
Bruce JC, Oatway MA and Weaver LC: Chronic
pain after clip-compression injury of the rat spinal cord. Exp
Neurol. 178:33–48. 2002. View Article : Google Scholar : PubMed/NCBI
|
5
|
Yezierski RP, Liu S, Ruenes GL, Kajander
KJ and Brewer KL: Excitotoxic spinal cord injury: behavioral and
morphological characteristics of a central pain model. Pain.
75:141–155. 1998. View Article : Google Scholar : PubMed/NCBI
|
6
|
Gomes-Leal W, Corkill DJ, Freire MA,
Picanço-Diniz CW and Perry VH: Astrocytosis, microglia activation,
oligodendrocyte degeneration, and pyknosis following acute spinal
cord injury. Exp Neurol. 190:456–467. 2004. View Article : Google Scholar : PubMed/NCBI
|
7
|
Coderre TJ, Xanthos DN, Francis L and
Bennett GJ: Chronic post-ischemia pain (CPIP): a novel animal model
of complex regional pain syndrome-type 1 (CRPS-I; reflex
sympathetic dystrophy) produced by prolonged hindpaw ischemia and
reperfusion in the rat. Pain. 112:94–105. 2004. View Article : Google Scholar
|
8
|
Bodera P, Stankiewicz W, Zawada K,
Antkowiak B, Paluch M, Kieliszek J, Kalicki B, Bartosiński A and
Wawer I: Changes in antioxidant capacity of blood due to mutual
action of electromagnetic field (1800 MHz) and opioid drug
(tramadol) in animal model of persistent inflammatory state.
Pharmacol Rep. 65:421–428. 2013. View Article : Google Scholar
|
9
|
Kwak KH, Han CG, Lee SH, Jeon Y, Park SS,
et al: Reactive oxygen species in rats with chronic post ischemia
pain. Acta Anaesthesiol Scand. 53:648–656. 2009. View Article : Google Scholar : PubMed/NCBI
|
10
|
Hacimuftuoglu A, Handy CR, Goettl VM, Lin
CG, Dane S and Stephens RL Jr: Antioxidants attenuate multiple
phases of formalin induced nociceptive response in mice. Behav
Brain Res. 173:211–216. 2006. View Article : Google Scholar : PubMed/NCBI
|
11
|
Guedes RP, Bosco LD, Teixeira CM, et al:
Neuropathic pain modifies antioxidant activity in rat spinal cord.
Neurochem Res. 31:603–609. 2006. View Article : Google Scholar : PubMed/NCBI
|
12
|
Park ES, Gao X, Chung JM and Chung K:
Levels of mitochondrial reactive oxygen species increase in rat
neuropathic spinal dorsal horn neurons. Neurosci Lett. 391:108–111.
2006. View Article : Google Scholar : PubMed/NCBI
|
13
|
Gao X, Kim HK, Chung JM and Chung K:
Reactive oxygen species (ROS) are involved in enhancement of
NMDA-receptor phosphorylation in animal models of pain. Pain.
131:262–271. 2007. View Article : Google Scholar : PubMed/NCBI
|
14
|
Zou X, Lin Q and Wills WD: Enhanced
phosphorylation of NMDA receptor 1 subunits in spinal cord dorsal
horn and spinothalamic tract neurons after intradermal injection of
capsaicin in rats. J Neurosci. 20:6989–6997. 2000.
|
15
|
Yezierski RP: Pain following spinal cord
injury: the clinical problem and experimental studies. Pain.
68:185–189. 1996. View Article : Google Scholar : PubMed/NCBI
|
16
|
Allen JW and Yaksh TL: Tissue injury
models of persistent nociception in rats. Methods Mol Med.
99:25–34. 2004.PubMed/NCBI
|
17
|
Zou X and Willis WD: Role of protein
kinase A in phosphorylation of NMDA receptor 1 subunits in dorsal
horn and spinothalamic tract neurons after intradermal injection of
capsaicin in rats. Neuroscience. 115:775–786. 2002. View Article : Google Scholar : PubMed/NCBI
|
18
|
Levy D and Zochodne DW: Local nitric oxide
synthase activity in a model of neuropathic pain. Eur J Neurosci.
10:1846–1855. 1998. View Article : Google Scholar : PubMed/NCBI
|
19
|
Caban A, Oczkowicz G and Cierpka L:
Influence of ischemic precondition and nitric oxide on
microcirculation and the degree of rat liver injury in the model of
ischemia and reperfusion. Transplant Proc. 38:196–198. 2006.
View Article : Google Scholar : PubMed/NCBI
|
20
|
Halliwell B and Gutteridge JMC: Free
Radicals in Biology and Medicine. Oxford University Press; New
York: pp. 222007
|
21
|
Li C and Jackson RM: Reactive species
mechanisms of cellular hypoxia-reoxygenation injury. Am J Physiol
Cell Physiol. 282:C227–C241. 2002. View Article : Google Scholar : PubMed/NCBI
|
22
|
Kim KW, Ha MJ, Jung KY, et al: Reactive
oxygen species and N-methyl-D-aspartate receptor-mediated central
sensitization in hindlimb ischemia/reperfusion injury-induced
neuropathic pain rats. Korean J Anesthesiol. 56:186–194. 2009.
View Article : Google Scholar
|
23
|
Muscoli C, Mollace V, Wheatley J, et al:
Superoxide-mediated nitration of spinal manganese superoxide
dismutase: a novel pathway in N-methyl-D-aspartate-mediated
hyperalgesia. Pain. 111:96–103. 2010. View Article : Google Scholar : PubMed/NCBI
|
24
|
Mori H and Mishina M: Structure and
function of the NMDA receptor channel. Neuropharmacology.
34:1219–1237. 1995. View Article : Google Scholar : PubMed/NCBI
|
25
|
Willis WD: Long-term potentiation in
spinothalamic neurons. Brain Res. 40:202–214. 2002. View Article : Google Scholar : PubMed/NCBI
|
26
|
Sandkühler J: Learning and memory in pain
pathways. Pain. 88:113–118. 2000.
|
27
|
Boyce S, Wyatt A, Webb JK, et al:
Selective NMDA NR2B antagonists induce antinociception without
motor dysfunction: correlation with restricted localisation of NR2B
subunit in dorsal horn. Neuropharmacology. 38:611–623. 1999.
View Article : Google Scholar
|
28
|
Bedi SS, Yang Q, Crook RJ, et al: Chronic
spontaneous activity generated in the somatic of primary
nociceptors is associated with pain-related behavior following
spinal cord injury. J Neurosci. 30:14870–14882. 2010. View Article : Google Scholar : PubMed/NCBI
|