|
1
|
Sarin SK, Kumar A, Almeida JA, et al:
Acute-on-chronic liver failure: consensus recommendations of the
Asian Pacific Association for the study of the liver (APASL).
Hepatol Int. 3:269–282. 2009. View Article : Google Scholar
|
|
2
|
Shawcross DL, Wright GA, Stadlbauer V, et
al: Ammonia impairs neutrophil phagocytic function in liver
disease. Hepatology. 48:1202–1212. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Tandon P and Garcia-Tsao G: Bacterial
infections, sepsis, and multiorgan failure in cirrhosis. Semin
Liver Dis. 28:26–42. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Mookerjee RP, Stadlbauer V, Lidder S, et
al: Neutrophil dysfunction in alcoholic hepatitis superimposed on
cirrhosis is reversible and predicts the outcome. Hepatology.
46:831–840. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Bone RC, Grodzin CJ and Balk RA: Sepsis: a
new hypothesis for pathogenesis of the disease process. Chest.
112:235–243. 1997. View Article : Google Scholar : PubMed/NCBI
|
|
6
|
Cazzaniga M, Dionigi E, Gobbo G, et al:
The systemic inflammatory response syndrome in cirrhotic patients:
relationship with their in-hospital outcome. J Hepatol. 51:475–482.
2009. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Rolando N, Wade J, Davalos M, et al: The
systemic inflammatory response syndrome in acute liver failure.
Hepatology. 32:734–739. 2000. View Article : Google Scholar : PubMed/NCBI
|
|
8
|
Li J, Kokkola R, Tabibzadeh S, et al:
Structural basis for the proinflammatory cytokine activity of high
mobility group box 1. Mol Med. 9:37–45. 2003.PubMed/NCBI
|
|
9
|
Wang H, Yang H and Tracey KJ:
Extracellular role of HMGB1 in inflammation and sepsis. J Intern
Med. 255:320–331. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Scaffidi P, Misteli T and Bianchi ME:
Release of chromatin protein HMGB1 by necrotic cells triggers
inflammation. Nature. 418:191–195. 2002. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Wang H, Bloom O, Zhang M, et al: HMG-1 as
a late mediator of endotoxin lethality in mice. Science.
285:248–251. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
12
|
Yasuda T, Ueda T, Shinzeki M, et al:
Increase of high-mobility group box chromosomal protein 1 in blood
and injured organs in experimental severe acute pancreatitis.
Pancreas. 34:487–488. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Ueno H, Matsuda T, Hashimoto S, et al:
Contributions of high mobility group box protein in experimental
and clinical acute lung injury. Am J Respir Crit Care Med.
170:1310–1316. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Hamada T, Torikai M, Kuwazuru A, et al:
Extracellular high mobility group box chromosomal protein 1 is a
coupling factor for hypoxia and inflammation in arthritis.
Arthritis Rheum. 58:2675–2685. 2008. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Fan J, Li Y, Levy RM, et al: Hemorrhagic
shock induces NAD(P)H oxidase activation in neutrophils: role of
HMGB1-TLR4 signaling. J Immunol. 178:6573–6580. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
16
|
Tsung A, Sahai R, Tanaka H, et al: The
nuclear factor HMGB1 mediates hepatic injury after murine liver
ischemia-reperfusion. J Exp Med. 201:1135–1143. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
17
|
Liu HB, Fan XG, Huang JJ, et al: Serum
level of HMGB1 in patients with hepatitis B and its clinical
significance. Zhonghua Gan Zang Bing Za Zhi. 15:812–815. 2007.(In
Chinese).
|
|
18
|
Oshima G, Shinoda M, Tanabe M, et al:
Increased plasma levels of high mobility group box 1 in patients
with acute liver failure. Eur Surg Res. 48:154–162. 2012.
View Article : Google Scholar : PubMed/NCBI
|
|
19
|
Duan XZ, Hu JH, Li C, et al: Relation
between serum levels of high mobility group box 1 and hepatitis B
virus-related acute-on-chronic liver failure. Zhonghua Gan Zang
Bing Za Zhi. 21:434–437. 2013.(In Chinese).
|
|
20
|
Saijyo S, Kudo T, Suzuki M, et al:
Establishment of a new extrahepatic bile duct carcinoma cell line,
TFK-1. Tohoku J Exp Med. 177:61–71. 1995. View Article : Google Scholar : PubMed/NCBI
|
|
21
|
Huang D, Gao Q, Guo L, et al: Isolation
and identification of cancer stem-like cells in esophageal
carcinoma cell lines. Stem Cells Dev. 18:465–473. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
22
|
Li LW, Yu XY, Yang Y, et al: Expression of
esophageal cancer related gene 4 (ECRG4), a novel tumor suppressor
gene, in esophageal cancer and its inhibitory effect on the tumor
growth in vitro and in vivo. Int J Cancer. 125:1505–1513. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
23
|
Zhou RR, Zhao SS, Zou MX, et al: HMGB1
cytoplasmic translocation in patients with acute liver failure. BMC
Gastroenterol. 11:212011. View Article : Google Scholar : PubMed/NCBI
|
|
24
|
Liver Failure and Artificial Liver Group,
Chinese Society of Infectious Diseases, Chinese Medical
Association; Severe Liver Diseases and Artificial Liver Group,
Chinese Society of Hepatology, Chinese Medical Association.
Diagnostic and treatment guidelines for liver failure. Zhonghua Gan
Zang Bing Za Zhi. 14:643–646. 2006.(In Chinese).
|
|
25
|
Leifeld L, Dumoulin FL, Purr I, et al:
Early up-regulation of chemokine expression in fulminant hepatic
failure. J Pathol. 199:335–344. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
26
|
Ambrosino G, Naso A, Feltracco P, et al:
Cytokines and liver failure: modification of TNF- and IL-6 in
patients with acute on chronic liver decompensation treated with
Molecular Adsorbent Recycling System (MARS). Acta Biomed. 74(Suppl
2): 7–9. 2003.PubMed/NCBI
|
|
27
|
Liu S, Stolz DB, Sappington PL, et al:
HMGB1 is secreted by immunostimulated enterocytes and contributes
to cytomix-induced hyperpermeability of Caco-2 monolayers. Am J
Physiol Cell Physiol. 290:C990–C999. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
28
|
Wang H, Vishnubhakat JM, Bloom O, et al:
Proinflammatory cytokines (tumor necrosis factor and interleukin 1)
stimulate release of high mobility group protein-1 by pituicytes.
Surgery. 126:389–392. 1999. View Article : Google Scholar : PubMed/NCBI
|
|
29
|
Sitia G, Iannacone M, Muller S, Bianchi ME
and Guidotti LG: Treatment with HMGB1 inhibitors diminishes
CTL-induced liver disease in HBV transgenic mice. J Leukoc Biol.
81:100–107. 2007. View Article : Google Scholar : PubMed/NCBI
|
|
30
|
Rovere-Querini P, Capobianco A, Scaffidi
P, et al: HMGB1 is an endogenous immune adjuvant released by
necrotic cells. EMBO Rep. 5:825–830. 2004. View Article : Google Scholar : PubMed/NCBI
|
|
31
|
Bell CW, Jiang W, Reich CF III and
Pisetsky DS: The extracellular release of HMGB1 during apoptotic
cell death. Am J Physiol Cell Physiol. 291:C1318–C1325. 2006.
View Article : Google Scholar : PubMed/NCBI
|
|
32
|
Feghali K, Iwasaki K, Tanaka K, et al:
Human gingival fibroblasts release high-mobility group box-1
protein through active and passive pathways. Oral Microbiol
Immunol. 24:292–298. 2009. View Article : Google Scholar : PubMed/NCBI
|
|
33
|
Katoonizadeh A, Laleman W, Verslype C, et
al: Early features of acute-on-chronic alcoholic liver failure: a
prospective cohort study. Gut. 59:1561–1569. 2010. View Article : Google Scholar : PubMed/NCBI
|
|
34
|
Pisetsky DS, Erlandsson-Harris H and
Andersson U: High-mobility group box protein 1 (HMGB1): an alarmin
mediating the pathogenesis of rheumatic disease. Arthritis Res
Ther. 10:2092008. View
Article : Google Scholar : PubMed/NCBI
|
|
35
|
Andersson U and Tracey KJ: HMGB1 is a
therapeutic target for sterile inflammation and infection. Annu Rev
Immunol. 29:139–162. 2011. View Article : Google Scholar : PubMed/NCBI
|
|
36
|
Duan XZ, Hu JH, Li C, et al: Relation
between serum levels of high mobility group box 1 and hepatitis B
virus-related acute-on-chronic liver failure. Zhonghua Gan Zang
Bing Za Zhi. 21:434–437. 2013.(In Chinese).
|
