1
|
Lang VB, Baretić M and Pavić E: Kidney
disease in diabetic patients-the role of family medicine physician.
Acta Med Croatica. 70:319–324. 2016.(In Croatian). PubMed/NCBI
|
2
|
Campos EJ, Campos A, Martins J and
Ambrósio AF: Opening eyes to nanomedicine: Where we are, challenges
and expectations on nanotherapy for diabetic retinopathy.
Nanomedicine. 13:2101–2113. 2017. View Article : Google Scholar : PubMed/NCBI
|
3
|
Richter GM, Choudhury F, Torres M, Azen SP
and Varma R: Los Angeles Latino Eye Study Group: Risk factors for
incident cortical, nuclear, posterior subcapsular, and mixed lens
opacities: The Los Angeles Latino eye study. Ophthalmology.
119:2040–2047. 2012. View Article : Google Scholar : PubMed/NCBI
|
4
|
Zablocki GJ, Ruzycki PA, Overturf MA,
Palla S, Reddy GB and Petrash JM: Aldose reductase-mediated
induction of epithelium-to-mesenchymal transition (EMT) in lens.
Chem Biol Interact. 191:351–356. 2011. View Article : Google Scholar : PubMed/NCBI
|
5
|
Hegde KR and Varma SD: Cataracts in
experimentally diabetic mouse: Morphological and apoptotic changes.
Diabetes Obes Metab. 7:200–204. 2005. View Article : Google Scholar : PubMed/NCBI
|
6
|
Cho HG and Yoo J: Rho activation is
required for transforming growth factor-beta-induced
epithelial-mesenchymal transition in lens epithelial cells. Cell
Biol Int. 31:1225–1230. 2007. View Article : Google Scholar : PubMed/NCBI
|
7
|
Struck HG, Heider C and Lautenschläger C:
Changes in the lens epithelium of diabetic and non-diabetic
patients with various forms of opacities in senile cataract. Klin
Monbl Augenheilkd. 216:204–209. 2000.(In German). PubMed/NCBI
|
8
|
Deckers M, van Dinther M, Buijs J, Que I,
Löwik C, van der Pluijm G and ten Dijke P: The tumor suppressor
Smad4 is required for transforming growth factor beta-induced
epithelial to mesenchymal transition and bone metastasis of breast
cancer cells. Cancer Res. 66:2202–2209. 2006. View Article : Google Scholar : PubMed/NCBI
|
9
|
Valcourt U, Kowanetz M, Niimi H, Heldin CH
and Moustakas A: TGF-beta and the Smad signaling pathway support
transcriptomic reprogramming during epithelial-mesenchymal cell
transition. Mol Biol Cell. 16:1987–2002. 2005. View Article : Google Scholar : PubMed/NCBI
|
10
|
Tsapara A, Luthert P, Greenwood J, Hill
CS, Matter K and Balda MS: The RhoA activator GEF-H1/Lfc is a
transforming growth factor-beta target gene and effector that
regulates alpha-smooth muscle actin expression and cell migration.
Mol Biol Cell. 21:860–870. 2010. View Article : Google Scholar : PubMed/NCBI
|
11
|
Sebe A, Leivonen SK, Fintha A, Masszi A,
Rosivall L, Kähäri VM and Mucsi I: Transforming growth
factor-beta-induced alpha-smooth muscle cell actin expression in
renal proximal tubular cells is regulated by p38beta
mitogen-activated protein kinase, extracellular signal-regulated
protein kinase1,2 and the Smad signalling during
epithelial-myofibroblast transdifferentiation. Nephrol Dial
Transplant. 23:1537–1545. 2008. View Article : Google Scholar : PubMed/NCBI
|
12
|
Gordon-Thomson C, de Iongh RU, Hales AM,
Chamberlain CG and McAvoy JW: Differential cataractogenic potency
of TGF-beta1, -beta2, and -beta3 and their expression in the
postnatal rat eye. Invest Ophthalmol Vis Sci. 39:1399–1409.
1998.PubMed/NCBI
|
13
|
Hales AM, Chamberlain CG and McAvoy JW:
Cataract induction in lenses cultured with transforming growth
factor-beta. Invest Ophthalmol Vis Sci. 36:1709–1713.
1995.PubMed/NCBI
|
14
|
Liu J, Hales AM, Chamberlain CG and McAvoy
JW: Induction of cataract-like changes in rat lens epithelial
explants by transforming growth factor beta. Invest Ophthalmol Vis
Sci. 35:388–401. 1994.PubMed/NCBI
|
15
|
Saika S, Kono-Saika S, Ohnishi Y, Sato M,
Muragaki Y, Ooshima A, Flanders KC, Yoo J, Anzano M, Liu CY, et al:
Smad3 signaling is required for epithelial-mesenchymal transition
of lens epithelium after injury. Am J Pathol. 164:651–663. 2004.
View Article : Google Scholar : PubMed/NCBI
|
16
|
Du L, Hao M, Li C, Wu W, Wang W, Ma Z,
Yang T, Zhang N, Isaaca AT, Zhu X, et al: Quercetin inhibited
epithelial mesenchymal transition in diabetic rats,
high-glucose-cultured lens, and SRA01/04 cells through transforming
growth factor-β2/phosphoinositide 3-kinase/Akt pathway. Mol Cell
Endocrinol. 452:44–56. 2017. View Article : Google Scholar : PubMed/NCBI
|
17
|
Kim YS, Kim NH, Jung DH, Jang DS, Lee YM,
Kim JM and Kim JS: Genistein inhibits aldose reductase activity and
high glucose-induced TGF-beta2 expression in human lens epithelial
cells. Eur J Pharmacol. 594:18–25. 2008. View Article : Google Scholar : PubMed/NCBI
|
18
|
Kim NH, Kim YS, Jung DH and Kim JS:
KIOM-79 prevents xylose-induced lens opacity and inhibits TGF-beta2
in human lens epithelial cells cultured under high glucose. J
Ethnopharmacol. 130:599–606. 2010. View Article : Google Scholar : PubMed/NCBI
|
19
|
Lu Q, Yang T, Zhang M, Du L, Liu L, Zhang
N, Guo H, Zhang F, Hu G and Yin X: Preventative effects of Ginkgo
biloba extract (EGb761) on high glucose-cultured opacity of rat
lens. Phytother Res. 28:767–773. 2014. View
Article : Google Scholar : PubMed/NCBI
|
20
|
MacKay CE and Knock GA: Control of
vascular smooth muscle function by Src-family kinases and reactive
oxygen species in health and disease. J Physiol. 593:3815–3828.
2015. View Article : Google Scholar : PubMed/NCBI
|
21
|
Hovater MB and Sanders PW: Effect of
dietary salt on regulation of TGF-β in the kidney. Semin Nephrol.
32:269–276. 2012. View Article : Google Scholar : PubMed/NCBI
|
22
|
Montagner A, Delgado MB, Tallichet-Blanc
C, Chan JS, Sng MK, Mottaz H, Degueurce G, Lippi Y, Moret C,
Baruchet M, et al: Src is activated by the nuclear receptor
peroxisome proliferator-activated receptor β/δ in ultraviolet
radiation-induced skin cancer. EMBO Mol Med. 6:80–98. 2014.
View Article : Google Scholar : PubMed/NCBI
|
23
|
Tang CH, Hsu CJ, Yang WH and Fong YC:
Lipoteichoic acid enhances IL-6 production in human synovial
fibroblasts via TLR2 receptor, PKCdelta and c-Src dependent
pathways. Biochem Picalharmacol. 79:1648–1657. 2010. View Article : Google Scholar
|
24
|
Boyer B, Bourgeois Y and Poupon MF: Src
kinase contributes to the metastatic spread of carcinoma cells.
Oncogene. 21:2347–2356. 2002. View Article : Google Scholar : PubMed/NCBI
|
25
|
Giehl K and Menke A: Microenvironmental
regulation of E-cadherin-mediated adherens junctions. Front Biosci.
13:3975–3985. 2008. View
Article : Google Scholar : PubMed/NCBI
|
26
|
Humar B, Fukuzawa R, Blair V, Dunbier A,
More H, Charlton A, Yang HK, Kim WH, Reeve AE, Martin I and
Guilford P: Destabilized adhesion in the gastric proliferative zone
and c-Src kinase activation mark the development of early diffuse
gastric cancer. Cancer Res. 67:2480–2489. 2007. View Article : Google Scholar : PubMed/NCBI
|
27
|
Lawler K, O'Sullivan G, Long A and Kenny
D: Shear stress induces internalization of E-cadherin and
invasiveness in metastatic oesophageal cancer cells by a
Src-dependent pathway. Cancer Sci. 100:1082–1087. 2009. View Article : Google Scholar : PubMed/NCBI
|
28
|
Summy JM and Gallick GE: Src family
kinases in tumor progression and metastasis. Cancer Metastasis Rev.
22:337–358. 2003. View Article : Google Scholar : PubMed/NCBI
|
29
|
Elsberger B: Translational evidence on the
role of Src kinase and activated Src kinase in invasive breast
cancer. Crit Rev Oncol Hematol. 89:343–351. 2014. View Article : Google Scholar : PubMed/NCBI
|
30
|
Zhou J and Menko AS: The role of Src
family kinases in cortical cataract formation. Invest Ophthalmol
Vis Sci. 43:2293–2300. 2002.PubMed/NCBI
|
31
|
Zhou J and Menko AS: Coordinate signaling
by Src and p38 kinases in the induction of cortical cataract.
Invest Ophthalmol Vis Sci. 45:2314–2323. 2004. View Article : Google Scholar : PubMed/NCBI
|
32
|
Walker JL, Wolff IM, Zhang L and Menko AS:
Activation of SRC kinases signals induction of posterior capsule
opacification. Invest Ophthalmol Vis Sci. 48:2214–2223. 2007.
View Article : Google Scholar : PubMed/NCBI
|
33
|
Peng L, Yang J, Ning C, Zhang J, Xiao X,
He D, Wang X, Li Z, Fu S and Ning J: Rhein inhibits integrin-linked
kinase expression and regulates matrix metalloproteinase-9/tissue
inhibitor of metalloproteinase-1 ratio in high glucose-induced
epithelial-mesenchymal transition of renal tubular cell. Biol Pharm
Bull. 35:1676–1685. 2012. View Article : Google Scholar : PubMed/NCBI
|
34
|
Gu L, Gao Q, Ni L, Wang M and Shen F:
Fasudil inhibits epithelial-myofibroblast transdifferentiation of
human renal tubular epithelial HK-2 cells induced by high glucose.
Chem Pharm Bull (Tokyo). 61:688–694. 2013. View Article : Google Scholar : PubMed/NCBI
|
35
|
Sutariya B, Jhonsa D and Saraf MN: TGF-β:
The connecting link between nephropathy and fibrosis.
Immunopharmacol Immunotoxicol. 38:39–49. 2016. View Article : Google Scholar : PubMed/NCBI
|
36
|
Wang JY, Gao YB, Zhang N, Zou DW, Wang P,
Zhu ZY, Li JY, Zhou SN, Wang SC, Wang YY and Yang JK: miR-21
overexpression enhances TGF-β1-induced epithelial-to-mesenchymal
transition by target smad7 and aggravates renal damage in diabetic
nephropathy. Mol Cell Endocrinol. 392:163–172. 2014. View Article : Google Scholar : PubMed/NCBI
|
37
|
Zhang N, Gao Y, Zou D, Wang J, Li J, Zhou
S, Zhu Z, Zhao X, Xu L and Zhang H: Effects of Chinese Medicine
Tong xinluo on Diabetic nephropathy via inhibiting TGF- β 1-induced
epithelial-to-mesenchymal transition. Evid Based Complent Alternat
Med. 2014:1234972014.
|
38
|
Lu Y, Tang L, Li Y and He Q: High
glucose-induced fibronectin upregulation in cultured mesangial
cells involves caveolin-1-dependent RhoA-GTP activation via Src
kinase. Mol Med Rep. 14:963–968. 2016. View Article : Google Scholar : PubMed/NCBI
|
39
|
Sayin N, Kara N and Pekel G: Ocular
complications of diabetes mellitus. World J Diabetes. 6:92–108.
2015. View Article : Google Scholar : PubMed/NCBI
|
40
|
Xie X, Lan T, Chang X, Huang K, Huang J,
Wang S, Chen C, Shen X, Liu P and Huang H: Connexin43 mediates
NF-κB signalling activation induced by high glucose in GMCs:
Involvement of c-Src. Cell Commun Signal. 11:382013. View Article : Google Scholar : PubMed/NCBI
|
41
|
Alisson-Silva F, Freire-de-Lima L, Donadio
JL, Lucena MC, Penha L, Sá-Diniz JN, Dias WB and Todeschini AR:
Increase of O-glycosylated oncofetal fibronectin in high
glucose-induced epithelial-mesenchymal transition of cultured human
epithelial cells. PLoS One. 8:e604712013. View Article : Google Scholar : PubMed/NCBI
|
42
|
DeMaio L, Buckley ST, Krishnaveni MS,
Flodby P, Dubourd M, Banfalvi A, Xing Y, Ehrhardt C, Minoo P, Zhou
B, et al: Ligand-independent transforming growth factor-β type I
receptor signalling mediates type I collagen-induced
epithelial-mesenchymal transition. J Pathol. 226:633–644. 2012.
View Article : Google Scholar : PubMed/NCBI
|
43
|
Wilson C, Nicholes K, Bustos D, Lin E,
Song Q, Stephan JP, Kirkpatrick DS and Settleman J: Overcoming
EMT-associated resistance to anti-cancer drugs via Src/FAK pathway
inhibition. Oncotarget. 5:7328–7341. 2014. View Article : Google Scholar : PubMed/NCBI
|
44
|
Lee JY, Chang JW, Yang WS, Kim SB, Park
SK, Park JS and Lee SK: Albumin-induced epithelial-mesenchymal
transition and ER stress are regulated through a common ROS-c-Src
kinase-mTOR pathway: effect of imatinib mesylate. Am J Physiol
Renal Physiol. 300:F1214–F1222. 2011. View Article : Google Scholar : PubMed/NCBI
|
45
|
Pang L, Li Q, Wei C, Zou H, Li S, Cao W,
He J, Zhou Y, Ju X, Lan J, et al: TGF-β1/Smad signaling pathway
regulates epithelial-to-mesenchymal transition in esophageal
squamous cell carcinoma: In vitro and clinical analyses of cell
lines and nomadic Kazakh patients from northwest Xinjiang, China.
PLoS One. 9:e1123002014. View Article : Google Scholar : PubMed/NCBI
|
46
|
Maeda M, Shintani Y, Wheelock MJ and
Johnson KR: Src activation is not necessary for transforming growth
factor (TGF)-beta-mediated epithelial to mesenchymal transitions
(EMT) in mammary epithelial cells. PP1 directly inhibits TGF-beta
receptors I and II. J Biol Chem. 281:59–68. 2006. View Article : Google Scholar : PubMed/NCBI
|
47
|
Bartscht T, Lehnert H, Gieseler F and
Ungefroren H: The Src family kinase inhibitors PP2 and PP1
effectively block TGF-beta1-induced cell migration and invasion in
both established and primary carcinoma cells. Cancer Chemother
Pharmacol. 70:221–230. 2012. View Article : Google Scholar : PubMed/NCBI
|
48
|
Hoshino Y, Katsuno Y, Ehata S and Miyazono
K: Autocrine TGF-β protects breast cancer cells from apoptosis
through reduction of BH3-only protein, Bim. J Biochem. 149:55–65.
2011. View Article : Google Scholar : PubMed/NCBI
|
49
|
Itoh S and Itoh F: Implication of TGF-β as
a survival factor during tumour development. J Biochem.
151:559–562. 2012. View Article : Google Scholar : PubMed/NCBI
|
50
|
Hovater MB and Sanders PW: Effect of
dietary salt on regulation of TGF-β in the kindney. Semin Nephrol.
32:269–276. 2012. View Article : Google Scholar : PubMed/NCBI
|
51
|
Deharvengt S, Marmarelis M and Korc M:
Concomitant targeting of EGF receptor, TGF-beta and SRC points to a
novel therapeutic approach in pancreatic cancer. PLoS One.
7:e396842012. View Article : Google Scholar : PubMed/NCBI
|
52
|
Samarakoon R, Chitnis SS, Higgins SP,
Higgins CE, Krepinsky JC and Higgins PJ: Redox-induced Src kinase
and caveolin-1 signaling in TGF-β1-initiated SMAD2/3 activation and
PAI-1 expression. PLoS One. 6:e228962011. View Article : Google Scholar : PubMed/NCBI
|
53
|
Dong S, Khoo A, Wei J, Bowser RK,
Weathington NM, Xiao S, Zhang L, Ma H, Zhao Y and Zhao J: Serum
starvation regulates E-cadherin upregulation via activation of
c-Src in non-small-cell lung cancer A549 cells. Am J Physiol Cell
Physiol. 307:C893–C899. 2014. View Article : Google Scholar : PubMed/NCBI
|