1
|
Qu B, Ma Y, Yan M, Wu HH, Fan L, Liao DF,
Pan XM and Hong Z: The economic burden of fracture patients with
osteoporosis in western China. Osteoporos Int. 25:1853–1860. 2014.
View Article : Google Scholar : PubMed/NCBI
|
2
|
Hendrickx G, Boudin E and Van Hul W: A
look behind the scenes: The risk and pathogenesis of primary
osteoporosis. Nat Rev Rheumatol. 11:462–474. 2015. View Article : Google Scholar : PubMed/NCBI
|
3
|
Rull Ochoa-Hortal MA, Cano-García MC,
Arrabal-Martín M and Arrabal-Polo MA: Lithogenic factors in
postmenopausal women with osteoporotic fracture. Minerva
Endocrinol. 42:41–45. 2017.PubMed/NCBI
|
4
|
Rull MA, Cano-García MC, Arrabal-Martín M
and Arrabal-Polo MA: The importance of urinary calcium in
postmenopausal women with osteoporotic fracture. Can Urol Assoc J.
9:E183–E186. 2015. View Article : Google Scholar : PubMed/NCBI
|
5
|
Li GF, Pan YZ, Sirois P, Li K and Xu YJ:
Iron homeostasis in osteoporosis and its clinical implications.
Osteoporos Int. 23:2403–2408. 2012. View Article : Google Scholar : PubMed/NCBI
|
6
|
Tian Q, Wu S, Dai Z, Yang J, Zheng J,
Zheng Q and Liu Y: Iron overload induced death of osteoblasts in
vitro: Involvement of the mitochondrial apoptotic pathway. PeerJ.
4:e26112016. View Article : Google Scholar : PubMed/NCBI
|
7
|
Sinigaglia L, Fargion S, Fracanzani AL,
Binelli L, Battafarano N, Varenna M, Piperno A and Fiorelli G: Bone
and joint involvement in genetic hemochromatosis: Role of cirrhosis
and iron overload. J Rheumatol. 24:1809–1813. 1997.PubMed/NCBI
|
8
|
Mahachoklertwattana P, Sirikulchayanonta
V, Chuansumrit A, Karnsombat P, Choubtum L, Sriphrapradang A,
Domrongkitchaiporn S, Sirisriro R and Rajatanavin R: Bone
histomorphometry in children and adolescents with beta-thalassemia
disease: Iron-associated focal osteomalacia. J Clin Endocrinol
Metab. 88:3966–3972. 2003. View Article : Google Scholar : PubMed/NCBI
|
9
|
Valenti L, Varenna M, Fracanzani AL, Rossi
V, Fargion S and Sinigaglia L: Association between iron overload
and osteoporosis in patients with hereditary hemochromatosis.
Osteoporos Int. 20:549–555. 2009. View Article : Google Scholar : PubMed/NCBI
|
10
|
Tsay J, Yang Z, Ross FP,
Cunningham-Rundles S, Lin H, Coleman R, Mayer-Kuckuk P, Doty SB,
Grady RW, Giardina PJ, et al: Bone loss caused by iron overload in
a murine model: Importance of oxidative stress. Blood.
116:2582–2589. 2010. View Article : Google Scholar : PubMed/NCBI
|
11
|
Chen B, Li GF, Shen Y, Huang XI and Xu YJ:
Reducing iron accumulation: A potential approach for the prevention
and treatment of postmenopausal osteoporosis. Exp Ther Med.
10:7–11. 2015. View Article : Google Scholar : PubMed/NCBI
|
12
|
Krause A, Neitz S, Mägert HJ, Schulz A,
Forssmann WG, Schulz-Knappe P and Adermann K: LEAP-1, a novel
highly disulfide-bonded human peptide, exhibits antimicrobial
activity. FEBS Lett. 480:147–150. 2000. View Article : Google Scholar : PubMed/NCBI
|
13
|
Pigeon C, Ilyin G, Courselaud B, Leroyer
P, Turlin B, Brissot P and Loréal O: A new mouse liver-specific
gene, encoding a protein homologous to human antimicrobial peptide
hepcidin, is overexpressed during iron overload. J Biol Chem.
276:7811–7819. 2001. View Article : Google Scholar : PubMed/NCBI
|
14
|
Truksa J, Gelbart T, Peng H, Beutler E,
Beutler B and Lee P: Suppression of the hepcidin-encoding gene Hamp
permits iron overload in mice lacking both hemojuvelin and
matriptase-2/TMPRSS6. Br J Haematol. 147:571–581. 2009. View Article : Google Scholar : PubMed/NCBI
|
15
|
Ganz T: Hepcidin, a key regulator of iron
metabolism and mediator of anemia of inflammation. Blood.
102:783–788. 2003. View Article : Google Scholar : PubMed/NCBI
|
16
|
Beaumont-Epinette MP, Delobel JB, Ropert
M, Deugnier Y, Loréal O, Jouanolle AM, Brissot P and Bardou-Jacquet
E: Hereditary hypotransferrinemia can lead to elevated transferrin
saturation and, when associated to HFE or HAMP mutations, to iron
overload. Blood Cells Mol Dis. 54:151–154. 2015. View Article : Google Scholar : PubMed/NCBI
|
17
|
Xu Y, Li G, Du B, Zhang P, Xiao L, Sirois
P and Li K: Hepcidin increases intracellular Ca2+ of
osteoblast hFOB1.19 through L-type Ca2+ channels. Regul
Pept. 172:58–61. 2011. View Article : Google Scholar : PubMed/NCBI
|
18
|
Sun L, Guo W, Yin C, Zhang S, Qu G, Hou Y,
Rong H, Ji H and Liu S: Hepcidin deficiency undermines bone
load-bearing capacity through inducing iron overload. Gene.
543:161–165. 2014. View Article : Google Scholar : PubMed/NCBI
|
19
|
Gulec S, Anderson GJ and Collins JF:
Mechanistic and regulatory aspects of intestinal iron absorption.
Am J Physiol Gastrointest Liver Physiol. 307:G397–G409. 2014.
View Article : Google Scholar : PubMed/NCBI
|
20
|
Rossi E: Hepcidin - the iron regulatory
hormone. Clin Biochem Rev. 26:47–49. 2005.PubMed/NCBI
|
21
|
Rossi F, Perrotta S, Bellini G, Luongo L,
Tortora C, Siniscalco D, Francese M, Torella M, Nobili B, Di Marzo
V, et al: Iron overload causes osteoporosis in thalassemia major
patients through interaction with transient receptor potential
vanilloid type 1 (TRPV1) channels. Haematologica. 99:1876–1884.
2014. View Article : Google Scholar : PubMed/NCBI
|
22
|
Choi HS, Song SH, Lee JH, Kim HJ and Yang
HR: Serum hepcidin levels and iron parameters in children with iron
deficiency. Korean J Hematol. 47:286–292. 2012. View Article : Google Scholar : PubMed/NCBI
|
23
|
Ambroszkiewicz J, Klemarczyk W, Mazur J,
Gajewska J, Rowicka G, Strucińska M and Chełchowska M: Serum
hepcidin and soluble transferrin receptor in the assessment of iron
metabolism in children on a vegetarian diet. Biol Trace Elem Res.
180:182–190. 2017. View Article : Google Scholar : PubMed/NCBI
|